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Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Proteins (KCNJ16)

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Additionally we are shipping Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Antibodies (55) and Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Kits (2) and many more products for this protein.

list all proteins Gene Name GeneID UniProt
KCNJ16 3773 Q9NPI9
KCNJ16 16517 Q9Z307
Rat KCNJ16 KCNJ16 29719  
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Top Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 Proteins at antibodies-online.com

Showing 5 out of 5 products:

Catalog No. Origin Source Conjugate Images Quantity Supplier Delivery Price Details
Insect Cells Mouse rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.25 mg Log in to see 49 to 54 Days
$4,244.78
Details
HOST_Escherichia coli (E. coli) Mouse His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details
HOST_Escherichia coli (E. coli) Human His tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 1 mg Log in to see 29 to 34 Days
$4,331.68
Details
Insect Cells Human rho-1D4 tag „Crystallography Grade“ protein due to multi-step, protein-specific purification process 0.5 mg Log in to see 49 to 54 Days
$6,041.49
Details
HOST_Wheat germ Human GST tag 10 μg Log in to see 9 Days
$405.71
Details

KCNJ16 Proteins by Origin and Source

Origin Expressed in Conjugate
Human , ,
, ,
Mouse (Murine) ,
,

More Proteins for Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) Interaction Partners

Xenopus laevis Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) interaction partners

  1. Kir4.1 (show KCNJ10 Proteins) and Kir5.1 potassium channels bind with a heteromeric interaction that influences rectification, gating, and pH sensitivity

  2. these results suggest that the Kir4.1-Kir5.1 but not the homomeric Kir4.1 channel is strongly inhibited by PKC activation.

Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) interaction partners

  1. Variability has been found in a three-generation family with Pierre Robin sequence, acampomelic campomelic dysplasia, and intellectual disability due to a novel approximately 1 Mb deletion upstream of SOX9 (show SOX9 Proteins), and including KCNJ2 (show KCNJ2 Proteins) and KCNJ16.

  2. Five de novo mutations were identified in four genes (SCNN1A (show SCNN1A Proteins), KCNJ16, KCNB2 (show KCNB2 Proteins), and KCNT1 (show KCNT1 Proteins)) in three Brugada syndrome patients

  3. study provides an explanation for the pathophysiology of the p.A167V KCNJ10 (show KCNJ10 Proteins) mutation, which had not been considered pathogenic on its own; findings provide evidence for functional cooperation of KCNJ10 (show KCNJ10 Proteins) and KCNJ16; in vitro ascertainment of KCNJ10 (show KCNJ10 Proteins) function may necessitate co-expression with KCNJ16

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) interaction partners

  1. thyroglobulin (show TG Proteins) trafficking might be modulated by Kir4.1 (show KCNJ10 Proteins)/5.1.

  2. Kir5.1 is inhibited in cortical collecting duct cells by dopamine

  3. These results highlight the important role that Kir5.1 plays as a pH-sensitive regulator of salt transport in the DCT (show DCT Proteins), and the implication of these results for the correct genetic diagnosis of renal tubulopathies is discussed.

  4. Kir5.1 may be involved in the response to hypercapnic acidosis

  5. Kir4.1/5.1 and Kir4.1 expression appeared to occur only in astrocytes, specifically in the membrane domains facing the pia mater and blood vessels or in the processes surrounding synapses.

  6. the Kir4.1 (show KCNJ10 Proteins)/Kir5.1 channel is a major component of the K(+) conductance in the basolateral membrane of mouse cortical collecting duct principal cells

Potassium Inwardly-Rectifying Channel, Subfamily J, Member 16 (KCNJ16) Protein Profile

Protein Summary

Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium\; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. KCNJ16 may be involved in the regulation of fluid and pH balance.

Gene names and symbols associated with KCNJ16

  • potassium inwardly-rectifying channel, subfamily J, member 16 (kcnj16-A)
  • potassium inwardly-rectifying channel, subfamily J, member 16 (KCNJ16)
  • potassium inwardly-rectifying channel, subfamily J, member 16 (kcnj16)
  • potassium inwardly-rectifying channel, subfamily J, member 16 (Kcnj16)
  • 6430410F18Rik protein
  • AI132396 protein
  • BIR9 protein
  • KCNJ16 protein
  • kcnj16-A protein
  • KIR5.1 protein

Protein level used designations for KCNJ16

potassium inwardly-rectifying channel, subfamily J, member 16 , potassium inwardly-rectifying channel J16 , inward rectifier potassium channel 16 , inwardly-rectifying potassium channel Kir5.1 , inward rectifier K(+) channel Kir5.1 , inward rectifier K+ channel KIR5.1 , potassium channel, inwardly rectifying subfamily J member 16 , BIR9 , potassium inwardly-rectifying channel subfamily J member 16

GENE ID SPECIES
373785 Xenopus laevis
427812 Gallus gallus
468311 Pan troglodytes
613890 Bos taurus
694188 Macaca mulatta
100145730 Xenopus (Silurana) tropicalis
100379597 Cavia porcellus
3773 Homo sapiens
16517 Mus musculus
29719 Rattus norvegicus
611045 Canis lupus familiaris
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