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anti-Tetrahydrocannabinol (THC) Antibodies

The Wiskott-Aldrich syndrome (WAS) family of proteins share similar domain structure, and are involved in transduction of signals from receptors on the cell surface to the actin cytoskeleton. Additionally we are shipping and many more products for this protein.

list all antibodies Gene Name GeneID UniProt
THC 7454 P42768
THC    
THC    
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Top anti-Tetrahydrocannabinol Antibodies at antibodies-online.com

Showing 10 out of 130 products:

Catalog No. Reactivity Host Conjugate Application Images Quantity Supplier Delivery Price Details
Cannabis Mouse Un-conjugated LF, ELISA   1 mg Log in to see 4 to 5 Days
$335.87
Details
Chemical Mouse Un-conjugated EIA   1 mg Log in to see 6 to 8 Days
$368.50
Details
Cannabis Mouse Un-conjugated   1 mg Log in to see 4 to 5 Days
$401.87
Details
Chemical Mouse Un-conjugated EIA, RIA   0.2 mg Log in to see 6 to 8 Days
$412.50
Details
Chemical Mouse Un-conjugated ELISA Graph shows data from competitive ELISA with analyte. 1 mg Log in to see 5 to 6 Days
$311.85
Details
Chemical Sheep Un-conjugated ELISA   1 mL Log in to see 7 to 8 Days
$2,423.63
Details
Human Sheep Un-conjugated ELISA, WB   0.1 mL Log in to see 2 to 6 Days
$652.00
Details
All Species Mouse Un-conjugated ELISA   0.1 mg Log in to see 7 to 9 Days
$381.94
Details
Chemical Mouse Un-conjugated Con   1 mg Log in to see 3 to 4 Days
$243.57
Details
All Species Mouse Alexa Fluor 488 ELISA   0.1 mL Log in to see 12 to 15 Days
$500.06
Details

THC Antibodies by Reactivity, Application, Clonality and Conjugate

Attributes Applications Host Clonality Conjugate
Human
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Chemical , , , ,
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All Species
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More Antibodies against Tetrahydrocannabinol Interaction Partners

Human Tetrahydrocannabinol (THC) interaction partners

  1. Authors show that knock-down of WASp (show WASL Antibodies) or expression of Y102F mutant of WASp (show WASL Antibodies) decreases colony formation and in vivo tumor growth. Results show that WASp (show WASL Antibodies) is a novel substrate of ALK (show ALK Antibodies) and has a critical role in regulating invasiveness and oncogenesis of ALCL.

  2. this study describes an Iranian boy with Wiskott-Aldrich syndrome (show WASL Antibodies) with a novel WASP (show WASL Antibodies) mutation

  3. The inducible recruitment of WASp (show WASL Antibodies) to the TCR-CD3 (show CD3 Antibodies) complex is partially dependent of tyrosine phosphorylation of Cd3e (show CD3E Antibodies).

  4. retrospectively investigated the outcome of hematopoietic stem cell transplantation in a cohort of 24 patients with the X-linked thrombocytopenia phenotype and mutations in the WAS gene

  5. This suggests that N-WASP's failure to compensate for WASP (show WASL Antibodies) in rescuing chemotaxis could be due to the absence of this I30 region.

  6. N-WASP (show WASL Antibodies) is downregulated in clear cell renal cell carcinoma (show MOK Antibodies)

  7. Studies indicate that mutations in the Wiskott-Aldrich syndrome protein (WASp) gene cause a continuum of clinical symptoms ranging from intermittent X-linked thrombocytopenia to full classical Wiskott-Aldrich syndrome (show WASL Antibodies) (WAS).

  8. Platelet actin nodule formation is dependent on WASp (show WASL Antibodies) and the ARP2 (show ACTR2 Antibodies)/3 complex.

  9. conclude that tyrosine phosphorylation of WIP is a crucial regulator of WASP stability and function as an actin-nucleation-promoting factor

  10. WASP (show WASL Antibodies), RUNX1 (show RUNX1 Antibodies), and ANKRD26 (show ANKRD26 Antibodies) genes are important for normal TPO (show THPO Antibodies) signaling and the network underlying thrombopoiesis.

Tetrahydrocannabinol (THC) Antigen Profile

Protein Summary

The Wiskott-Aldrich syndrome (WAS) family of proteins share similar domain structure, and are involved in transduction of signals from receptors on the cell surface to the actin cytoskeleton. The presence of a number of different motifs suggests that they are regulated by a number of different stimuli, and interact with multiple proteins. Recent studies have demonstrated that these proteins, directly or indirectly, associate with the small GTPase, Cdc42, known to regulate formation of actin filaments, and the cytoskeletal organizing complex, Arp2/3. Wiskott-Aldrich syndrome is a rare, inherited, X-linked, recessive disease characterized by immune dysregulation and microthrombocytopenia, and is caused by mutations in the WAS gene. The WAS gene product is a cytoplasmic protein, expressed exclusively in hematopoietic cells, which show signalling and cytoskeletal abnormalities in WAS patients. A transcript variant arising as a result of alternative promoter usage, and containing a different 5' UTR sequence, has been described, however, its full-length nature is not known.

Gene names and symbols associated with THC

  • Wiskott-Aldrich syndrome (WAS) antibody
  • IMD2 antibody
  • SCNX antibody
  • THC antibody
  • THC1 antibody
  • WASP antibody

Protein level used designations for THC

eczema-thrombocytopenia , thrombocytopenia 1 (X-linked) , wiskott-Aldrich syndrome protein

GENE ID SPECIES
7454 Homo sapiens
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