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The protein encoded by PTH is a hormone secreted by parathyroid cells. Additionally we are shipping PTH Antibodies (540) and PTH Proteins (66) and many more products for this protein.
Showing 10 out of 63 products:
Rat (Rattus) PTH ELISA Kit for Competition ELISA - ABIN416371
Naghii, Ghanizadeh, Darvishi, Ebrahimpour, Mofid, Torkaman, Asgari, Hedayati: Whole body vibration is a safe exercise training method and induces no impaired alterations on rat plasma parameters. in Acta physiologica Hungarica 2011
Show all 9 references for ABIN416371
Human PTH ELISA Kit for Competition ELISA - ABIN1979230
Prahalad, Hickey, Huang, Hoelz, Dobrolecki, Murthy, Winata, Hock: Serum proteome profiles identifies parathyroid hormone physiologic response. in Proteomics 2006
Show all 9 references for ABIN1979230
Mouse (Murine) PTH ELISA Kit for Competition ELISA - ABIN415620
Xiao, Cui, Shi, Sun, Wang, Le: Effects of duodenal redox status on calcium absorption and related genes expression in high-fat diet-fed mice. in Nutrition (Burbank, Los Angeles County, Calif.) 2010
Show all 3 references for ABIN415620
Human PTH ELISA Kit for Competition ELISA - ABIN415023
Wang, Yang, Ren, Li, He, Wu, Liu, Lin, Li, Sun: Biomarkers identified by urinary metabonomics for noninvasive diagnosis of nutritional rickets. in Journal of proteome research 2014
Show all 2 references for ABIN415023
Parathyroid hormone gene rs6256 variants are not associated with susceptibility to colorectal cancer
PTH levels were significantly higher in patients with aldosterone producing adenomas.
Although there was a trend for a negative association in women, no statistically significant association was found between endogenous PTH and knee osteoarthritis.
The purpose of this study was to investigate the prevalence of adenomatous colon polyps (ACP) as they occur in subjects with diabetes mellitus (DM) and chronic kidney disease.
PTH [parathyroid hormone]together with other markers of heart failure may provide valuable information both in the diagnosis and staging of heart failure syndromes
Patients with a serum PTH decrease to low values after 1 year of hemodialysis treatment are at high risk of short-term cardiovascular death.
a cell model of human dermal fibroblasts in order to investigate the functions of sclerostin (show SOST ELISA Kits), is reported.
recombinant human klotho (show KL ELISA Kits) inhibits the expression of 1-OH by PTH both in vitro and in vivo.
results suggest that NO-mediated vasorelaxation plays partly a role in the anabolic action of PTH on cortical bone
In this large community-based cohort, PTH levels, overall, were not independently associated with the risk of hypertension. However, we found some evidence that PTH may be associated with hypertension in blacks.
These data highlight the ability of PTH to phosphorylate beta-catenin (show CTNNB1 ELISA Kits) directly via PKA.
These data suggest that prostaglandin E2 acting via EP4R (show PTGER4 ELISA Kits) on bone marrow macrophages committed to the osteoblast cell lineage, stimulated secretion of a factor or factors that acted to suppress PTH-stimulated osteoblast differentiation.
These results suggest that in vivo PTH treatment increased in vitro osteoclastogenesis and resorption without altering the number of osteoclast precursors.
Osteoblast 2-deoxyglucose uptake and glycogen (show GYS2 ELISA Kits) synthesis were increased after exposure to low concentrations (0.1 nmol/l and above) of PTH.
Parathyroid hormone stimulated growth and decreased Col-X deposition via phosphotidylinositol-3,4,5 triphosphate kinase and mitogen activating protein kinase pathways in avian sterna.
likely involvement of the Sp family in regulating PTH gene expression through interactions with an Sp1 (show SP1 ELISA Kits) DNA element in the hormone's promoter.
alternative cis (show CISH ELISA Kits)-acting protein-binding elements may determine the regulation of PTH mRNA stability in response to changes in serum calcium and phosphate
a possible role for the Wnt (show WNT2 ELISA Kits) signaling pathway in PTH actions in bone
Data suggest that calcium-mediated destabilization of parathyroid hormone mRNA requires gene transcription and involves increases in cytosolic Ca.
PTHrP (show PTHLH ELISA Kits) and PTH mediate wasting through a common mechanism involving PTHR (show PTH1R ELISA Kits).
Usp2 (show USP2 ELISA Kits) is required for the PTH1-34-induced proliferation of osteoblasts
These results highlight the role of distal enhancers in the regulation of RANKL (show TNFSF11 ELISA Kits) expression by PTH and perhaps 1,25(OH)2D3 and suggest that the RL-D2 and RL-D5 enhancers contribute in either an additive or synergistic manner to regulate bone remodeling.
These results indicate that PTH-mediated inhibition of renal phosphate transport involves phosphorylation of S77 of the NHERF-1 (show SLC9A3R1 ELISA Kits) PDZ (show INADL ELISA Kits) I domain and the dissociation of NHERF-1 (show SLC9A3R1 ELISA Kits)/Npt2a (show SLC34A1 ELISA Kits) complexes.
It was concluded that endogenously secreted PTH and GHR (show GHR ELISA Kits) signaling in bone are necessary to establish radial bone growth and optimize mineral acquisition during growth.
findings suggest that XLalphas (show GNAS ELISA Kits) enhances Gq/11 signaling to mediate the renal actions of PTH during early postnatal development.
bone adaptation during exercise is not only a function of dynamic loading, but also PTH release, and that PTH signaling contributes differently at the structural and tissue levels.
IGF1 (show IGF1 ELISA Kits) signaling plays a greater role in the skeletal actions of cPTH in the female mouse than in the male mouse, which may underlie the sex differences in the response to cPTH.
Downregulation of PTH receptor expression mediated by intracellular oxidant stress as the mechanisms in hyperlipidemia-induced PTH resistance.
Chondrogenesis induced by PTH in mesenchymal progenitor cells is mediated by Runx1 (show RUNX1 ELISA Kits), which involves the activation of PKA.
The protein encoded by this gene is a hormone secreted by parathyroid cells. This hormone elevates blood Ca2+ level by dissolving the salts in bone and preventing their renal excretion. Defects in this gene are a cause of familial isolated hypoparathyroidism (FIH).
, parathyroid hormone 1
, preproparathyroid hormone
, hypothalamic parathyroid hormone
, thyroid hormone
, probable peptidyl-tRNA hydrolase