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anti-Potassium Channel, Subfamily V, Member 2 (KCNV2) Antibodies

Voltage-gated potassium (Kv) channels represent the most complex class of voltage-gated ion channels from both functional and structural standpoints. Additionally we are shipping KCNV2 Proteins (4) and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
KCNV2 169522 Q8TDN2
KCNV2 240595 Q8CFS6
KCNV2    
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Top anti-KCNV2 Antibodies at antibodies-online.com

Showing 10 out of 53 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Supplier Delivery Price Details
Human Rabbit Un-conjugated WB WB Suggested Anti-KCNV2 Antibody Titration: 0.2-1 ug/mlELISA Titer: 1:1562500Positive Control: 293T cell lysate 100 μL Log in to see 2 to 3 Days
$289.00
Details
Human Rabbit Un-conjugated EIA, IHC (p), WB Immunohistochemistry analysis in formalin fixed and paraffin embedded human pancreas tissue reacted with KCNV2 Antibody (C-term) followed by peroxidase conjugation of the secondary antibody and DAB staining. Western blot analysis of KCNV2 Antibody (C-term) in 293, K562, HepG2, Jurkat, ZR-75-1 cell line lysates (35ug/lane). This demonstrates the KCNV2 antibody detected the KCNV2 protein (arrow). 0.4 mL Log in to see 6 to 8 Days
$390.50
Details
Human Rabbit Un-conjugated ELISA, WB Western blot analysis of extracts from MCF-7 cells, using KCNV2 Antibody. The lane on the right is treated with the synthesized peptide. 100 μg Log in to see 2 to 3 Days
$302.50
Details
Human Rabbit Un-conjugated IHC (p), WB KCNV2 Antibody (C-term)  western blot analysis in 293,K562,HepG2,Jurkat,ZR-75-1 cell line lysates (35ug/lane).This demonstrates the KCNV2 antibody detected the KCNV2 protein (arrow). KCNV2 Antibody (C-term) immunohistochemistry analysis in formalin fixed and paraffin embedded human pancreas tissue followed by peroxidase conjugation of the secondary antibody and DAB staining.This data demonstrates the use of KCNV2 Antibody (C-term) for immunohistochemistry.  Clinical relevance has not been evaluated. 400 μL Log in to see 10 to 11 Days
$324.50
Details
Human Rabbit Un-conjugated ELISA, WB 50 μg Log in to see 2 to 3 Days
$433.13
Details
Human Rabbit Un-conjugated ELISA, WB 100 μL Log in to see 8 to 10 Days
$478.50
Details
Human Rabbit Un-conjugated WB 50 μg Log in to see 8 to 10 Days
$551.83
Details
Human Rabbit Un-conjugated ELISA, WB 100 μg Log in to see 11 to 16 Days
$412.86
Details
Human Rabbit Un-conjugated IHC, WB 400 μL Log in to see 6 Days
$457.14
Details
Human Rabbit PE IHC, ELISA, WB   200 μL Log in to see 8 to 10 Days
$969.83
Details

KCNV2 Antibodies by Reactivity, Application, Clonality and Conjugate

Attributes Applications Host Clonality Conjugate
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Top referenced anti-KCNV2 Antibodies

  1. Human Polyclonal KCNV2 Primary Antibody for EIA, IHC (p) - ABIN953019 : Robson, Webster, Michaelides, Downes, Cowing, Hunt, Moore, Holder: \Cone dystrophy with supernormal rod electroretinogram\": a comprehensive genotype/phenotype study including fundus autofluorescence and extensive electrophysiology." in Retina (Philadelphia, Pa.) 2010 (PubMed)
    Show all 2 references for ABIN953019

  2. Human Polyclonal KCNV2 Primary Antibody for WB - ABIN2776273 : Ben Salah, Kamei, Sénéćhal, Lopez, Bazalgette, Bazalgette, Eliaou, Zanlonghi, Hamel: Novel KCNV2 mutations in cone dystrophy with supernormal rod electroretinogram. in American journal of ophthalmology 2008 (PubMed)

  3. Human Polyclonal KCNV2 Primary Antibody for ELISA, WB - ABIN1535194 : Humphray, Oliver, Hunt, Plumb, Loveland, Howe, Andrews, Searle, Hunt, Scott, Jones, Ainscough, Almeida, Ambrose, Ashwell, Babbage, Babbage, Bagguley, Bailey, Banerjee, Barker, Barlow, Bates, Beasley et al.: DNA sequence and analysis of human chromosome 9. ... in Nature 2004 (PubMed)

More Antibodies against KCNV2 Interaction Partners

Human Potassium Channel, Subfamily V, Member 2 (KCNV2) interaction partners

  1. This is the first report of genetic and clinical analysis of cone dystrophy with supernormal rod response in the Israeli population leading to the identification of 4 novel KCNV2 mutations.

  2. Compound heterozygosity for the two alleles of KCNV2, p.C177R and p.G461R, in three patients, and homozygosity for complex alleles, p.R27H and p.R206P, in one patient from the consanguineous family, is reported.

  3. Central vision parameters progressively worsen in KCNV2 cone dystrophy, structural retinal and lipofuscin accumulation abnormalities are commonly present and macular cone photoreceptor mosaic is markedly disrupted early in the disease.

  4. KCNV2 mutations cause a unique form of retinal disorder illustrating the importance of K(+)-channels for the resting potential, activation and deactivation of photoreceptors, while phototransduction remains unchanged

  5. important finding leading to identification of KCNV2 as a candidate gene for causative mutations was the characteristic pattern of findings on full field ERGs.

  6. two pore mutations (W467G and G478R) led to the formation of nonconducting heteromeric Kv2.1 (show KCNB1 Antibodies)/Kv8.2 channels

  7. For all patients, KCNV2 sequencing revealed one of three homozygous recessive mutations

  8. In this study, we found that KCNV2 mutations are present in a substantial fraction (2.2-4.3%) of a sample of 367 independent patients with a variety of initial clinical diagnoses of cone malfunctino.

  9. In KCNV2 retinopathy foveal morphological changes are evident on SD-OCT (show Plxna2 Antibodies) even in the early stages of disease.

  10. Early ocular phenotype in siblings with a homozygous p.G461R mutation in the KCNV2 gene presented nystagmus, increased light sensitivity, reduced color discrimination, and relative central scotomas.

Mouse (Murine) Potassium Channel, Subfamily V, Member 2 (KCNV2) interaction partners

  1. We conclude that nucleotide changes in evolutionary conserved crx (show CRX Antibodies) binding site could impact retina-specific expression levels of Kcnv2.

  2. two pore mutations (W467G and G478R) led to the formation of nonconducting heteromeric Kv2.1 (show KCNB1 Antibodies)/Kv8.2 channels

  3. Results demonstrate that altered potassium subunit function influences epilepsy susceptibility and implicate Kcnv2 as an epilepsy gene.

  4. Data show that total Kv11.1 alpha subunit (show POLG Antibodies) protein is more abundant in neonatal heart and is distributed throughout both adult and neonatal ventricles with greater abundance in epicardia.

  5. Kcnv2 is a strong functional candidate for epilepsy modifier.

KCNV2 Antigen Profile

Protein Summary

Voltage-gated potassium (Kv) channels represent the most complex class of voltage-gated ion channels from both functional and structural standpoints. Their diverse functions include regulating neurotransmitter release, heart rate, insulin secretion, neuronal excitability, epithelial electrolyte transport, smooth muscle contraction, and cell volume. This gene encodes a member of the potassium voltage-gated channel subfamily V. This member is identified as a 'silent subunit', and it does not form homomultimers, but forms heteromultimers with several other subfamily members. Through obligatory heteromerization, it exerts a function-altering effect on other potassium channel subunits. This protein is strongly expressed in pancreas and has a weaker expression in several other tissues.

Gene names and symbols associated with KCNV2

  • potassium channel, subfamily V, member 2 (KCNV2) antibody
  • potassium channel, subfamily V, member 2 (Kcnv2) antibody
  • Kv8.2 antibody
  • KV11.1 antibody
  • RCD3B antibody

Protein level used designations for KCNV2

potassium voltage-gated channel subfamily V member 2 , voltage-gated potassium channel subunit Kv8.2

GENE ID SPECIES
169522 Homo sapiens
240595 Mus musculus
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