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NFIA encodes a member of the NF1 (nuclear factor 1) family of transcription factors. Additionally we are shipping NFIA Antibodies (64) and NFIA Proteins (7) and many more products for this protein.
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Altogether, these results demonstrated that miR (show MLXIP ELISA Kits)-370 suppressed hepatitis B virus gene expression and replication through repressing NFIA expression, which stimulates hepatitis B virus replication via direct regulation on hepatitis B virus Enhancer I activities.
We verified that NFIA binds to the IGFBP2 (show IGFBP2 ELISA Kits) promoter and transcriptionally enhances IGFBP2 (show IGFBP2 ELISA Kits) expression levels. We identified that NFIA-mediated IGFBP2 (show IGFBP2 ELISA Kits) signaling pathways are involved in miR (show MLXIP ELISA Kits)-302b-induced glioma cell death.
miR (show MLXIP ELISA Kits)-191 was upregulated in patients with middle- and late-stage NSCLC, and in NSCLC cell lines, under mild hypoxic conditions. miR (show MLXIP ELISA Kits)-191 promoted the proliferation and migration of NSCLC under chronic hypoxic conditions, and this promotion may be associated with its targeting of NFIA.
Dihydrocapsaicin can significantly decrease proinflammatory cytokines through enhancing NFIA and inhibiting NF-kappaB (show NFKB1 ELISA Kits) expression
These results demonstrated that RP5833A20.1 inhibited tumor cell proliferation, induced apoptosis and inhibited cellcycle progression by suppressing the expression of NFIA in U251 cells.
NFI-A is involved in the miR (show MLXIP ELISA Kits)-21-induced expression of IL-10 (show IL10 ELISA Kits) in B cells in nasopharyngeal carcinoma; Il-10 (show IL10 ELISA Kits) is capable of suppressing CD8 (show CD8A ELISA Kits)+ T-cell activities.
microRNA-136 targeted and degraded NFIA, which induced the release of microRNA-223, promoting CD11b (show ITGAM ELISA Kits) expression. Direct base pairing occurs between miR (show MLXIP ELISA Kits)-136 and the 3' UTR (show UTS2R ELISA Kits) of NFIA mRNA.
this family also carried a microdeletion affecting solely the NFIA gene, this study substantiates the importance of this gene in craniofacial development.
TGF-beta (show TGFB1 ELISA Kits)-mediated suppression of ANT2 (show SLC25A5 ELISA Kits) through NF1 (show NF1 ELISA Kits)/Smad4 (show SMAD4 ELISA Kits) complexes contributes to oxidative stress and DNA damage during induction of cellular senescence.
RP5-833A20.1/miR (show MLXIP ELISA Kits)-382-5p/NFIA pathway was essential to the regulation of cholesterol homeostasis and inflammatory reactions.
NFIA expression regulates glioma tumorigenesis and glia-specific enhancers enhancers contribute to glioma formation.
NFIA activates the cell-type-specific enhancers and facilitates the binding of PPARgamma (show PPARG ELISA Kits) to control the brown fat gene program.
Astrocyte induction by Zbtb20 is suppressed by knockdown of NFIA.
NFI-A disrupts myeloid cell differentiation and maturation in septic mice
NFIA and NFIB (show NFIB ELISA Kits) are endogenously expressed in gradients with high caudo-medial to low rostro-lateral expression ... this expression pattern deviates from that of PAX6 (show PAX6 ELISA Kits), suggesting that PAX6 (show PAX6 ELISA Kits) does not drive Nfi (show NFIC ELISA Kits) expression
Nfia cooperates with sonic hedgehog (show SHH ELISA Kits) signaling to initiate medulloblastoma.
Our data thus suggest that NFI (show NFIC ELISA Kits) nucleosomal interaction may contribute to the partitioning of distinct chromatin domains and to epigenetic gene expression regulation
As maturing cerebellar granule neurons become more hyperpolarized, NFATc4 (show NFATC4 ELISA Kits) binding declines leading to onset of NFI (show NFIC ELISA Kits) temporal binding and the NFI (show NFIC ELISA Kits) switch program
This gene encodes a member of the NF1 (nuclear factor 1) family of transcription factors. Multiple transcript variants encoding different isoforms have been found for this gene.
nuclear factor I/A
, CCAAT-box-binding transcription factor
, TGGCA-binding protein
, nuclear factor 1 A-type
, nuclear factor 1/A
, nuclear factor I-A