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The protein encoded by SEMA7A binds to cell surfaces through a glycosylphosphatidylinositol (GPI) linkage. Additionally we are shipping SEMA7A Kits (42) and SEMA7A Proteins (7) and many more products for this protein.
Showing 10 out of 90 products:
Human Monoclonal SEMA7A Primary Antibody for FACS - ABIN93946
Suzuki, Okuno, Yamamoto, Pasterkamp, Takegahara, Takamatsu, Kitao, Takagi, Rennert, Kolodkin, Kumanogoh, Kikutani: Semaphorin 7A initiates T-cell-mediated inflammatory responses through alpha1beta1 integrin. in Nature 2007
Show all 6 references for ABIN93946
Human Monoclonal SEMA7A Primary Antibody for FACS, IP - ABIN110773
Delorme, Saltel, Bonnelye, Jurdic, Machuca-Gayet: Expression and function of semaphorin 7A in bone cells. in Biology of the cell / under the auspices of the European Cell Biology Organization 2005
Show all 5 references for ABIN110773
Human Polyclonal SEMA7A Primary Antibody for WB - ABIN1881787
Yamada, Kubo, Takeshita, Harashima, Kawano, Mine, Sagawa, Sugamura, Itoh: Molecular cloning of a glycosylphosphatidylinositol-anchored molecule CDw108. in Journal of immunology (Baltimore, Md. : 1950) 1999
Show all 5 references for ABIN1881787
Human Monoclonal SEMA7A Primary Antibody for BR, FACS - ABIN2663992
Sato, Takahashi: Molecular cloning and expression of murine homologue of semaphorin K1 gene. in Biochimica et biophysica acta 1999
Show all 4 references for ABIN2663992
SEMA7A plays a critical role in IgE-mediated neutrophilic airway inflammation
SEMA7A has a role in the development of lung injury
Data suggest that sema7A is involved in the pathogenesis of experimental autoimmune encephalomyelitis, in the modulation of the immune response and in the neurodegeneration that take place in the CNS.
This study demonstrates that Sema7A controls the assembly of actin-based protrusions that drive Dendritic cell migration in response to CCL21 (show CCL21 Antibodies).
Sem7a and its receptors regulate Chi3l1 (show CHI3L1 Antibodies) in pulmonary melanoma metastasis
These data identify Sema7A as a regulator of thalamocortical and local circuit development in layer 4 and provide a molecular handle that can be used to explore the coordinated generation of excitatory and inhibitory cortical circuits.
these results suggest that Sema3A (show SEMA3A Antibodies) and Sema7A have opposite effects and are involved in different stages of synapse elimination.
SEMA7A was expressed in the liver and was increased in the course of liver fibrosis, both in mice and in humans.
Oncogenic Ras may play a role in epithelial mesenchymal transition via the ERF and regulation of Semaphorin-7a.
We report here the expression and induction of semaphorin 7A (SEMA7A) on endothelium through hypoxia-inducible factor 1alpha during hypoxia.
Sema3A (show SEMA3A Antibodies) and sema7A expression correlated with the inflammatory activity of the multiple sclerosis (MS) lesions, suggesting their involvement in the immunological process that takes place in MS.
These results suggest that SEMA7a plays a role as a CSF (show CSF2 Antibodies) biomarkers associated with the conversion to clinically definite multiple sclerosis in clinically isolated syndromes patients
heterozygous missense variants in SEMA3A (show SEMA3A Antibodies) and SEMA7A may modify the phenotype of Kallmann syndrome but most likely are not alone sufficient to cause the disorder
Semaphorin 7A is a potentially important modulator of eosinophil profibrotic functions in the airway remodeling of patients with chronic asthma.
two MTRAP monomers interact via their tandem TSR (show CFL1 Antibodies) domains with the Sema (show SEMA3B Antibodies) domains of a Semaphorin-7A homodimer
Semaphorin 7A protein variants differentially regulate T-cell activity.
Sema7A markedly reduces the production rates of megakaryocytes and platelets from CD34 (show CD34 Antibodies)(+) progenitor cells.
The protein encoded by this gene binds to cell surfaces through a glycosylphosphatidylinositol (GPI) linkage. The encoded glycoprotein is found on activated lymphocytes and erythrocytes. This protein may be involved in immunomodulatory and neuronal processes. Defects in this gene can result in loss of bone mineral density (BMD). Three transcript variants encoding different isoforms have been found for this gene.
, Semaphorin K1
, sema K1
, sema L
, sema domain, immunoglobulin domain (Ig), and GPI membrane anchor, (semaphorin) 7A
, JMH blood group antigen
, John Milton Hagen blood group H-Sema K1
, john-Milton-Hargen human blood group Ag
, sema domain, immunoglobulin domain (Ig), and GPI membrane anchor, (semaphorin) 7A (JMH blood group)
, sema domain, immunoglobulin domain (Ig), and GPI membrane anchor, 7A
, semaphorin K1
, semaphorin L
, semaphorin 7A, GPI membrane anchor (John Milton Hagen blood group) isoform 1
, semaphorin 7A, GPI membrane anchor (John Milton Hagen blood group) isoform 2