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displays Na+/H+ exchanger transport activity\; mediates sodium ion transport across the renal proximal tubule [RGD, Feb 2006].. Additionally we are shipping SLC9A3 Kits (6) and SLC9A3 Proteins (6) and many more products for this protein.
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Mouse (Murine) Polyclonal SLC9A3 Primary Antibody for ICC, IF - ABIN863199
Kim, Ecelbarger, Knepper, Packer: Regulation of thick ascending limb ion transporter abundance in response to altered acid/base intake. in Journal of the American Society of Nephrology : JASN 1999
Show all 4 references for ABIN863199
Mouse (Murine) Polyclonal SLC9A3 Primary Antibody for ICC, IF - ABIN2486552
Dynia, Steinmetz, Kocinsky: NHE3 function and phosphorylation are regulated by a calyculin A-sensitive phosphatase. in American journal of physiology. Renal physiology 2010
Show all 2 references for ABIN2486552
The expression of NHE3 in the rumen of sheep and cattle and its role in sodium transport are reported.
Lysophosphatidic acid stimulation of NHE3 exocytosis includes a signaling pathway that regulates fixation of NHE3 to the microvillar cytoskeleton.
1) NHE3 basal activity is regulated by a signaling complex that is controlled by sequential effects of two kinases, Akt (show AKT1 Antibodies) and GSK-3 (show GSK3b Antibodies), which act on a Ser (show SIGLEC1 Antibodies) cluster in the same NHE3 C-terminal domain that binds ezrin (show EZR Antibodies)
CaMKII (show CAMK2G Antibodies) binding to and phosphorylation of the NHE3 C terminus are parts of the physiologic regulation of NHE3 that occurs in fibroblasts as well as in the brush border of an intestinal Na(+)-absorptive cell.
Regulation of NHE3 depends on the nature of the NHERF (show SLC9A3R1 Antibodies) family member associating with NHE3 and the accompanying NHE3 complexes.
azilsartan (but not candesartan) improved salt sensitivity possibly by decreasing NHE3 expression via ubiquitin-proteasomal degradation.
NHE3 in proximal tubules of the kidney, along with NHE3 in intestines, is required for maintaining basal blood pressure as well as the full development of ANG II (show AGT Antibodies)-induced hypertension.
Inflammation-induced loss of PDZK1 (show PDZK1 Antibodies) expression may contribute to the NHE3 dysfunction observed in the inflamed intestine.
these findings indicate the importance of NHE3 in diabetic diarrhea and suggest LPA administration as a potential therapeutic strategy for management of diabetic diarrhea.
Myosin VI (show MYO6 Antibodies) mediates the movement of NHE3 to the microvillus in intestinal epithelial cells.
Cyclic GMP kinase (show GUK1 Antibodies) II (cGKII (show PRKG2 Antibodies)) inhibits NHE3 by altering its trafficking and phosphorylating NHE3 at three required sites
Both NHE3 and NHE8 likely play a role in neonatal acidification.
NHE3 partially associates with lipid rafts in the brush border membrane.
The gut (show GUSB Antibodies) microbiome plays a prominent role in the pathogenesis of colitis in NHE3 knockout mice, and, reciprocally, NHE3 also plays a critical role in shaping the gut (show GUSB Antibodies) microbiota.
Saline drinking significantly enhanced fluid intake and increased NHE3 transcript levels in the jejunum.
Recessive mutations in NHE3, a downstream target of GC-C (show GUCY2C Antibodies), caused congenital sodium diarrhea and implied primary basal NHE3 malfunction as a predisposition for inflammatory bowel disease in a subset of patients.
CCH-mediated inhibition of NHE3 activity is not dependent on clathrin and involves lipid rafts and requires Cdc42 (show CDC42 Antibodies).
Data demonstrate a role for Per1 (show PER1 Antibodies) in the transcriptional regulation of NHE3 and SGLT1 (show SLC5A1 Antibodies) in the kidney.
Results demonstrate decreased transport activity in three SNPs of NHE3 and provide mechanistic insight into how these SNPs impact NHE3 function.
NHE3 may play a role in the pathogenesis of human cholesterol gallstone disease
Study identifies RSK2 (show RPS6KA3 Antibodies) as a new kinase that regulates NHE3 activity by direct phosphorylation.
the three NHE3 SNPs are unlikely to play a major role in the pathogenesis of SIDS (show IDS Antibodies) in Caucasian infants;further genetic investigations in different ethnicities are required to determine whether variations in NHE3 are associated with an increased SIDS (show IDS Antibodies) risk
C. difficile inhibits NHE3 in vivo, which creates an altered environment favored by C. difficile.
We propose that ouabain can simultaneously regulate renal tubule basolateral Na(+)-K(+)-ATPase (show ATP1A1 Antibodies) and apical NHE3, leading to inhibition of transepithelial Na(+) transport.
displays Na+/H+ exchanger transport activity\; mediates sodium ion transport across the renal proximal tubule
sodium proton exchanger
, sodium/hydrogen exchanger 3
, solute carrier family 9 (sodium/hydrogen exchanger), member 3
, sodium/hydrogen exchanger 3 (nhe3)
, Sodium/hydrogen exchanger 3
, Na(+)/H(+) exchanger 3
, Na+/H+ exchanger homolog
, solute carrier family 9 member 3
, Solute carrier family 9 (sodium/hydrogen exchanger 3), antiporter 3, Na+/H+ (amiloride insensitive)
, plasma membrane ion transport protein
, solute carrier family 9, member 3
, solute carrier family 9 (sodium/hydrogen exchanger)
, sodium/hydrogen exchanger