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Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. Additionally we are shipping Retinoic Acid Receptor Responder (Tazarotene Induced) 3 Antibodies (53) and Retinoic Acid Receptor Responder (Tazarotene Induced) 3 Kits (1) and many more products for this protein.
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Letter: Interleukin-22 (show IL22 Proteins) inhibits tazarotene-induced gene 3 expression in keratinocytes via MAPK (show MAPK1 Proteins)-ERK1/2 and JAK2 (show JAK2 Proteins)/STAT3 (show STAT3 Proteins) signaling.
TRIM25 (show TRIM25 Proteins) plays an additional role in RIG-I (show DDX58 Proteins)/MDA5 (show IFIH1 Proteins) signaling other than RIG-I (show DDX58 Proteins) ubiquitination via activation of NF-kappaB (show NFKB1 Proteins).
These observations highlight the importance of RIG-I (show DDX58 Proteins) signaling in anti-HIV innate immunity in macrophages, which may be beneficial for the treatment of HIV and aid in the understanding of the neuropathogenesis of HAND.
Gal1 (show LGALS1 Proteins) and gal3 (show LGALS3 Proteins) regulate the inflammatory response in airway epithelial cells exposed to microbial neuraminidase (show NEU Proteins) by modulating the expression of SOCS1 (show SOCS1 Proteins) and RIG1.
findings provide a new insight into the molecular link between p53 (show TP53 Proteins), protein acylation and Wnt (show WNT2 Proteins)/beta-catenin (show CTNNB1 Proteins) signaling whereby RARRES3 plays a pivotal role in modulating the acylation status of signaling proteins.
During Crimean-Congo hemorrhagic fever virus infection, RIG-I (show DDX58 Proteins) mediated a type I interferon (show IFNA Proteins) response via MAVS (show MAVS Proteins).
the flexible main loop of H-REV107, but not that of TIG3, is critical for its NTD to modulate its CTD in inducing cell death.
Viral pseudo-enzymes activate RIG-I (show DDX58 Proteins) via deamidation to evade cytokine production.
critical for the initiation of IRF-3 (show IRF3 Proteins) phosphorylation, dimerization and downstream gene expression during antiviral innate immune response
RARRES3 downregulation engages metastasis-initiating capabilities by facilitating adhesion of the tumor cells to the lung parenchyma.
Retinoids exert biologic effects such as potent growth inhibitory and cell differentiation activities and are used in the treatment of hyperproliferative dermatological diseases. These effects are mediated by specific nuclear receptor proteins that are members of the steroid and thyroid hormone receptor superfamily of transcriptional regulators. RARRES1, RARRES2, and RARRES3 are genes whose expression is upregulated by the synthetic retinoid tazarotene. RARRES3 is thought act as a tumor suppressor or growth regulator.
retinoic acid receptor responder (tazarotene induced) 3
, retinoic acid receptor responder protein 3
, HRAS-like suppressor 4
, RAR-responsive protein TIG3
, retinoic acid-inducible gene 1
, retinoid-inducible gene 1 protein
, tazarotene-induced gene 3 protein