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NOL3 encodes an anti-apoptotic protein that has been shown to down-regulate the enzyme activities of caspase 2, caspase 8 and tumor protein p53. Additionally we are shipping Nucleolar Protein 3 (Apoptosis Repressor with CARD Domain) Antibodies (84) and Nucleolar Protein 3 (Apoptosis Repressor with CARD Domain) Proteins (12) and many more products for this protein.
a novel genetic primary myelofibrosis-like mouse model and identify a tumor suppressor role for NOL3 in the pathogenesis of myeloid malignancies.
Increased ARC expression is associated with liver metastasis of colorectal cancer.
RUNX3 (show RUNX3 ELISA Kits), miR (show MLXIP ELISA Kits)-185 and ARC regulate the sensitivity of gastric cancer cells to chemotherapy.
ARC is regulated via BIRC2 (show BIRC2 ELISA Kits)/MAP3K14 (show MAP3K14 ELISA Kits) signalling and its overexpression in AML (show RUNX1 ELISA Kits) or MSCs can function as a resistant factor to birinapant-induced leukaemia cell death.
high expression of ARC plays an important role in the pathogenesis of nasopharyngeal carcinoma and leads to X-radiation and cisplatin resistance in nasopharyngeal carcinoma.
ARC is a previously unrecognized inhibitor of apoptosis in beta-cells and that its protective effects are mediated through suppression of the ER stress response pathway.
This study utilized unbiased, genome-wide approaches to identify a NOL3 mutation that likely causes Familial cortical myoclonus.
(9)-THC for 24 h produces a dose-dependent NOP receptor down-regulation & decrease in NOP mRNA levels.
HIF-1alpha (show HIF1A ELISA Kits) directly bound to hypoxia-responsive element located at -419 to -414 of ARC gene, which is essential for HIF-1 (show HIF1A ELISA Kits)-induced expression.
Data show that ARC promotes breast carcinogenesis by driving primary tumor growth, invasion, and metastasis as well as by promoting chemoresistance in invasive cells.
Although mice lacking Men1 developed insulinomas as expected, elimination of ARC in this context did not significantly alter tumor load. Cellular rates of proliferation and death in these tumors were also not perturbed in the absence of ARC.
interaction of ARC with TNF (show TNF ELISA Kits) receptor 1 Interferes with recruitment of RIP1 (show RALBP1 ELISA Kits), a critical mediator of TNFalpha (show TNF ELISA Kits)-induced regulated necrosis.
Arc deficiency in dystrophic muscle exacerbates disease pathogenesis due to a Bax (show BAX ELISA Kits)-mediated sensitization of mitochondria-dependent death mechanisms
ARC, previously unlinked to pulmonary hypertension, is a critical determinant of vascular remodeling in this syndrome.
On biomechanical stress induced by aortic banding, ARC-deficient mice developed accelerated cardiomyopathy, which was characterized by reduced contractile function, cardiac enlargement, and myocardial fibrosis
catalase (show CAT ELISA Kits), CK2 (show CSNK2A1 ELISA Kits), and ARC constitute an anti-hypertrophic pathway in the heart.
endogenous nociceptin (show PNOC ELISA Kits) differentially modulates diet preference depending on macronutrient content and homeostatic state, independently of the motivating, rewarding or orosensory properties of food, but may involve metabolic or postingestive processes.
This gene encodes an anti-apoptotic protein that has been shown to down-regulate the enzyme activities of caspase 2, caspase 8 and tumor protein p53. Multiple transcript variants encoding different isoforms have been found for this gene.
muscle-enriched cytoplasmic protein
, nucleolar protein 3
, nucleolar protein of 30 kDa
, apoptosis repressor with CARD