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The transmembrane semaphorin SEMA6A is expressed in developing neural tissue and is required for proper development of the thalamocortical projection (Leighton et al., 2001 [PubMed 11242070]).[supplied by OMIM, Feb 2011].. Additionally we are shipping SEMA6A Proteins (8) and many more products for this protein.
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Human Polyclonal SEMA6A Primary Antibody for ELISA - ABIN1998407
Schmutz, Martin, Terry, Couronne, Grimwood, Lowry, Gordon, Scott, Xie, Huang, Hellsten, Tran-Gyamfi, She, Prabhakar, Aerts, Altherr, Bajorek, Black, Branscomb, Caoile, Challacombe, Chan, Denys et al.: The DNA sequence and comparative analysis of human chromosome 5. ... in Nature 2004
Show all 3 references for ABIN1998407
Human Polyclonal SEMA6A Primary Antibody for EIA, IHC (p) - ABIN357889
Prislei, Mozzetti, Filippetti, De Donato, Raspaglio, Cicchillitti, Scambia, Ferlini: From plasma membrane to cytoskeleton: a novel function for semaphorin 6A. in Molecular cancer therapeutics 2008
Show all 2 references for ABIN357889
Human Polyclonal SEMA6A Primary Antibody for WB - ABIN650714
Landers, Melki, Meininger, Glass, van den Berg, van Es, Sapp, van Vught, McKenna-Yasek, Blauw, Cho, Polak, Shi, Wills, Broom, Ticozzi, Silani, Ozoguz, Rodriguez-Leyva, Veldink, Ivinson, Saris, Hosler, Barnes-Nessa, Couture, Wokke, Kwiatkowski, Ophoff, Cro: Reduced expression of the Kinesin-Associated Protein 3 (KIFAP3) gene increases survival in sporadic amyotrophic lateral sclerosis. in Proceedings of the National Academy of Sciences of the United States of America 2009
Human Polyclonal SEMA6A Primary Antibody for IHC (p), WB - ABIN389214
Gautier, de Saint-Vis, Sénéchal, Pin, Bates, Caux, Geissmann, Garrone: The class 6 semaphorin SEMA6A is induced by interferon-gamma and defines an activation status of langerhans cells observed in pathological situations. in The American journal of pathology 2006
Sema6a and Plxna2 (show Plxna2 Antibodies) have roles in promoting eye vesicle cohesion through mediating spatially regulated repulsion within the developing zebrafish eye
our findings indicated that SEMA6A may be a potential prognostic biomarker in the treatment of Glioblastoma multiforme
Data suggest that Sema6A and Mical1 (show MICAL1 Antibodies) may represent new potential therapeutic targets in BRAFV600E melanoma.
We identified the SEMA6A and HLA-DP (show HLA-DPB1 Antibodies) loci as significant contributors to risk for granulomatosis with polyangiitis.
Data show that miR (show MLXIP Antibodies)-27a/b regulated the expression of the angiogenesis inhibitor semaphorin 6A (SEMA6A) in vitro and in vivo and targeted the 3'-untranslated region of SEMA6A.
Strong expression of SEMA6A is associated with langerhans cells of histiocytosis or dermatopathic lymphadenitis
NANOG (show NANOG Antibodies)-, SOX2 (show SOX2 Antibodies)-, and POU5F1 (OCT3/OCT4 (show POU5F1 Antibodies))-binding sites within intron 1 of the SEMA6A gene
findings suggest for SEMA6A a novel function in the cytoskeleton and a role in modulating tubulin (show TUBB Antibodies) isotype composition and microtubule dynamics
findings reveal a unique function of Nrf2 (show NFE2L2 Antibodies) in reprogramming ischemic tissue toward neurovascular repair via Sema6A regulation, providing a potential therapeutic strategy for ischemic retinal and CNS diseases
Tangential migration is selectively altered in cerebellar neurons of Sema6A knockouts.
semaphorin 6A is expressed in On starburst amacrine cell.
Sema6A expressed in oligodendrocyte precursor cells is involved in their autonomous migration through ligand-receptor interaction with Plexin-A4 (show PLXNA4 Antibodies) expressed in surrounding cells.
The results of this study suggested that Sema6A might have a role in myelination by controlling oligodendrocyte differentiation.
Adult Sema6A-null mice exhibit reduced tumor, matrigel, and choroidal angiogenesis compared with controls. Here we show that it also regulates vascular development and adult angiogenesis.
null mutations in Sema6A or PlexinA4 (PlexA4 (show PLXNA4 Antibodies)) exhibit a pronounced defect in OPL (show ZIC1 Antibodies) stratification of horizontal cell axons without any apparent deficits in bipolar cell dendrite or photoreceptor axon targeting.
Mice with mutations in Sema6A or the interacting genes may thus represent a highly informative model for how neurodevelopmental defects can lead to anatomical dysconnectivity.
the transmembrane semaphorin Sema6A signals through its receptor PlexinA4 (PlexA4 (show PLXNA4 Antibodies)) to control lamina-specific neuronal stratification in the mouse retina
crystal structures of cognate complexes of the semaphorin-binding regions of plexins B1 and A2 with semaphorin ectodomains (human PLXNB1 (show PLXNB1 Antibodies)(1-2)-SEMA4D (show SEMA4D Antibodies)(ecto (show TRIM33 Antibodies)) and murine PlxnA2 (show Plxna2 Antibodies)(1-4)-Sema6A(ecto (show TRIM33 Antibodies))), plus unliganded structures of PlxnA2 (show Plxna2 Antibodies)(1-4) and Sema6A(ecto (show TRIM33 Antibodies))
The transmembrane semaphorin SEMA6A is expressed in developing neural tissue and is required for proper development of the thalamocortical projection (Leighton et al., 2001
, semaphorin 6A1
, sema domain, transmembrane domain (TM), and cytoplasmic domain, (semaphorin) 6A
, sema VIa
, semaphorin 6A-1
, semaphorin VIA
, Mutant line 997
, sema Q
, sema VIA
, semaphorin Q