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SAMD9L encodes a sterile alpha motif domain-containing protein. Additionally we are shipping Sterile alpha Motif Domain Containing 9-Like Kits (11) and and many more products for this protein.
Showing 10 out of 24 products:
Human Polyclonal SAMD9L Primary Antibody for IHC (p), WB - ABIN651359
Olsen, Blagoev, Gnad, Macek, Kumar, Mortensen, Mann: Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. in Cell 2006
Missense Mutations in SAMD9L gene is Associated with Ataxia (show USP14 Antibodies)-Pancytopenia Syndrome.
Replication of the K1L(-)C7L(-) mutant vaccinia virus virus was enabled by multiple siRNAs to SAMD9 (show SAMD9 Antibodies) or WDR6 (show WDR6 Antibodies).
SAMD9 (show SAMD9 Antibodies) is down-regulated in non-small cell lung cancer. Knockdown of SAMD9 (show SAMD9 Antibodies) expression increased the invasion, migration and proliferation in H1299 cells in vitro and overexpression of SAMD9 (show SAMD9 Antibodies) suppressed proliferation and invasion in A549 cells.
When SAMD9 (show SAMD9 Antibodies) is stimulated due to failure of the viral antagonism during infection, the resulting antiviral granules exhibit properties different from those of the canonical stress granules.
The findings highlight a novel tumor-suppressive role of SAMD9L inactivation by somatic mutation and decreased expression in human HBV-related HCC (show FAM126A Antibodies)
Over-expression of SAMD9 (show SAMD9 Antibodies) is correlated with the metastasis of esophageal squamous cell carcinoma
M062 also binds and antagonizes cellular SAMD9 (show SAMD9 Antibodies) in human cells, suggesting that SAMD9 (show SAMD9 Antibodies) is a novel innate antiviral factor against poxviruses.
SAMD9 (show SAMD9 Antibodies), an IFN-gamma (show IFNG Antibodies)-responsive protein, interacts with RGL2 (show RGL2 Antibodies) to diminish the expression of EGR1 (show EGR1 Antibodies), a protein of direct relevance to the pathogenesis of ectopic calcification and inflammation.
SAMD9 (show SAMD9 Antibodies) could be a key molecule to control cancer cell death by inactivated Sendai virus particle or IFN-beta (show IFNB1 Antibodies) treatment.
Mutation analysis revealed a homozygous mutation in the SAMD9 (show SAMD9 Antibodies) gene (K1495E), which was found to segregate with the disease in all families and to interfere with the protein expression.
Haploinsufficiency of SAMD9L, an endosome fusion facilitator, causes myeloid malignancies in mice mimicking human diseases with monosomy 7.
Mouse Samd9l is not a functional paralogue of the human SAMD9 (show SAMD9 Antibodies).
mouse Samd9L revealed near-ubiquitous expression, with the highest level in the kidney, a major organ regulating calcium-phosphate homeostasis and the site of calcitonin (show CALCA Antibodies) hormonal action.
This gene encodes a sterile alpha motif domain-containing protein. The encoded protein localizes to the cytoplasm and may play a role in regulating cell proliferation and apoptosis. Mutations in this gene are the cause of normophosphatemic familial tumoral calcinosis. Alternate splicing results in multiple transcript variants that encode the same protein.
SAM domain-containing protein 9
, expressed in aggressive fibromatosis
, sterile alpha motif domain-containing protein 9
, SAM domain-containing protein 9-like
, sterile alpha motif domain-containing protein 9-like
, v-myc myelocytomatosis viral related oncogene, neuroblastoma derived