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The protein encoded by SLC22A11 is involved in the sodium-independent transport and excretion of organic anions, some of which are potentially toxic. Additionally we are shipping SLC22A11 Proteins (4) and many more products for this protein.
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Dog (Canine) Polyclonal SLC22A11 Primary Antibody for WB - ABIN2781664
Carballo, Johnson, Sedgwick, Cha: Phosphorylation of the axial element protein Hop1 by Mec1/Tel1 ensures meiotic interhomolog recombination. in Cell 2008
The first genome-wide association study for serum uric acid level in Indians revealed association of SLC2A9 (show SLC2A9 Antibodies), SLC22A11 and ABCG2 (show ABCG2 Antibodies) gene variants at genome wide significance level in Type 2 diabetes patients.
The regulation of hOAT4 (show SLC22A9 Antibodies) activity was mediated by sgk2 (show SGK2 Antibodies) acting through Nedd4-2 (show NEDD4L Antibodies).
SLC22A11 at the basal plasma membrane of human placental syncytiotrophoblasts plays a predominant role in the uptake of 16alpha-OH DHEAS (show SULT2A1 Antibodies) for placental estriol synthesis.
Our analysis provides evidence for multiple ancestral-specific effects across the SLC22A11/SLC22A12 (show SLC22A12 Antibodies) locus that presumably influence the activity of OAT4 (show SLC22A9 Antibodies) and URAT1 (show SLC22A12 Antibodies) and risk of gout.
When investigating the genes separately, SLC22A11 and SLC2A9 (show SLC2A9 Antibodies) showed a significant interaction, consistent with the former encoding an organic anion/dicarboxylate exchanger, which mediates diuretic transport in the kidney.
Genetic variants of human organic anion transporter 4 (show SLC22A9 Antibodies) demonstrate altered transport of endogenous substrates.
The down-regulation of hOAT4 (show SLC22A9 Antibodies) activity by activation of protein kinase C (show PKC Antibodies) and the up-regulation of hOAT4 (show SLC22A9 Antibodies) activity by NHERF-1 (show SLC9A3R1 Antibodies) are mediated through alteration of hOAT4 (show SLC22A9 Antibodies) internalization.
Glycosylation serves as a means to specifically regulate hOAT4 (show SLC22A9 Antibodies) function in vivo.
hOAT4 (show SLC22A9 Antibodies) is the long-postulated, low-affinity apical urate anion exchanger that facilitates hydrochlorothiazide-associated hyperuricemia.
The interaction of PDZ (show INADL Antibodies) proteins with hOAT4 (show SLC22A9 Antibodies) may be cell-specific. In placenta, a different set of interacting proteins from PDZK1 (show PDZK1 Antibodies) and NHERF1 (show SLC9A3R1 Antibodies) may be required to modulate hOAT4 (show SLC22A9 Antibodies) activity.
The protein encoded by this gene is involved in the sodium-independent transport and excretion of organic anions, some of which are potentially toxic. The encoded protein is an integral membrane protein and is found mainly in the kidney and in the placenta, where it may act to prevent potentially harmful organic anions from reaching the fetus.
solute carrier family 22 (organic anion/urate transporter), member 11
, solute carrier family 22 member 11
, solute carrier family 22 (organic anion/cation transporter), member 11
, organic anion transporter 4