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anti-Potassium Inwardly-Rectifying Channel, Subfamily J, Member 13 (KCNJ13) Antibodies

KCNJ13 encodes a member of the inwardly rectifying potassium channel family of proteins. Additionally we are shipping KCNJ13 Proteins (4) and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
KCNJ13 3769 O60928
KCNJ13 100040591 P86046
KCNJ13 94341 O70617
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Top anti-KCNJ13 Antibodies at antibodies-online.com

Showing 10 out of 34 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Supplier Delivery Price Details
Human Rabbit Un-conjugated IHC (p), WB KCNJ13 Antibody (N-term) (ABIN656321) immunohistochemistry analysis in formalin fixed and paraffin embedded human stomach tissue followed by peroxidase conjugation of the secondary antibody and DAB staining. 400 μL Log in to see 10 to 11 Days
$324.50
Details
Human Rabbit Un-conjugated EIA, IHC (p), WB Immunohistochemistry analysis in formalin fixed and paraffin embedded human stomach tissue reacted with KCNJ13 Antibody (N-term) followed which was peroxidase conjugated to the secondary antibody and  followed by DAB staining. Western blot analysis of KCNJ13 Antibody (N-term) in MDA-MB435 cell line lysates (35ug/lane). This demonstrates the KCNJ13 antibody detected the KCNJ13 protein (arrow). 0.4 mL Log in to see 6 to 8 Days
$390.50
Details
Human Rabbit Un-conjugated IHC (p), WB Human Adrenal: Formalin-Fixed, Paraffin-Embedded (FFPE) 200 μL Log in to see 8 to 10 Days
$451.00
Details
Human Rabbit Un-conjugated IHC, WB   100 μL Log in to see 7 to 8 Days
$454.67
Details
Human Rabbit Un-conjugated IHC, WB 400 μL Log in to see 6 Days
$457.14
Details
Human Rabbit APC IHC, ELISA, WB   200 μL Log in to see 8 to 10 Days
$969.83
Details
Human Rabbit Un-conjugated WB   500 μg Log in to see 8 to 10 Days
$493.17
Details
Human Rabbit Un-conjugated IHC, ELISA, WB   200 μL Log in to see 11 to 16 Days
$532.71
Details
Human Rabbit APC IHC, ELISA, WB   200 μL Log in to see 16 Days
$710.29
Details
Human Rabbit FITC IHC, ELISA, WB   200 μL Log in to see 16 Days
$710.29
Details

KCNJ13 Antibodies by Reactivity, Application, Clonality and Conjugate

Attributes Applications Host Clonality Conjugate
Human , ,


, , , , ,
Mouse (Murine)


Rat (Rattus)


Top referenced anti-KCNJ13 Antibodies

  1. Human Polyclonal KCNJ13 Primary Antibody for IHC, WB - ABIN350391 : Derst, Doering, Preisig-Mueller, Daut, Karschin, Jeck, Weber, Engel, Grzeschik: Partial gene structure and assignment to chromosome 2q37 of the human inwardly rectifying K+ channel (Kir7.1) gene (KCNJ13). in Genomics 1999 (PubMed)
    Show all 4 references for ABIN350391

  2. Human Polyclonal KCNJ13 Primary Antibody for EIA, IHC (p) - ABIN953008 : Zhang, Zitron, Bloehs, Müller-Krebs, Scholz, Zeier, Katus, Karle, Schwenger: Dual regulation of renal Kir7.1 potassium channels by protein Kinase A and protein Kinase C. in Biochemical and biophysical research communications 2008 (PubMed)
    Show all 3 references for ABIN953008

  3. Human Polyclonal KCNJ13 Primary Antibody for IHC (p), WB - ABIN656321 : Fang, Li, Welling: Hypertension resistance polymorphisms in ROMK (Kir1.1) alter channel function by different mechanisms. in American journal of physiology. Renal physiology 2010 (PubMed)
    Show all 2 references for ABIN656321

More Antibodies against KCNJ13 Interaction Partners

Cow (Bovine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 13 (KCNJ13) interaction partners

  1. the activity of Kir channels in the RPE is critically dependent on the regeneration of membrane PIP2 by PI4 kinases and that this may explain the dependence of these channels on hydrolyzable ATP.

Zebrafish Potassium Inwardly-Rectifying Channel, Subfamily J, Member 13 (KCNJ13) interaction partners

  1. These results suggest that the cellular defect of the Kir7.1 mutation is directly responsible for the pattern change in the jaguar/obelix mutant.

Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 13 (KCNJ13) interaction partners

  1. Kir7.1 mutations are associated with vision disorders to include novel insights into the molecular mechanism of disease pathobiology in Leber Congenital Amaurosis.

  2. Juvenile or early-adult-onset cataract in the setting of a congenital vitreo-retinal dystrophy (show MERTK Antibodies) notable for fibrosis over the disc and clumped pigmentation in the posterior pole is a unique phenotype that suggests recessive KCNJ13 mutations.

  3. Kir7.1 regulates the transition from quiescence to contractions in the pregnant uterus.

  4. Kir7.1, R162W mutant showed a reduction of IKir7.1 and positive shift in '0' current potential.

  5. Kir7.1 expression was found in 100% of choroid plexus tumors and was absent in endolymphatic sac (show ADCY10 Antibodies) tumors.

  6. A homozygous nonsense mutation was found in the potassium channel subunit (show KCNT1 Antibodies) gene KCNJ13 that caused leber congenital amaurosis.

  7. This study confirms the expression of Kir7.1 in human RPE (show RPE Antibodies), identifies a Kir7.1 splice variant resulting in predicted changes in protein sequence, and indicates that there is no functional interaction between this splice variant and full-length Kir7.1.

  8. Kir7.1 channels are modulated by intracellular protons by diverse mechanisms; H26 is important for channel activation at physiological pH(i) and it influences an unidentified proton-induced inhibitory mechanism.

  9. These results indicate that the KCNJ13 R162W mutation can cause Snowflake vitreoretinal degeneration and further show that vitreoretinal degeneration can arise through mutations in genes whose products are not structural components of the vitreous.

  10. This study demonstrates the dual regulation of Kir7.1 channel function by PKA and PKC.

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 13 (KCNJ13) interaction partners

  1. These results suggest that KCNJ13 expression is required for RPE (show RPE Antibodies) cells to maintain photoreceptor survival.

  2. Kir7.1 regulates the transition from quiescence to contractions in the pregnant uterus.

  3. Coupling of MC4R (show MC4R Antibodies) to Kir7.1 may explain unusual aspects of the control of energy homeostasis by melanocortin signalling, including the gene dosage effect of MC4R (show MC4R Antibodies) and the sustained effects of AgRP (show AGRP Antibodies) on food intake.

KCNJ13 Antigen Profile

Protein Summary

This gene encodes a member of the inwardly rectifying potassium channel family of proteins. Members of this family form ion channel pores that allow potassium ions to pass into a cell. The encoded protein belongs to a subfamily of low signal channel conductance proteins that have a low dependence on potassium concentration. Mutations in this gene are associated with snowflake vitreoretinal degeneration. Alternate splicing results in multiple transcript variants.

Gene names and symbols associated with KCNJ13

  • potassium inwardly-rectifying channel, subfamily J, member 13 (KCNJ13) antibody
  • potassium inwardly-rectifying channel, subfamily J, member 13 (kcnj13) antibody
  • potassium inwardly-rectifying channel, subfamily J, member 13 (Kcnj13) antibody
  • KIR1.4 antibody
  • Kir7.1 antibody
  • LCA16 antibody
  • SVD antibody

Protein level used designations for KCNJ13

potassium inwardly-rectifying channel, subfamily J, member 13 , inward rectifier potassium channel 13 , jag , obe , jaguar , obelix , inwardly rectifying potassium channel 7.1 , inward rectifier potassium channel 13-like , inward rectifier K(+) channel Kir7.1 , potassium channel, inwardly rectifying subfamily J member 13 , inwardly rectifying potassium channel Kir7.1

GENE ID SPECIES
477411 Canis lupus familiaris
512288 Bos taurus
555691 Danio rerio
100125048 Xenopus (Silurana) tropicalis
100402605 Callithrix jacchus
100595442 Nomascus leucogenys
100615579 Pan troglodytes
3769 Homo sapiens
100040591 Mus musculus
94341 Rattus norvegicus
100379277 Cavia porcellus
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