You are viewing an incomplete version of our website. Please click to reload the website as full version.

Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) ELISA Kits

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Additionally we are shipping Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Antibodies (55) and Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Proteins (5) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
Anti-Human KCNJ16 KCNJ16 3773 Q9NPI9
Anti-Mouse KCNJ16 KCNJ16 16517 Q9Z307
Anti-Rat KCNJ16 KCNJ16 29719  
How to order from antibodies-online
  • +1 877 302 8632
  • +1 888 205 9894 (toll-free)
  • Order online
  • orders@antibodies-online.com

Top Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 ELISA Kits at antibodies-online.com

Showing 1 out of 2 products:

Catalog No. Reactivity Sensitivity Range Quantity Supplier Delivery Price Details
Please inquire
96 Tests Log in to see 2 to 3 Days
$450.00
Details

More ELISA Kits for Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Interaction Partners

Xenopus laevis Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) interaction partners

  1. Kir4.1 (show KCNJ10 ELISA Kits) and Kir5.1 potassium channels bind with a heteromeric interaction that influences rectification, gating, and pH sensitivity

  2. these results suggest that the Kir4.1-Kir5.1 but not the homomeric Kir4.1 channel is strongly inhibited by PKC activation.

Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) interaction partners

  1. Variability has been found in a three-generation family with Pierre Robin sequence, acampomelic campomelic dysplasia, and intellectual disability due to a novel approximately 1 Mb deletion upstream of SOX9 (show SOX9 ELISA Kits), and including KCNJ2 (show KCNJ2 ELISA Kits) and KCNJ16.

  2. Five de novo mutations were identified in four genes (SCNN1A (show SCNN1A ELISA Kits), KCNJ16, KCNB2 (show KCNB2 ELISA Kits), and KCNT1 (show KCNT1 ELISA Kits)) in three Brugada syndrome patients

  3. study provides an explanation for the pathophysiology of the p.A167V KCNJ10 (show KCNJ10 ELISA Kits) mutation, which had not been considered pathogenic on its own; findings provide evidence for functional cooperation of KCNJ10 (show KCNJ10 ELISA Kits) and KCNJ16; in vitro ascertainment of KCNJ10 (show KCNJ10 ELISA Kits) function may necessitate co-expression with KCNJ16

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) interaction partners

  1. thyroglobulin (show TG ELISA Kits) trafficking might be modulated by Kir4.1 (show KCNJ10 ELISA Kits)/5.1.

  2. Kir5.1 is inhibited in cortical collecting duct cells by dopamine

  3. These results highlight the important role that Kir5.1 plays as a pH-sensitive regulator of salt transport in the DCT (show DCT ELISA Kits), and the implication of these results for the correct genetic diagnosis of renal tubulopathies is discussed.

  4. Kir5.1 may be involved in the response to hypercapnic acidosis

  5. Kir4.1 (show KCNJ10 ELISA Kits)/5.1 and Kir4.1 (show KCNJ10 ELISA Kits) expression appeared to occur only in astrocytes, specifically in the membrane domains facing the pia (show RICTOR ELISA Kits) mater (show NLRP5 ELISA Kits) and blood vessels or in the processes surrounding synapses.

  6. the Kir4.1 (show KCNJ10 ELISA Kits)/Kir5.1 channel is a major component of the K(+) conductance in the basolateral membrane of mouse cortical collecting duct principal cells

Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) Antigen Profile

Antigen Summary

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium\; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. KCNJ16 may be involved in the regulation of fluid and pH balance.

Gene names and symbols associated with KCNJ16

  • potassium inwardly-rectifying channel, subfamily J, member 16 (kcnj16-A) antibody
  • potassium inwardly-rectifying channel, subfamily J, member 16 (KCNJ16) antibody
  • potassium inwardly-rectifying channel, subfamily J, member 16 (kcnj16) antibody
  • potassium inwardly-rectifying channel, subfamily J, member 16 (Kcnj16) antibody
  • 6430410F18Rik antibody
  • AI132396 antibody
  • BIR9 antibody
  • KCNJ16 antibody
  • kcnj16-A antibody
  • KIR5.1 antibody

Protein level used designations for KCNJ16

potassium inwardly-rectifying channel, subfamily J, member 16 , potassium inwardly-rectifying channel J16 , inward rectifier potassium channel 16 , inwardly-rectifying potassium channel Kir5.1 , inward rectifier K(+) channel Kir5.1 , inward rectifier K+ channel KIR5.1 , potassium channel, inwardly rectifying subfamily J member 16 , BIR9 , potassium inwardly-rectifying channel subfamily J member 16

GENE ID SPECIES
373785 Xenopus laevis
427812 Gallus gallus
468311 Pan troglodytes
613890 Bos taurus
694188 Macaca mulatta
100145730 Xenopus (Silurana) tropicalis
100379597 Cavia porcellus
3773 Homo sapiens
16517 Mus musculus
29719 Rattus norvegicus
611045 Canis lupus familiaris
Selected quality suppliers for Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) ELISA Kits
Did you look for something else?