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May have a role in the regulation of spermatogenesis.. Additionally we are shipping PD-1 Proteins (75) and PD-1 Kits (13) and many more products for this protein.
Showing 10 out of 579 products:
Human Polyclonal PD-1 Primary Antibody for EIA, WB - ABIN401428
Zhong, Bai, Gao, Strom, Rothstein: Suppression of expression and function of negative immune regulator PD-1 by certain pattern recognition and cytokine receptor signals associated with immune system danger. in International immunology 2004
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Human Polyclonal PD-1 Primary Antibody for EIA, IHC (p) - ABIN500457
Holling, Schooten, van Den Elsen: Function and regulation of MHC class II molecules in T-lymphocytes: of mice and men. in Human immunology 2004
Show all 4 references for ABIN500457
Human Monoclonal PD-1 Primary Antibody for EIA, WB - ABIN263912
Freeman, Long, Iwai, Bourque, Chernova, Nishimura, Fitz, Malenkovich, Okazaki, Byrne, Horton, Fouser, Carter, Ling, Bowman, Carreno, Collins, Wood, Honjo: Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation. in The Journal of experimental medicine 2000
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Human Polyclonal PD-1 Primary Antibody for ELISA, WB - ABIN185400
Nielsen, Laustrup, Voss, Junker, Husby, Lillevang: A putative regulatory polymorphism in PD-1 is associated with nephropathy in a population-based cohort of systemic lupus erythematosus patients. in Lupus 2004
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Mouse (Murine) Monoclonal PD-1 Primary Antibody for FACS - ABIN951271
Agata, Kawasaki, Nishimura, Ishida, Tsubata, Yagita, Honjo: Expression of the PD-1 antigen on the surface of stimulated mouse T and B lymphocytes. in International immunology 1997
Show all 2 references for ABIN951271
Mouse (Murine) Monoclonal PD-1 Primary Antibody for FACS - ABIN951269
Ansari, Salama, Chitnis, Smith, Yagita, Akiba, Yamazaki, Azuma, Iwai, Khoury, Auchincloss, Sayegh: The programmed death-1 (PD-1) pathway regulates autoimmune diabetes in nonobese diabetic (NOD) mice. in The Journal of experimental medicine 2003
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Study demonstrates that PD-1(+) TIMC and intratumoral PD-L1 (show CD274 Antibodies)(+) expression did not significantly impact tumor aggressiveness or clinical outcome in non-ccRCC specimens.
Immune surveillance is an important mechanism in prevention of cancer development and inhibition of growth of tumors and metastasis. Checkpoint pathways prevent immune response through multiple methods including CTLA-4 (show CTLA4 Antibodies) and PD-1/PD-L1 (show CD274 Antibodies) pathways.A variety of immune checkpoint inhibitors (PD-1/PD-L1 (show CD274 Antibodies) and CTLA-4 (show CTLA4 Antibodies) inhibitors) have been studied in recurrent and metastatic head and neck cancers with encouraging efficacy
n combination with anti-PD-1/PD-L1 (show CD274 Antibodies), the CTLA-4 (show CTLA4 Antibodies)-expanded T cells are prevented from being functionally inhibited when they enter into the tumor microenvironment.
The CTLA-4 (show CTLA4 Antibodies) receptor was one of the first checkpoint inhibitors to be targeted with limited exploration and efficacy in sarcoma.The PD-1/PD-L1 (show CD274 Antibodies) immunologic checkpoint pathway has demonstrated efficacy in many malignancies; studies are ongoing in sarcoma.
PD-1 expression is associated with a poor prognosis in gastric cancer after curative resection.
Data suggest that PD-1/PDCD1 (programmed cell death protein 1) and Tim-3/HAVCR2 (hepatitis A virus cellular receptor 2 (show HAVCR2 Antibodies)) operate within the functional immune-modulatory network to promote Th2-(type-2 helper T cell-)mediated immunity at the maternal-fetal interface.
High PD-1 expression is Correlated with Non-Small Cell Lung Cancer.
findings support the use of Tim-3 (show HAVCR2 Antibodies)-Tim-3L blockade together with PD-1-PD-L1 (show CD274 Antibodies) blockade to reverse tumor-induced T-cell exhaustion/dysfunction in patients with colorectal cancer.
Report increased expression of PD-1 in individuals that had recently been treated for tuberculosis, and a small increase in PD-1-expression in active TB disease.
PD-1 and CTLA-4 (show CTLA4 Antibodies) expression during the symptomatic phase was significantly higher in the T-cells of Acute Hepatitis A patients than in those of Acute Toxic Hepatitis patients or healthy controls.
Tim-3 (show HAVCR2 Antibodies)(+) PD-1(+) CD8 (show CD8A Antibodies)(+) T cells showed more evident properties associated with exhaustion than Tim-3 (show HAVCR2 Antibodies)(-) PD-1(+) CD8 (show CD8A Antibodies)(+) T cells.
that porcine islet-specific tolerance is dependent on PD-1, which could not be extended to skin grafts
Programmed cell death-1 engagement followed by zymosan stimulation might primarily attenuate the phosphorylation of tyrosine residue in Programmed cell death-1 receptor/ligand.
Study suggests that expression of the inhibitory receptors PD-1 and LAG-3 (show LAG3 Antibodies) on CD4 (show CD4 Antibodies)(+) T cells and their reduced IL-2 (show IL2 Antibodies) production are common characteristic features of Plasmodium infection.
these data suggest a scenario in which microglia are involved in the regulation of experimental autoimmune encephalomyelitis by suppressing Th1 (show HAND1 Antibodies)-cell differentiation via the PD-L1 (show CD274 Antibodies)-nitric oxide pathway.
Blockade of TGFbeta (show TGFB1 Antibodies) downregulated PD-1 and PD-L1 (show CD274 Antibodies) expression and precipitated graft rejection.
PD-1 regulates peripheral T-cell responses in both human and murine rheumatoid arthritis
LAG3 (show LAG3 Antibodies) and PD1 co-inhibitory molecules have roles in collaborating to limit CD8 (show CD8A Antibodies)+ T cell signaling and dampen antitumor immunity in a murine ovarian cancer model
Decidual NK, NKT (show CTSL1 Antibodies) and gamma/delta T cells showed increased PD-1 expression and reduced cytotoxic potential when compared to the periphery.
The endogenous PD-1/PD-L pathway does not limit acute experimental foreign antigen-induced circulating immune complex glomerulonephritis.
May have a role in the regulation of spermatogenesis.
programmed cell death 1
, programmed cell death protein 1
, protein PD-1
, programmed death 1
, spermatogenesis associated PD1
, spermatogenesis-associated protein 2
, spermatogenesis-associated protein PD1