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promotes cell adhesion and outgrowth of embryonic hippocampal neurons [RGD, Feb 2006].. Additionally we are shipping SPON2 Antibodies (83) and SPON2 Proteins (10) and many more products for this protein.
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Human SPON2 ELISA Kit for Sandwich ELISA - ABIN421013
Lucarelli, Rutigliano, Bettocchi, Palazzo, Vavallo, Galleggiante, Trabucco, Di Clemente, Selvaggi, Battaglia, Ditonno: Spondin-2, a secreted extracellular matrix protein, is a novel diagnostic biomarker for prostate cancer. in The Journal of urology 2013
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Data suggest that spondin-2 could be an independent diagnostic and prognostic biomarker of colon cancer.
Mindin protects against vascular hyperplasia by suppression of abnormal VSMC proliferation, migration and phenotypic switching in an AKT (show AKT1 ELISA Kits)-dependent manner.
Using tissue microarray by immunohistostaining, we found SPON2 to be over-expressed in prostate cancer (PCa (show FLVCR1 ELISA Kits)). SPON2 staining was more intense in Gleason score sum 7-8 and in PCa (show FLVCR1 ELISA Kits) patients with metastasis.
mindin serves as a novel mediator that protects against cardiac hypertrophy and the transition to heart failure by blocking AKT (show AKT1 ELISA Kits)/GSK3beta and TGF-beta1 (show TGFB1 ELISA Kits)-Smad (show SMAD1 ELISA Kits) signalling.
High Mindin is associated with diabetic nephropathy in type 2 diabetes.
Spondin 2, which is regulated by T(3), has an important role in cell invasion, cell migration, and hepatoma tumor progression.
The structure of the F-spondin (show SPON1 ELISA Kits) domain of mindin was determined at 1.8-A resolution.
Mindin is a novel modulator of hepatic I/R injury through regulating inflammatory responses, as well as hepatocyte apoptosis and proliferation via inactivation of the Akt (show AKT1 ELISA Kits) signaling pathway.
Spondin 2 regulates hepatic lipid metabolism and alleviates hepatic steatosis, obesity, inflammation, and insulin (show INS ELISA Kits) resistance via its interaction with PPARalpha (show PPARA ELISA Kits).
The data of this study demonstrated that mindin is a critical mediator of ischemic brain injury in an experimental stroke model.
Data show that mindin, as an intrinsic cardioprotective factor, prevents maladaptive remodelling and the transition to heart failure by blocking AKT/GSK3beta signalling.
Mindin expression is upregulated during intestinal inflammation and may induce NF-kappaBp65 promoter activation in a TLR-9 (show TLR9 ELISA Kits) mediated manner.
Mindin is a pattern-recognition molecule for microbial pathogens.
Mindin-integrin interactions play a key role in regulating Rho GTPase (show RACGAP1 ELISA Kits) expression in DCs and DC priming of T lymphocytes.
mindin plays an essential role in the host innate immune response to influenza virus infection and suggest that mindin may be used as an immune-enhancing agent in influenza infection.
mindin participates in integrin-dependent trafficking of eosinophils and can contribute to the severity of allergic airways disease.
promotes cell adhesion and outgrowth of embryonic hippocampal neurons
, differentially expressed in cancerous and non-cancerous lung cells 1