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The protein encoded by RECK is a cysteine-rich, extracellular protein with protease inhibitor-like domains whose expression is suppressed strongly in many tumors and cells transformed by various kinds of oncogenes. Additionally we are shipping RECK Antibodies (82) and RECK Kits (8) and many more products for this protein.
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the RECK gene polymorphism influences molecular carcinogenesis and clinic pathological features of hepatocellular carcinoma within the Egyptian population
Reversion-inducing, cysteine-rich protein (show SPARC Proteins) with kazal motifs (RECK) was identified as the direct and functional target of miR (show MLXIP Proteins)-92b in osteosarcoma
RECK expression in uterine leiomyoma is negatively regulated by miR (show MLXIP Proteins)-15b.
RECK could regulate the expressions of MMP-2 (show MMP2 Proteins), 9 and MT1-MMP (show MMP14 Proteins) as a cell surface-signaling molecule. Authors propose that RECK may play an important role in regulating MMPs in the ECM (show MMRN1 Proteins) degradation of periodontal diseases.
Low expression of RECK is associated with oral cancer.
RECK Gene Promoter rs10814325 Polymorphism is associated with metastasis in Hepatocellular Carcinoma
the aim of this study was to analyze the effect of RECK gene rs 11788747 single nucleotide polymorphism (SNP) on hepatocellular carcinoma (HCC (show FAM126A Proteins)) susceptibility.
Findings suggest that RECK transcript variants might have opposite roles in GBM biology and the ratio of their expression levels may be informative for the prognostic outcome of GBM patients.
miR (show MLXIP Proteins)-21 has a role in upregulating PTEN, RECK and PDCD4 (show PDCD4 Proteins) in glioma
RECK is a regulator of hMSC functions suggesting that modulation of RECK may improve the development of hMSC-based therapeutical approaches in regenerative medicine.
experiments indicate that Reck and Gpr124 are part of the cell surface protein complex that transduces Wnt7a- and Wnt7b-specific signals in mammalian CNS Epithelial Cells to promote angiogenesis and regulate the BBB.
These findings demonstrate the importance of appropriate cell-cell interactions and ECM (show MMRN1 Proteins) maintenance for angiogenesis and the involvement of Reck as a critical regulator of these events.
RECK-mediated beta1-Integrin regulation by TGF-beta1 (show TGFB1 Proteins) is critical for wound contraction in mice.
MiR (show MLXIP Proteins)-21 modulated the osteoporosis by targeting RECK.
microRNA-200b and microRNA-200c promote colorectal cancer cell proliferation via targeting RECK.
The RECK silencing-EGFR (show EGFR Proteins)-HIF-2alpha (show EPAS1 Proteins) axis might be a key molecular mechanism to induce hyperplastic phenotype of epithelial cells.
Angiotensin II suppresses RECK, but induces matrix metalloproteinases both in vivo and in vitro.
therapeutic concentrations of ASA (show ARSA Proteins) inhibited IL-18 (show IL18 Proteins)-induced H(2)O(2) generation, MMP9 (show MMP9 Proteins) activation, RECK suppression, and CF migration.
these data suggest that RECK is a novel transcriptional target of FXR (show NR1H4 Proteins) in mouse liver, and provide clues to better understanding the function of FXR (show NR1H4 Proteins) in liver.
RECK expression in the mouse uterus is steroidally regulated and within endometrial epithelial and stromal cells, RECK regulates MMP9 (show MMP9 Proteins), but not MMP2 (show MMP2 Proteins) activity.
The protein encoded by this gene is a cysteine-rich, extracellular protein with protease inhibitor-like domains whose expression is suppressed strongly in many tumors and cells transformed by various kinds of oncogenes. In normal cells, this membrane-anchored glycoprotein may serve as a negative regulator for matrix metalloproteinase-9, a key enzyme involved in tumor invasion and metastasis.
membrane-anchored glycoprotein (metastasis and invasion)
, reversion-inducing cysteine-rich protein with Kazal motifs
, suppression of tumorigenicity 15 (reversion-inducing-cysteine-rich protein with kazal motifs)
, suppression of tumorigenicity 5 (reversion-inducing-cysteine-rich protein with kazal motifs)
, suppressor of tumorigenicity 15 protein
, Reversion-inducing cysteine-rich protein with Kazal motifs precursor (mRECK)
, membrane-anchored glycoprotein RECK
, suppression of tumorigenicity 15
, reversion-inducing-cysteine-rich protein with Kasal motifs