Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
The protein encoded by SLURP1 is a member of the Ly6/uPAR family but lacks a GPI-anchoring signal sequence. Additionally we are shipping SLURP1 Proteins (7) and SLURP1 Kits (5) and many more products for this protein.
Showing 10 out of 35 products:
Chinese Hamster Polyclonal SLURP1 Primary Antibody for IHC, IHC (p) - ABIN4354634
Dijk, Beigneux, Larsson, Bensadoun, Young, Kersten: Angiopoietin-like 4 promotes intracellular degradation of lipoprotein lipase in adipocytes. in Journal of lipid research 2016
Human Polyclonal SLURP1 Primary Antibody for ELISA, WB - ABIN566094
Tjiu, Lin, Wu, Cheng, Chiu, Thong, Chiang, Yang, Jee: SLURP1 mutation-impaired T-cell activation in a family with mal de Meleda. in The British journal of dermatology 2010
Human Monoclonal SLURP1 Primary Antibody for IP, ELISA - ABIN566095
Narumoto, Horiguchi, Horiguchi, Moriwaki, Takano-Ohmuro, Shoji, Misawa, Yamashita, Nagase, Kawashima, Yamashita et al.: Down-regulation of secreted lymphocyte antigen-6/urokinase-type plasminogen activator receptor-related peptide-1 (SLURP-1), an endogenous allosteric alpha7 nicotinic acetylcholine receptor modulator, ... in Biochemical and biophysical research communications 2010
novel splice site mutation c.58+5G>T in mal (show MAL Antibodies) de Meleda in India
Results of this study suggest understand Mal (show MAL Antibodies) de Meleda, it will be important to identify proteins that interact with mutatated SLURP1. In any such studies, it will be important to assess binding of mutant SLURP1 proteins that cause Mal (show MAL Antibodies) de Meleda.
To our knowledge, the present study is the fi rst (show SLC22A12 Antibodies) report on molecular investigation of Mal (show MAL Antibodies) de Meleda from Libya.
This supports the hypothesis that the antiproliferative activity of SLURP-1 is related to 'metabotropic' signaling pathway through alpha7-nAChR (show CHRNA7 Antibodies), that activates intracellular signaling cascades without opening the receptor channel.
SLURP1 participates in pathophysiology of psoriasis by regulating keratinocyte proliferation and differentiation, and by suppressing the growth of S. aureus
Palmoplantar keratoderma of the Gamborg-Nielsen type is caused by mutations in the SLURP1 gene and represents a variant of Mal (show MAL Antibodies) de Meleda.
rSLURP-1 decreased production of TNFalpha (show TNF Antibodies) by T-cells, downregulated IL-1 beta (show IL1B Antibodies) and IL-6 (show IL6 Antibodies) secretion by macrophages, and moderately upregulated IL-10 (show IL10 Antibodies) production by both types of immunocytes
This report further expands the spectrum of clinical phenotypes associated with mutations in SLURP1 in the Mediterranean population.
mutations in SLURP1 as a cause for mal (show MAL Antibodies) de Meleda and suggest an ancient founder effect for p.W15R in the western European population.
Those findings suggested that SLURP-1 and stimulus through alpha7 nicotinic ACh (show FGFR3 Antibodies) receptors actively controlled asthmatic condition by stimulating ciliary beating and also by suppressing airway inflammation.
SLURP1 nonsense mutation is responsible for the pathogenesis of palmoplantar keratoderma.
These data describe the influence of age, sex, genetic background, and ocular surface health on mouse corneal expression of Slurp1, establish the baseline for human tear SLURP1 expression.
Slurp1 in mice has a role in neuromuscular and metabolic phenotypes in addition to its role in palmoplantar keratoderma
Those findings suggest that diminished expression of SLURP-1 in asthma attenuates its negative regulation of airway inflammation, and that perhaps changes in SLURP-1 expression could serve as a marker of airway damage in asthma.
The protein encoded by this gene is a member of the Ly6/uPAR family but lacks a GPI-anchoring signal sequence. It is thought that this secreted protein contains antitumor activity. Mutations in this gene have been associated with Mal de Meleda, a rare autosomal recessive skin disorder. This gene maps to the same chromosomal region as several members of the Ly6/uPAR family of glycoprotein receptors.
secreted Ly-6/uPAR-related protein 1
, secreted LY6/PLAUR domain containing 1
, ARS component B
, ARS(component B)-81/S
, anti-neoplastic urinary protein
, lymphocyte antigen 6-like secreted
, secreted Ly6/uPAR related protein 1
, Ars component B
, secreted Ly-6/uPAR related protein 1