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anti-Potassium Channel, Subfamily T, Member 1 (KCNT1) Antibodies

Na(+)-activated potassium channel; may be involved in regulating the firing properties of neurons. Additionally we are shipping Potassium Channel, Subfamily T, Member 1 Proteins (4) and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
KCNT1 57582 Q5JUK3
KCNT1 227632 Q6ZPR4
KCNT1 60444 Q9Z258
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Top anti-Potassium Channel, Subfamily T, Member 1 Antibodies at antibodies-online.com

Showing 10 out of 56 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Supplier Delivery Price Details
Human Rabbit Un-conjugated IHC, ELISA Immunohistochemistry analysis of paraffin-embedded human brain tissue, using KCNT1 Antibody. The picture on the right is treated with the synthesized peptide. 100 μg Log in to see 2 to 3 Days
$302.50
Details
Human Mouse Un-conjugated ICC, IF, IHC, WB Slack Rat Brain Membrane Western Blotting. 100 μg Log in to see 3 to 4 Days
$383.90
Details
Mouse Mouse Un-conjugated IHC (fro), IF, IP, WB 0.1 mg Log in to see 6 to 8 Days
$401.50
Details
Human Rabbit Un-conjugated WB WB Suggested Anti-Kcnt1 AntibodyTitration: 1.0 µg/mL  Positive Control: Mouse Heart 100 μL Log in to see 2 to 3 Days
$289.00
Details
Human Mouse HRP ICC, IF, IHC, WB   100 μg Log in to see 3 to 4 Days
$422.40
Details
Human Mouse FITC ICC, IF, IHC, WB   100 μg Log in to see 3 to 4 Days
$426.80
Details
Human Mouse Alkaline Phosphatase (AP) ICC, IF, IHC, WB   100 μg Log in to see 3 to 4 Days
$429.00
Details
Human Mouse Biotin ICC, IF, IHC, WB   100 μg Log in to see 3 to 4 Days
$431.20
Details
Human Mouse PE ICC, IF, IHC, WB   100 μg Log in to see 3 to 4 Days
$432.30
Details
Human Mouse Streptavidin ICC, IF, IHC, WB   100 μg Log in to see 3 to 4 Days
$433.40
Details

KCNT1 Antibodies by Reactivity, Application, Clonality and Conjugate

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Top referenced anti-Potassium Channel, Subfamily T, Member 1 Antibodies

  1. Human Monoclonal KCNT1 Primary Antibody for ICC, IF - ABIN863116 : Yuan, Santi, Wei, Wang, Pollak, Nonet, Kaczmarek, Crowder, Salkoff: The sodium-activated potassium channel is encoded by a member of the Slo gene family. in Neuron 2003 (PubMed)
    Show all 2 references for ABIN863116

  2. Human Polyclonal KCNT1 Primary Antibody for IHC, ELISA - ABIN1535188 : Humphray, Oliver, Hunt, Plumb, Loveland, Howe, Andrews, Searle, Hunt, Scott, Jones, Ainscough, Almeida, Ambrose, Ashwell, Babbage, Babbage, Bagguley, Bailey, Banerjee, Barker, Barlow, Bates, Beasley et al.: DNA sequence and analysis of human chromosome 9. ... in Nature 2004 (PubMed)

More Antibodies against Potassium Channel, Subfamily T, Member 1 Interaction Partners

Human Potassium Channel, Subfamily T, Member 1 (KCNT1) interaction partners

  1. We demonstrate that KCNT1 mutations are highly pleiotropic and are associated with phenotypes other than nocturnal frontal lobe epilepsy and malignant migrating focal seizures of infancy.

  2. This study demonstrate that KCNT1 mutations are strongly associated with early-onset epileptic encephalopathy.

  3. Five de novo mutations were identified in four genes (SCNN1A (show SCNN1A Antibodies), KCNJ16 (show KCNJ16 Antibodies), KCNB2 (show KCNB2 Antibodies), and KCNT1) in three Brugada syndrome patients (20%)

  4. Nine different mutations of the KCNT1 (Slack) Na(+)-activated K(+) channel (show KCNC4 Antibodies) give rise to three distinct forms of epilepsy.

  5. Slick (show KCNT2 Antibodies) channels, in contrast to the similar Slack channels, are the only high-conductance K+ channels strongly sensitive to small changes in cell volume.

  6. Genetic studies reveal two novel genes for Ohtahara Syndrome: KCNT1 and PIGQ (show PIGQ Antibodies).

  7. Novel variations in KCNT1 do not allow prediction of functional phenotypes that might explain, at least in part, the symptoms of malignant migrating partial seizures of infancy (MMPSI).

  8. This gene-wide tagging study revealed no association between KCNT1 17 common variations and susceptibility of GGEs or AEDs (anti-epileptic drugs) efficacy of genetic generalized epilepsies in Chinese population.

  9. This study demonistrated that KCNT1 mutations implicated in epilepsy cause a marked increase in function

  10. this study performed analysis of KCNT1 in two unrelated patients with malignant migrating partial seizures in infancy.Because the G-to-A transition was located at CG dinucleotide sequences as previously reported for KCNT1 mutations, the recurrent occurrence of de novo KCNT1 mutations indicated the hot spots of these locations.

Mouse (Murine) Potassium Channel, Subfamily T, Member 1 (KCNT1) interaction partners

  1. The authors find that knockout of Slo2.2, but not Slo2.1, results in enhanced itch and pain responses.

  2. Results provide evidence for a role for endogenous Slack channels in higher brain functions, i.e., learning and memory, cognitive flexibility, locomotoric and the ability to initially respond to novel situations and environments

  3. Global ablation of Slack led to increased hypersensitivity in models of neuropathic pain. Neuropathic pain behaviors were also exaggerated after ablation of Slack selectively in sensory neurons.

  4. partly colocalized with PSD-95 (show DLG4 Antibodies) in mouse neocortical neurons

  5. This is the first demonstration of an epithelial cell membrane, Na+-activated, large-conductance K+ channel (show KCNC4 Antibodies) resembling K(Na) channels of excitable cells. The Slo2.2 type, Na+- and Cl--activated K+ channel (show KCNC4 Antibodies) is located in the ascending limb of the kidney.

  6. Slick (show KCNT2 Antibodies) and Slack are expressed at high levels auditory brainstem. Activation of these KNa channels allows temporal accuracy of firing to be increased at high frequencies of stimulation.

Potassium Channel, Subfamily T, Member 1 (KCNT1) Antigen Profile

Protein Summary

Potassium channels represent the most complex class of voltage-gated ion channels from both functional and structural standpoints. Their diverse functions include regulating neurotransmitter release, heart rate, insulin secretion, neuronal excitability, epithelial electrolyte transport, smooth muscle contraction, and cell volume. This gene encodes a sodium-activated potassium channel subunit which is thought to function in ion conductance and developmental signaling pathways. Mutations in this gene cause the early-onset epileptic disorders, malignant migrating partial seizures of infancy and autosomal dominant nocturnal frontal lobe epilepsy. Alternative splicing results in multiple transcript variants.

Gene names and symbols associated with KCNT1

  • potassium channel, subfamily T, member 1 (KCNT1) antibody
  • potassium channel, subfamily T, member 1 (kcnt1) antibody
  • potassium channel, subfamily T, member 1 (Kcnt1) antibody
  • bA100C15.2 antibody
  • C030030G16Rik antibody
  • EIEE14 antibody
  • ENFL5 antibody
  • KCa4.1 antibody
  • MGC146594 antibody
  • MGC172997 antibody
  • mKIAA1422 antibody
  • rSlo2 antibody
  • Slack antibody
  • slo2 antibody
  • Slo2.2 antibody

Protein level used designations for KCNT1

potassium channel, subfamily T, member 1 , potassium channel subfamily T member 1-like , potassium channel subfamily T member 1 , sodium-activated potassium channel , potassium channel subunit (Slack) , sequence like a calcium-activated potassium channel subunit , potassium channel subunit

GENE ID SPECIES
100436526 Pongo abelii
100588032 Nomascus leucogenys
464846 Pan troglodytes
491258 Canis lupus familiaris
529468 Bos taurus
779818 Xenopus (Silurana) tropicalis
100016518 Monodelphis domestica
100316919 Xenopus (Silurana) tropicalis
57582 Homo sapiens
227632 Mus musculus
60444 Rattus norvegicus
395248 Gallus gallus
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