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anti-Tetrahydrocannabinol (THC) Antibodies

The Wiskott-Aldrich syndrome (WAS) family of proteins share similar domain structure, and are involved in transduction of signals from receptors on the cell surface to the actin cytoskeleton. Additionally we are shipping and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
THC 7454 P42768
THC    
THC    
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Top anti-Tetrahydrocannabinol Antibodies at antibodies-online.com

Showing 10 out of 123 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Supplier Delivery Price Details
Chemical Mouse Un-conjugated Con   1 mg Log in to see 3 to 4 Days
$243.57
Details
Cannabis Mouse Un-conjugated LF, ELISA   1 mg Log in to see 4 to 5 Days
$315.33
Details
Chemical Mouse Un-conjugated EIA   1 mg Log in to see 6 to 8 Days
$346.50
Details
Cannabis Mouse Un-conjugated   1 mg Log in to see 4 to 5 Days
$376.93
Details
Chemical Mouse Un-conjugated EIA, RIA   0.2 mg Log in to see 6 to 8 Days
$412.50
Details
Chemical Mouse Un-conjugated   1 mg Log in to see 3 to 4 Days
$243.57
Details
Chemical Mouse Un-conjugated ELISA Graph shows data from competitive ELISA with analyte. 1 mg Log in to see 5 to 6 Days
$311.85
Details
Chemical Sheep Un-conjugated ELISA   1 mL Log in to see 5 to 6 Days
$3,161.34
Details
Chemical Rabbit Un-conjugated   1 mL Log in to see 3 to 4 Days
$208.21
Details
Chemical Sheep Un-conjugated   1 mL Log in to see 4 to 5 Days
$328.53
Details

THC Antibodies by Reactivity, Application, Clonality and Conjugate

Attributes Applications Host Clonality Conjugate
Human
,
,
Chemical , , , ,
, , ,
,
, , , , , , ,
Cannabis


More Antibodies against Tetrahydrocannabinol Interaction Partners

Human Tetrahydrocannabinol (THC) interaction partners

  1. This suggests that N-WASP's failure to compensate for WASP (show WASL Antibodies) in rescuing chemotaxis could be due to the absence of this I30 region.

  2. N-WASP (show WASL Antibodies) is downregulated in clear cell renal cell carcinoma (show MOK Antibodies)

  3. Studies indicate that mutations in the Wiskott-Aldrich syndrome protein (WASp) gene cause a continuum of clinical symptoms ranging from intermittent X-linked thrombocytopenia to full classical Wiskott-Aldrich syndrome (show WASL Antibodies) (WAS).

  4. Platelet actin nodule formation is dependent on WASp (show WASL Antibodies) and the ARP2 (show ACTR2 Antibodies)/3 complex.

  5. conclude that tyrosine phosphorylation of WIP is a crucial regulator of WASP stability and function as an actin-nucleation-promoting factor

  6. WASP (show WASL Antibodies), RUNX1 (show RUNX1 Antibodies), and ANKRD26 (show ANKRD26 Antibodies) genes are important for normal TPO (show THPO Antibodies) signaling and the network underlying thrombopoiesis.

  7. The introduction of functional WASp (show WASL Antibodies) by GT corrected the alterations of both central and peripheral B cell tolerance checkpoints. WASp (show WASL Antibodies) plays an important role in the establishment and maintenance of B cell tolerance in humans.

  8. We describe two Malay patients with classical Wiskott-Aldrich Syndrome (show WASL Antibodies) with two different mutations in the WASP (show WASL Antibodies) gene

  9. we identify small ubiquitin-related modifier (SUMO)ylation as a novel posttranslational modification of WASp

  10. studies discovered that HMGB1 (show HMGB1 Antibodies) suppressed phosphorylation, nuclear translocation, and activation of CREB (show CREB1 Antibodies), by inhibiting nuclear translocation of PKA catalytic subunit

Tetrahydrocannabinol (THC) Antigen Profile

Protein Summary

The Wiskott-Aldrich syndrome (WAS) family of proteins share similar domain structure, and are involved in transduction of signals from receptors on the cell surface to the actin cytoskeleton. The presence of a number of different motifs suggests that they are regulated by a number of different stimuli, and interact with multiple proteins. Recent studies have demonstrated that these proteins, directly or indirectly, associate with the small GTPase, Cdc42, known to regulate formation of actin filaments, and the cytoskeletal organizing complex, Arp2/3. Wiskott-Aldrich syndrome is a rare, inherited, X-linked, recessive disease characterized by immune dysregulation and microthrombocytopenia, and is caused by mutations in the WAS gene. The WAS gene product is a cytoplasmic protein, expressed exclusively in hematopoietic cells, which show signalling and cytoskeletal abnormalities in WAS patients. A transcript variant arising as a result of alternative promoter usage, and containing a different 5' UTR sequence, has been described, however, its full-length nature is not known.

Gene names and symbols associated with THC

  • Wiskott-Aldrich syndrome (WAS) antibody
  • IMD2 antibody
  • SCNX antibody
  • THC antibody
  • THC1 antibody
  • WASP antibody

Protein level used designations for THC

eczema-thrombocytopenia , thrombocytopenia 1 (X-linked) , wiskott-Aldrich syndrome protein

GENE ID SPECIES
7454 Homo sapiens
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