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SLC2A9 encodes a member of the SLC2A facilitative glucose transporter family. Additionally we are shipping and SLC2A9 Kits (1) and many more products for this protein.
Showing 10 out of 91 products:
Mouse (Murine) Polyclonal SLC2A9 Primary Antibody for WB - ABIN540293
Le, Shafiu, Mu, Johnson: SLC2A9--a fructose transporter identified as a novel uric acid transporter. in Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2008
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Human Polyclonal SLC2A9 Primary Antibody for WB - ABIN540295
Anzai, Ichida, Jutabha, Kimura, Babu, Jin, Srivastava, Kitamura, Hisatome, Endou, Sakurai: Plasma urate level is directly regulated by a voltage-driven urate efflux transporter URATv1 (SLC2A9) in humans. in The Journal of biological chemistry 2008
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Cow (Bovine) Polyclonal SLC2A9 Primary Antibody for WB - ABIN2781547
Gaccioli, White, Capobianco, Powell, Jawerbaum, Jansson: Maternal overweight induced by a diet with high content of saturated fat activates placental mTOR and eIF2alpha signaling and increases fetal growth in rats. in Biology of reproduction 2013
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Human Polyclonal SLC2A9 Primary Antibody for ICC, IF - ABIN316364
Tomioka, Tamura, Takada, Shibata, Suzuki, Uchida, Hosoyamada: Immunohistochemical and in situ hybridization study of urate transporters GLUT9/URATv1, ABCG2, and URAT1 in the murine brain. in Fluids and barriers of the CNS 2016
Human Polyclonal SLC2A9 Primary Antibody for IHC (p), WB - ABIN438433
Roma, Ovadje, Steckle, Nicoletti, Saleem, Pandey: Selective Induction of Apoptosis by Azadarichta indica Leaf Extract by Targeting Oxidative Vulnerabilities in Human Cancer Cells. in Journal of pharmacy & pharmaceutical sciences : a publication of the Canadian Society for Pharmaceutical Sciences, Société canadienne des sciences pharmaceutiques 2015
In liver-specific Glut9 knockout mice, inosine-induced hyperuricemia was not associated with hypertension despite partial renal deficiency.
intestinal uric acid transporter SLC2A9 has a role in early-onset metabolic syndrome in a mouse model
blocking GLUT9 expression during preimplantation development (show MTA2 Antibodies) had no effect on glucose transport or apoptosis but transfer of these embryos resulted in increased pregnancy loss, suggesting that GLUT9 is critical for early preimplantation development (show MTA2 Antibodies).
Characterize in more detail the transport properties of mouse GLUT9 and provide evidence that it functions as a urate uniporter.
Glut9 is a major regulator of urate homeostasis and its genetic inactivation induces hyperuricosuria and urate nephropathy.
This meta-analysis shows that the rs12510549, rs16890979, and rs1014290 polymorphisms of SLC2A9 protect against the development of gout in Caucasians and/or Asians.
The results of the study confirmed the presence of GLUT-1 (show SLC2A1 Antibodies), GLUT-4 (show SLC2A4 Antibodies) and GLUT-9 (show SLC2A6 Antibodies) proteins in the trophoblast from both, uncomplicated and diabetic pregnancies. In addition, insulin (show INS Antibodies) therapy may increase placental expression of GLUT-4 (show SLC2A4 Antibodies) and GLUT-9 (show SLC2A6 Antibodies), and partially GLUT-1 (show SLC2A1 Antibodies), in women with pregestational and gestational diabetes mellitus.
Meta-analysis. This study demonstrated that the genetic susceptibility for gout is associated with the SLC2A9 gene polymorphisms. Four of them except for the rs3733591 are protective SNPs in Caucasians, and rs16890979 and rs1014290 are protective SNPs in both Caucasians and Asians, while rs3733591 may be susceptibility SNP in Asians.
Each A-allele of the SLC2A9 rs7442295 was associated with 9% higher plasma urate and hazard ratios of 1.07 (1.01-1.14) for cancer incidence and 1.07 (1.02-1.13) for all-cause mortality.
The present proof-of-principle paper demonstrates that the multilocus profiles of ABCG2 (show ABCG2 Antibodies), SLC2A9 and SLC22A12 (show SLC22A12 Antibodies) increase susceptibility to asymptomatic hyperuricaemia, gout and tophi.
Immunostaining and highly-sensitive in situ hybridization was used to assess the distribution of UA transporters: GLUT9/URATv1, ABCG2 (show ABCG2 Antibodies), and URAT1 (show SLC22A12 Antibodies). Immunostaining for GLUT9 (show SLC2A6 Antibodies) was observed in ependymal cells, neurons, and brain capillaries. Immunostaining for ABCG2 (show ABCG2 Antibodies) was observed in the choroid plexus epithelium and brain capillaries, but not in ependymal cells. These results were validated by in situ hybridization.
The first genome-wide association study for serum uric acid level in Indians revealed association of SLC2A9, SLC22A11 (show SLC22A11 Antibodies) and ABCG2 (show ABCG2 Antibodies) gene variants at genome wide significance level in Type 2 diabetes patients.
the exon 9 of SLC2A9 gene 109C/T polymorphism is not associated with HUA and diabetes
Demonstrate a significant association between rs2231142 in the ABCG2 (show ABCG2 Antibodies) gene and gout and identified novel SNPs, c.881A>G and c.1002+78G>A, in the SLC2A9 gene that may be associated with gout in a Korean population.
GLUT9 (show SLC2A6 Antibodies) gene polymorphism predicts higher risk for cardiovascular diseases in patient diagnosed with hyperuricemia.
This gene encodes a member of the SLC2A facilitative glucose transporter family. Members of this family play a significant role in maintaining glucose homeostasis. The encoded protein may play a role in the development and survival of chondrocytes in cartilage matrices. Two transcript variants encoding distinct isoforms have been identified for this gene.
solute carrier family 2 (facilitated glucose transporter), member 9
, solute carrier family 2, member 9 protein
, Solute carrier family 2, facilitated glucose transporter member 9
, glucose transporter type 9
, human glucose transporter-like protein-9
, solute carrier family 2, facilitated glucose transporter member 9
, urate voltage-driven efflux transporter 1
, facilitated glucose transporter, type 9
, solute carrier family 2, member 9