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The protein encoded by PTPLA contains a characteristic catalytic motif of the protein tyrosine phosphatases (PTPs) family. Additionally we are shipping PTPLA Kits (4) and and many more products for this protein.
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Data reveal that HACD1 is a key regulator of a lipid-dependent muscle fibre growth mechanism.
IRAP (show IL1RN Antibodies) is more highly expressed in pro-inflammatory M1-activated macrophages.
IRAP (show IL1RN Antibodies) deficiency may lead to suppression of PAI-1 (show SERPINE1 Antibodies) expression in adipocytes and upregulation of UCP-1 (show UCP1 Antibodies)-mediated thermogenesis in brown adipose tissue and increased energy expenditure to prevent the development of obesity.
The current study demonstrates that deletion of the IRAP (show IL1RN Antibodies) gene protects the brain from ischemic damage
IRAP (show IL1RN Antibodies)-deficient mice are less sensitive to the development of acute pentylenetetrazol-induced seizures, demonstrating that IRAP (show IL1RN Antibodies) may be involved in generalized seizure generation.
requirement of IRAP (show IL1RN Antibodies) in cross-presentation extends to steady-state conventional dendritic cells (cDCs);data suggest increased recruitment of an IRAP (show IL1RN Antibodies)(+)/Rab14 (show RAB14 Antibodies)(+) compartment to Ag-containing vesicles contributes to cross-presentation efficacy of CD8 (show CD8A Antibodies)(+)cDCs
Ptpla regulates myogenesis through Myog (show MYOG Antibodies).
These findings suggest that the IRAP (show IL1RN Antibodies) binding protein, vimentin (show VIM Antibodies), plays an important role in retention of GLUT4 (show SLC2A4 Antibodies) storage vesicles.
IRAP (show IL1RN Antibodies) functions in GLUT4 (show SLC2A4 Antibodies) sorting from endosomes to GLUT4 (show SLC2A4 Antibodies)-specialized compartments.
study shows the high-affinity binding site for angiotensin IV is absent in IRAP (show IL1RN Antibodies) knockout mice brain sections in parallel with loss of IRAP (show IL1RN Antibodies) immunostaining, providing irrefutable proof that IRAP (show IL1RN Antibodies) is the specific high-affinity binding site for AT(4) ligands
These data indicate that HACD1 is necessary for muscle function.
PTPLA-CAP expression was limited to cementum cells; promoted gingival fibroblast attachment. PTPLA-CAP is splice variant of PTPLA, and that, in the periodontium, cementum and cementum cells express this variant.
HACD1 is 3-hydroxyacyl-CoA dehydratase (show PTPLAD2 Antibodies) involved in elongation of very long-chain fatty acids. HACD1 (K64Q) exhibits normal enzyme activity, intracellular localization and interaction with other VLCFA enzymes, with no negative effect on VLCFA elongation
PTPLA SNPs show genotypic association with Alzheimer disease.
The protein encoded by this gene contains a characteristic catalytic motif of the protein tyrosine phosphatases (PTPs) family. The PTP motif of this protein has the highly conserved arginine residue replaced by a proline residue\; thus it may represent a distinct class of PTPs. Members of the PTP family are known to be signaling molecules that regulate a variety of cellular processes. This gene was preferentially expressed in both adult and fetal heart. A much lower expression level was detected in skeletal and smooth muscle tissues, and no expression was observed in other tissues. The tissue specific expression in the developing and adult heart suggests a role in regulating cardiac development and differentiation.
, cystinyl aminopeptidase
, leucyl-cystinyl aminopeptidase
, 3-hydroxyacyl-CoA dehydratase 1
, cementum attachment protein
, protein-tyrosine phosphatase-like member A
, very-long-chain (3R)-3-hydroxyacyl-[acyl-carrier protein] dehydratase 1
, Protein-tyrosine phosphatase-like member A
, protein tyrosine phosphatase-like, member a
, protein tyrosine phosphatase-like protein PTPLA