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The uptake of nucleosides by transporters, such as SLC29A2, is essential for nucleotide synthesis by salvage pathways in cells that lack de novo biosynthetic pathways. Additionally we are shipping SLC29A2 Antibodies (40) and SLC29A2 Proteins (4) and many more products for this protein.
two novel functional splice variants of equilibrative nucleoside transporter 2 (ENT2), which are present at the nuclear envelope, were identified.
Results showed that both SLC29A1 (show SLC29A1 ELISA Kits) and SLC29A2 were expressed at lower levels in colon cancer cell lines originating from metastatic sites than from primary sites.
Direct evidence for apical localization of ENT1 (show SLC29A1 ELISA Kits) and integral expression of ENT2 in intestinal epithelial cells.
Data suggest that SLC29A2 is localized to apical membrane of adult Sertoli cells. In contrast, SLC29A1 (show SLC29A1 ELISA Kits) is located on basolateral membrane of Sertoli cells; SLC29A1 (show SLC29A1 ELISA Kits) is primarily responsible for basolateral nucleoside uptake into Sertoli cells.
Data show that ENT1 (show SLC29A1 ELISA Kits), ENT2, ENT4 (show SLC29A4 ELISA Kits) and CNT3 (show SLC28A3 ELISA Kits) protein was detected on ovarian carcinoma cells in all effusions, with expression observed in 1-95% of tumor cells.
Studies show that ABCC4 (show ABCC4 ELISA Kits) and SLC29A2 expression were predictive of achieving CR, and the high expression of GSTP1 (show GSTP1 ELISA Kits) suggests that this may be a therapeutic target for relapsed AML (show RUNX1 ELISA Kits).
Equilibrative nucleoside transporters (hENT1, hENT2) together with adenosine kinase (show ADK ELISA Kits) and 5'-nucleotidase (show NT5E ELISA Kits) play a crucial role in the regulation of CFTR (show CFTR ELISA Kits) through an adenosine-dependent pathway in human airway epithelia.
Only a few endometrial carcinomas (15%) were found to be negative for hCNT1 (show SLC28A1 ELISA Kits), but they all retained hENT1 and hENT2 expression.
the corresponding residues in TMs 1 (show SERINC3 ELISA Kits) and 11 of hENT1, hENT2, and CeENT1 are important for dipyridamole interactions and nucleoside transport.
The low overall genetic diversity in SLC29A2 makes it unlikely that variation in the coding region contributes significantly to clinically observed differences in drug response.
Data suggest that crosstalk pathway between ENT2 and alveolar epithelial Adora2b (show ADORA2B ELISA Kits) in lung protection during acute lung injury (ALI) opens possibilities for combined therapies targeted to this protein set.
Inosine and ENT-2 contribute to hypoxic preconditioning in the murine cardiomyocyte HL-1 (show ASGR1 ELISA Kits) cell line.
HIF-1alpha (show HIF1A ELISA Kits)-dependent repression of ENT2 increases mucosal adenosine signaling and attenuates hypoxia-associated inflammation of the intestine.
Ruminal infusion of starch hydrolysate increased duodenal and ileal expression ENT2 mRNA. Abomasal infusion increased ileal and jejunal ENT2 mRNA expression.
The uptake of nucleosides by transporters, such as SLC29A2, is essential for nucleotide synthesis by salvage pathways in cells that lack de novo biosynthetic pathways. Nucleoside transport also plays a key role in the regulation of many physiologic processes through its effect on adenosine concentration at the cell surface (Griffiths et al., 1997
hypothetical protein LOC394022
, solute carrier family 29 (nucleoside transporters), member 2
, equilibrative nucleoside transporter 2
, 36 kDa nucleolar protein HNP36
, delayed-early response protein 12
, equilibrative NBMPR-insensitive nucleoside transporter
, equilibrative nitrobenzylmercaptopurine riboside-insensitive nucleoside transporter
, hydrophobic nucleolar protein, 36 kDa
, hydrophobic nucleolar protein, 36kD
, nucleoside transporter, ei-type
, solute carrier family 29 member 2
, solute carrier family 29, member 2
, 36 kDa hydrophobic nucleolar protein
, delayed early response gene 12
, NBMPR-insensitive nucleoside transporter ei