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Potassium Inwardly-Rectifying Channel, Subfamily J, Member 10 Proteins (KCNJ10)

KCNJ10 encodes a member of the inward rectifier-type potassium channel family, characterized by having a greater tendency to allow potassium to flow into, rather than out of, a cell. Additionally we are shipping KCNJ10 Antibodies (53) and KCNJ10 Kits (14) and many more products for this protein.

list all proteins Gene Name GeneID UniProt
KCNJ10 16513 Q9JM63
KCNJ10 3766 P78508
Rat KCNJ10 KCNJ10 29718 P49655
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Top KCNJ10 Proteins at antibodies-online.com

Showing 5 out of 5 products:

Catalog No. Origin Source Conjugate Images Quantity Supplier Delivery Price Details
HOST_Escherichia coli (E. coli) Human His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details
Insect Cells Human rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.5 mg Log in to see 49 to 54 Days
$6,041.49
Details
Insect Cells Mouse rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.25 mg Log in to see 49 to 54 Days
$4,244.78
Details
HOST_Escherichia coli (E. coli) Mouse His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details
HOST_Wheat germ Human GST tag 10 μg Log in to see 9 Days
$405.71
Details

KCNJ10 Proteins by Origin and Source

Origin Expressed in Conjugate
Mouse (Murine) ,
,
Human , ,
, ,

More Proteins for Potassium Inwardly-Rectifying Channel, Subfamily J, Member 10 (KCNJ10) Interaction Partners

Xenopus laevis Potassium Inwardly-Rectifying Channel, Subfamily J, Member 10 (KCNJ10) interaction partners

  1. Ordered disorder of the astrocytic dystrophin (show DMD Proteins)-associated protein complex in the norm and pathology.

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 10 (KCNJ10) interaction partners

  1. disruption of cav-1 (show CAV1 Proteins) decreases basolateral K(+) channel (show KCNC4 Proteins) activity and depolarizes the cell membrane potential in the DCT1 (show SLC11A2 Proteins) at least in part by suppressing the stimulatory effect of c-Src (show SRC Proteins) on Kcnj10

  2. Data suggest that astroglial Kir4.1 channels are crucially involved in extracellular potassium homeostasis regulating theta rhythmic activity.

  3. thyroglobulin (show TG Proteins) trafficking might be modulated by Kir4.1/5.1.

  4. Kcnj10 plays a role in Muller glia maturation during retinal development probably through ionic channel activities.

  5. Kir (show GEM Proteins).4.1 is expressed in the basolateral membrane of cortical thick ascending limb (cTAL) and that the disruption of Kir (show GEM Proteins).4.1 has no significant effect on the membrane potential of the cTAL limb and Na-K-Cl cotransporter (show SLC12A1 Proteins) 2 (show SLC12A5 Proteins) expression

  6. Kcnj10 is a main potassium channel (show KCNAB2 Proteins) expressed in corneal epithelial cells.

  7. Kcnj10 is a main contributor to the basolateral K conductance in the early distal convoluted tubule (DCT1 (show SLC11A2 Proteins)) and determines the expression of the apical Na-Cl cotransporter (NCC (show SLC12A3 Proteins)) in the DCT (show DCT Proteins).

  8. TBI (show TBPL1 Proteins) affects expression of Kir4.1 and GLT-1 (show SLC1A2 Proteins) genes in age- and time dependent manner and it may lead to accumulations of more K(+) and glutamate (show GRIN1 Proteins) early in the synapse of old mice as compared to adult

  9. Ordered disorder of the astrocytic dystrophin (show DMD Proteins)-associated protein complex in the norm and pathology.

  10. the modulation of tyrosine phosphorylation of KCNJ10 should play a role in regulating membrane transport function in DCT1 (show SLC11A2 Proteins).

Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 10 (KCNJ10) interaction partners

  1. Previous research had shown that Kir4.1 protein autoantibodies were specific for multiple sclerosis but they found that they weren't.

  2. Previous research had shown that Kir4.1 protein autoantibodies were specific for multiple sclerosis but they found that they weren't.

  3. This study identifies potential SNPs of KCNJ10 gene that may contribute to seizure susceptibility and anti-epileptic drug resistance.

  4. disruption of cav-1 (show CAV1 Proteins) decreases basolateral K(+) channel (show KCNC4 Proteins) activity and depolarizes the cell membrane potential in the DCT1 (show SLC11A2 Proteins) at least in part by suppressing the stimulatory effect of c-Src (show SRC Proteins) on Kcnj10

  5. anti-KIR4.1 antibody levels differed in multiple sclerosis patients during relapse and remission; as such, they may represent a marker of disease exacerbation

  6. This study showed that rs2486253, but not rs61822012, polymorphism of KCNJ10 gene was associated with childhood idiopathic generalized epilepsy.

  7. we confirmed the presence of anti-Kir4.1 antibodies in multiple sclerosis patients, but at a much lower prevalence than previously reported.

  8. KCNJ10 SNP is not associated with nonsyndromic enlargement of vestibular aqueduct in Chinese patients.

  9. No KIR4.1-specific antigen is detected in serum or cerebrospinal fluid of multiple sclerosis (MS) patients; the target antigen of MS remains elusive.

  10. This study observed a decrease of astroglial KIR4.1 but not glial fibrillary acidic protein (show GFAP Proteins) IR. In chronic inactive and remyelinating MS lesions, KIR4.1 IR was restored on astrocytes and found in a subset of presumably new myelinating oligodendrocytes.

KCNJ10 Protein Profile

Protein Summary

This gene encodes a member of the inward rectifier-type potassium channel family, characterized by having a greater tendency to allow potassium to flow into, rather than out of, a cell. The encoded protein may form a heterodimer with another potassium channel protein and may be responsible for the potassium buffering action of glial cells in the brain. Mutations in this gene have been associated with seizure susceptibility of common idiopathic generalized epilepsy syndromes.

Gene names and symbols associated with KCNJ10

  • potassium inwardly-rectifying channel, subfamily J, member 10 (kcnj10)
  • potassium inwardly-rectifying channel, subfamily J, member 10 (KCNJ10)
  • potassium inwardly-rectifying channel, subfamily J, member 10 (Kcnj10)
  • BIR10 protein
  • BIRK-1 protein
  • BIRK-10 protein
  • KCNJ13-PEN protein
  • KIR1.2 protein
  • KIR4.1 protein
  • MGC145926 protein
  • SESAME protein

Protein level used designations for KCNJ10

potassium inwardly-rectifying channel, subfamily J, member 10 , ATP-sensitive inward rectifier potassium channel 10 , potassium inwardly-rectifying channel J10 , inward rectifier K(+) channel Kir4.1 , potassium channel, inwardly rectifying subfamily J member 10 , ATP-dependent inwardly rectifying potassium channel Kir4.1 , glial ATP-dependent inwardly rectifying potassium channel KIR4.1 , inward rectifier K(+) channel Kir1.2 , inward rectifier K+ channel KIR1.2 , ATP-sensitive inward rectifier potassium channel KAB-2 , BIR10 , BIRK1 , brain-specific inwardly rectifying K(+) channel 1 , kir4.1

GENE ID SPECIES
494778 Xenopus laevis
533343 Bos taurus
779765 Xenopus (Silurana) tropicalis
16513 Mus musculus
3766 Homo sapiens
29718 Rattus norvegicus
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