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TIMP4 belongs to the TIMP gene family. Additionally we are shipping TIMP4 Kits (60) and TIMP4 Proteins (35) and many more products for this protein.
Showing 10 out of 176 products:
Human Monoclonal TIMP4 Primary Antibody for IHC (fro), IF - ABIN371660
Greene, Wang, Liu, Raymond, Rosen, Shi: Molecular cloning and characterization of human tissue inhibitor of metalloproteinase 4. in The Journal of biological chemistry 1997
Show all 9 references for ABIN371660
Human Polyclonal TIMP4 Primary Antibody for EIA, IHC (p) - ABIN955223
Liss, Sreedhar, Keshgegian, Sauter, Chernick, Prendergast, Wallon: Tissue inhibitor of metalloproteinase-4 is elevated in early-stage breast cancers with accelerated progression and poor clinical course. in The American journal of pathology 2009
Show all 2 references for ABIN955223
Mouse (Murine) Monoclonal TIMP4 Primary Antibody for IHC, ELISA - ABIN1043744
Donover, Wojciechowski, Thirumaran, Zemba-Palko, Prendergast, Wallon: Development of a monoclonal antibody that specifically detects tissue inhibitor of metalloproteinase-4 (TIMP-4) in formalin-fixed, paraffin-embedded human tissues. in Journal of cellular biochemistry 2010
Concluding, miR (show MLXIP Antibodies)-200b-3p mediates regulation of TIMP4 expression in prostate cancer but exact mechanism needs to be investigated.
TIMP1 (show TIMP1 Antibodies), TIMP2 (show TIMP2 Antibodies), and TIMP4 are increased in aqueous humor from primary open angle glaucoma patients.
ncreased TGFB1 (show TGFB1 Antibodies) expression and decreased TIMP-4 expression correlated with atrial fibrosis and extracellular matrix changes in the atria of rheumatic heart disease patients with atrial fibrillation.
Upregulation of plasma TIMP-4 might contribute to PIH [pregnancy-induced hypertension] processes
Expression levels of TIMP-1 (show TIMP1 Antibodies), TIMP-3 (show TIMP3 Antibodies) and TIMP-4 were negligible (<10% of cells) in primary spontaneous pneumothorax lesions.
No evidence was found for any associations between the TIMP-1,-2,-3, or -4 gene single nucleotide polymorphisms with unexplained recurrent spontaneous abortions in this Han Chinese Han population.
This study provides evidence that the promoter TIMP4 rs3755724 is a new focal epilepsy susceptibility variant that is plausibly involved in inflammation-induced seizures in Malaysian Chinese.
Data show that gene polymorphisms of TIMP metallopeptidase (show ECEL1 Antibodies) inhibitors TIMP-3 (show TIMP3 Antibodies) -1296 T>C (rs9619311) and TIMP-4 -55 T>C (rs3755724) were associated with the susceptibility of hepatocellular carcinoma among Taiwan women.
Selective myocardial targeting for TIMP-4 induction through either a viral or transgenic approach favorably altered the course of adverse left ventricular remodeling post-myocardial infarction.
The rs3755724 in TIMP4 protein was nominally associated with schizophrenia with poor concentration.
Our findings highlight a protective mechanism induced by PPARgamma (show PPARG Antibodies) in deoxycorticosterone acetate-salt treatment, establishing a novel mechanistic link between PPARgamma (show PPARG Antibodies) and TIMP-4.
TIMP4 augments contractility and induces differentiation of embryonic stem cells into cardiac phenotype.
TIMP-4 might act as a negative regulator of adipogenesis through NFkB cascade modulation.
Replenishment of myocardial TIMP4 could serve as an effective therapy in post-I/R recovery for patients with reduced TIMP4.
TIMP-4 protein was localized to corpus luteum and theca-intera cells in various periods throughout the pregnancy and postpartum day 1.
Timp4(-/-) mice are more susceptible to MI but not to pressure overload, and TIMP4 functions in its capacity as a metalloproteinase inhibitor after myocardial infarction
Identification of an initiator-like element essential for the expression of the tissue inhibitor of metalloproteinases-4 (Timp-4) gene
molecular cloning and localization of the gene within intron 5 of the synapsin 2 (show SYN2 Antibodies) gene
This study suggests that tissue inhibitor of metalloproteinase-4 may restrict mouse blastocyst outgrowth and embryo implantation by inhibiting the activities of matrix metalloproteinases-2 and -9.
This gene belongs to the TIMP gene family. The proteins encoded by this gene family are inhibitors of the matrix metalloproteinases, a group of peptidases involved in degradation of the extracellular matrix. The secreted, netrin domain-containing protein encoded by this gene is involved in regulation of platelet aggregation and recruitment and may play role in hormonal regulation and endometrial tissue remodeling.
TIMP metallopeptidase inhibitor 4
, tissue inhibitor of metalloproteinase 4
, metalloproteinase inhibitor 4
, tissue inhibitor of metalloproteinases 4
, tissue inhibitor of metalloproteinase
, tissue inhibitor of metalloproteinase-4