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anti-Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 (KCNJ3) Antibodies

Potassium channels are present in most mammalian cells, where they participate in a wide range of physiologic responses. Additionally we are shipping Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 Kits (5) and Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 Proteins (5) and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
KCNJ3 3760 P48549
KCNJ3 16519 P63250
KCNJ3 50599 P63251
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Top anti-Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 Antibodies at antibodies-online.com

Showing 10 out of 94 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Supplier Delivery Price Details
Mouse Rabbit Un-conjugated WB Western blot analysis of rat brain membranes: 1. Anti-Kir3.1 (GIRK1) antibody, (1:200). 2. Anti-Kir3.1 (GIRK1) antibody, preincubated with the control antigen. 25 μL Log in to see 10 to 11 Days
$535.71
Details
Dog Rabbit Un-conjugated IF (p), IHC (p), WB Antigen: 0.2 µg/100 µL  Primary: Antiserum, 1:500, 1:1000, 1:2000, 1:4000, 1:8000, 1:16000, 1:32000;  Secondary: HRP conjugated Goat-Anti-Rabbit IgG at 1: 5000;  TMB staining;  Read the data in MicroplateReader by 450 Mouse kidney lysate probed with Rabbit Anti-GIRK1 Polyclonal Antibody  at 1:5000 90min in 37˚C 100 μL Log in to see 3 to 7 Days
$269.50
Details
Human Mouse Un-conjugated IF, WB Anti-KCNJ3 mouse monoclonal antibody (ABIN2455445) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY KCNJ3 (RC205322). HEK293T cells were transfected with the pCMV6-ENTRY control (Left lane) or pCMV6-ENTRY KCNJ3 (Right lane) cDNA for 48 hrs and lysed. Equivalent amounts of cell lysates (5 µg per lane) were separated by SDS-PAGE and immunoblotted with anti-KCNJ3. 100 μL Log in to see 2 to 3 Days
$423.50
Details
Human Mouse Un-conjugated IF, WB Anti-KCNJ3 mouse monoclonal antibody (ABIN2455446) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY KCNJ3 (RC205322). HEK293T cells were transfected with the pCMV6-ENTRY control (Left lane) or pCMV6-ENTRY KCNJ3 (Right lane) cDNA for 48 hrs and lysed. Equivalent amounts of cell lysates (5 µg per lane) were separated by SDS-PAGE and immunoblotted with anti-KCNJ3. 100 μL Log in to see 2 to 3 Days
$423.50
Details
Human Mouse Un-conjugated FACS, IF, IHC, WB Anti-KCNJ3 mouse monoclonal antibody (ABIN2455488) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY KCNJ3 (RC205322). HEK293T cells were transfected with the pCMV6-ENTRY control (Left lane) or pCMV6-ENTRY KCNJ3 (Right lane) cDNA for 48 hrs and lysed. Equivalent amounts of cell lysates (5 µg per lane) were separated by SDS-PAGE and immunoblotted with anti-KCNJ3. 100 μL Log in to see 2 to 3 Days
$423.50
Details
Human Mouse Un-conjugated FACS, IF, WB HEK293T cells transfected with either RC205322 overexpress plasmid (Red) or empty vector control plasmid (Blue) were immunostained by anti-KCNJ3 antibody (ABIN2455489), and then analyzed by flow cytometry. Anti-KCNJ3 mouse monoclonal antibody (ABIN2455489) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY KCNJ3 (RC205322). 100 μL Log in to see 2 to 3 Days
$423.50
Details
Human Mouse Un-conjugated FACS, WB HEK293T cells transfected with either RC205322 overexpress plasmid (Red) or empty vector control plasmid (Blue) were immunostained by anti-KCNJ3 antibody (ABIN2455444), and then analyzed by flow cytometry. HEK293T cells were transfected with the pCMV6-ENTRY control (Left lane) or pCMV6-ENTRY KCNJ3 (Right lane) cDNA for 48 hrs and lysed. Equivalent amounts of cell lysates (5 µg per lane) were separated by SDS-PAGE and immunoblotted with anti-KCNJ3. 100 μL Log in to see 2 to 3 Days
$423.50
Details
Human Mouse Un-conjugated FACS, IF HEK293T cells transfected with either RC205322 overexpress plasmid (Red) or empty vector control plasmid (Blue) were immunostained by anti-KCNJ3 antibody (ABIN2455485), and then analyzed by flow cytometry. Anti-KCNJ3 mouse monoclonal antibody (ABIN2455485) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY KCNJ3 (RC205322). 100 μL Log in to see 2 to 3 Days
$423.50
Details
Human Mouse Un-conjugated FACS, IF, IHC, WB Anti-KCNJ3 mouse monoclonal antibody (ABIN2455456) immunofluorescent staining of COS7 cells transiently transfected by pCMV6-ENTRY KCNJ3 (RC205322). HEK293T cells were transfected with the pCMV6-ENTRY control (Left lane) or pCMV6-ENTRY KCNJ3 (Right lane) cDNA for 48 hrs and lysed. Equivalent amounts of cell lysates (5 µg per lane) were separated by SDS-PAGE and immunoblotted with anti-KCNJ3. 100 μL Log in to see 2 to 3 Days
$482.90
Details
Human Rabbit Un-conjugated IF, IHC, ELISA, WB Western blot analysis of extracts from RAW264.7 cells treated with Insulin 0.01U/ml 15', using GIRK1/KIR3.1/KCNJ3 (Phospho-Ser185) Antibody. The lane on the right is treated with the synthesized peptide. Immunohistochemistry analysis of paraffin-embedded human colon carcinoma, using GIRK1/KIR3.1/KCNJ3 (Phospho-Ser185) Antibody. The picture on the right is treated with the synthesized peptide. 100 μg Log in to see 2 to 3 Days
$302.50
Details

KCNJ3 Antibodies by Reactivity, Application, Clonality and Conjugate

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Top referenced anti-Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 Antibodies

  1. Mouse (Murine) Polyclonal KCNJ3 Primary Antibody for WB - ABIN1742154 : Jin, Lu: A novel high-affinity inhibitor for inward-rectifier K+ channels. in Biochemistry 1998 (PubMed)

  2. Dog (Canine) Polyclonal KCNJ3 Primary Antibody for IF (p), IHC (p) - ABIN719741 : Zhao, Xu, Yun, Zhao, Li, Gong, Yuan, Yan, Zhang, Ding, Wang, Zhang, Dong, Xiu, Yang, Liu, Xue, Li: Chronic obstructive sleep apnea causes atrial remodeling in canines: mechanisms and implications. in Basic research in cardiology 2014 (PubMed)

More Antibodies against Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 Interaction Partners

Human Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 (KCNJ3) interaction partners

  1. The findings of this study suggest that variations in KCNJ3 genes are associated with both mild and severe persistent breast pain after breast cancer surgery.

  2. For KCNJ3 rs7574878, individuals who were heterozygous or homozygous for the rare G allele (TT versus TG+ GG) had a 48% reduction in the odds of reporting preoperative breast pain.

  3. we show that Kir3.1, in the absence of trafficking partner subunits, can exit the endoplasmic reticulum (ER) and reach the Golgi (though not the plasma membrane)

  4. Conformational changes at the Gbetagamma/Kir3 interface were lost when Kir3.1 subunits were replaced.

  5. These data suggest that the KCNJ3 gene is genetically associated with schizophrenia in Asian populations and add further evidence to the "channelopathy theory of psychiatric illnesses".

  6. Kir3.1 channel is involved in the TLR4 (show TLR4 Antibodies)-mediated signal at an early event by facilitating the recruitment of TLR4 (show TLR4 Antibodies) into lipid raft.

  7. halothane (show RYR1 Antibodies) predominantly interferes with Gbetagamma-mediated Kir3 currents, such as those functioning during inhibitory synaptic activity

  8. The very high abundance of mRNA's encoding GIRK1 together with the presence of GIRK1 protein suggests a pathophysiological role in breast cancer

  9. We identified several known single nucleotide polymorphisms in KCNJ3 and KCNJ5 (show KCNJ5 Antibodies), but no mutations in either of the genes.

  10. glutamate (show GRIN1 Antibodies) residue at the C terminus regulates activity -- implications for Andersen Disease (inward rectifier potassium channel 2 (show KCNJ2 Antibodies); IRK2 (show KCNJ12 Antibodies))

Mouse (Murine) Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 (KCNJ3) interaction partners

  1. Kir (show GEM Antibodies) 3.1 channels are important for supraspinal antinociception and presynaptic GABA release inhibition by oxycodone in the femur bone cancer model

  2. GABA neurons in the ventral tegmental area express GIRK1 (and GIRK2 (show KCNJ6 Antibodies)) subunits.

  3. siRNA knock-down of NgR1 (show NEUROG1 Antibodies) resulted in a selective increase of GABAB R1 and GABAB R2 protein and an increase in GIRK1.

  4. Discontinuous and sometime opposing elements in Girk1 underlie the Girk1-dependent potentiation of receptor-dependent and receptor-independent heteromeric channel activity.

  5. Mechanism for functional dysregulation in the dorsal raphe follows tyrosine phosphorylation of repeated stress-activated Kir3.1 channels.

  6. NMR analyses of the Gbetagamma binding and conformational rearrangements of the cytoplasmic pore of G protein-activated inwardly rectifying potassium channel 1 (show KCNA5 Antibodies) (GIRK1).

  7. Agonist-induced localization of Gq-coupled receptors and G protein-gated inwardly rectifying K+ (GIRK) channels to caveolae determines receptor specificity of phosphatidylinositol 4,5-bisphosphate signaling

  8. The atrial potassium channel (show KCNAB2 Antibodies), I(KACH), ion channel gating is accelerated in atrial myocytes through the RGS6 (show RGS6 Antibodies)/Gbeta5 (show GNB5 Antibodies) complex.

  9. Dopamine neurons from Girk1 knock-out mice (and Girk2 (show KCNJ6 Antibodies)) exhibited elevated glutamatergic neurotransmission and increased synaptic levels of AMPA (show GRIA3 Antibodies) glutamate (show GRIN1 Antibodies) receptors.

  10. Data suggest HL-1 (show ASGR1 Antibodies) cells express GIRK1/4 and M2 muscarinic receptors and are a good model to study acetylcholine-activated potassium currents.

Potassium Inwardly-Rectifying Channel, Subfamily J, Member 3 (KCNJ3) Antigen Profile

Protein Summary

Potassium channels are present in most mammalian cells, where they participate in a wide range of physiologic responses. The protein encoded by this gene is an integral membrane protein and inward-rectifier type potassium channel. The encoded protein, which has a greater tendency to allow potassium to flow into a cell rather than out of a cell, is controlled by G-proteins and plays an important role in regulating heartbeat. It associates with three other G-protein-activated potassium channels to form a heteromultimeric pore-forming complex that also couples to neurotransmitter receptors in the brain and whereby channel activation can inhibit action potential firing by hyperpolarizing the plasma membrane. These multimeric G-protein-gated inwardly-rectifying potassium (GIRK) channels may play a role in the pathophysiology of epilepsy, addiction, Down's syndrome, ataxia, and Parkinson's disease. Alternative splicing results in multiple transcript variants encoding distinct proteins.

Gene names and symbols associated with KCNJ3

  • potassium inwardly-rectifying channel, subfamily J, member 3 (KCNJ3) antibody
  • potassium inwardly-rectifying channel, subfamily J, member 3 (Kcnj3) antibody
  • girk1 antibody
  • Kcnf3 antibody
  • KGA antibody
  • Kir3.1 antibody

Protein level used designations for KCNJ3

G protein-activated inward rectifier potassium channel 1 , GIRK-1 , inward rectifier K(+) channel Kir3.1 , inward rectifier K+ channel KIR3.1 , potassium channel, inwardly rectifying subfamily J member 3 , potassium inwardly-rectifying channel subfamily J member 3 splice variant 1e , potassium inwardly-rectifying channel J3 , GIRK1 , KGA , KGB1 , potassium channel inwarding rectifying channel subfamily J member 3 , potassium channel subunit Kir3.1 type 3 delta , inward rectifying potassium channel, Kir3.1

GENE ID SPECIES
3760 Homo sapiens
16519 Mus musculus
50599 Rattus norvegicus
100190980 Cavia porcellus
396369 Gallus gallus
488355 Canis lupus familiaris
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