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TGFB3 encodes a member of the TGF-beta family of proteins. Additionally we are shipping TGFB3 Kits (93) and TGFB3 Antibodies (92) and many more products for this protein.
Showing 10 out of 62 products:
Human TGFB3 Protein expressed in CHO Cells - ABIN2666748
Ferguson, Duncan, Bond, Bush, Durani, So, Taylor, Chantrey, Mason, James, Laverty, Occleston, Sattar, Ludlow, OKane: Prophylactic administration of avotermin for improvement of skin scarring: three double-blind, placebo-controlled, phase I/II studies. in Lancet (London, England) 2009
Show all 5 references for ABIN2666748
Human TGFB3 Protein expressed in Nicotiana benthamiana - ABIN1112031
ten Dijke, Hansen, Iwata, Pieler, Foulkes: Identification of another member of the transforming growth factor type beta gene family. in Proceedings of the National Academy of Sciences of the United States of America 1988
Show all 4 references for ABIN1112031
Results identified a novel human TGFB3 mutation, which contributes to the clinical delineation of the emerging connective tissue disorder tentatively called Rienhoff syndrome, a disorder overlapping with Marfan and Loeys-Dietz syndrome.
TGFbeta (show TGFB1 Proteins) signaling via both SMAD2 (show SMAD2 Proteins)/7 and PARD6/SMURF1 (show SMURF1 Proteins) pathways plays a role in trophoblast growth and differentiation.
Fluocinolone Acetonide enhances TGF-beta3-associated chondrogenesis of bone marrow derived mesenchymal stem cells
Our study uncovers the PITX2-induced expression of TGFB1/2/3 as well as INHBA genes (p < 0.01) followed by SMAD2/3-dependent TGF-b signalling pathway in ovarian cancer cells
TGF-beta3-expressing CD4 (show CD4 Proteins)+CD25 (show IL2RA Proteins)(-)LAG3 (show LAG3 Proteins)+ regulatory T cells have a role in controlling humoral immune responses
There was no association between TGF-beta (show TGFB1 Proteins) gene polymorphisms and juvenile-onset systemic lupus erythematosus in an Iranian cohort.
TGF-beta3 mediates the attenuating effect of MSCs on both the proliferation and extracellular matrix production of human keloid fibroblasts and decreases skin fibrosis
TGFbeta1 (show TGFB1 Proteins) and TGFbeta2 appeared to play a significant role in physiological and pathological conditions in the fetus.
TGF-beta3 existed in synovium and LBs of SC, and was responsible for the pathogenesis of SC.
TGFB3 polymorphism, significant differences were observed for allele and genotype frequencies between caries free and caries affected individuals in oral cleft group (p = 0.013 and 0.006 for allele and genotype frequencies respectively).
TGFbeta3 increases IRF6 (show IRF6 Proteins) expression and subsequently regulates SNAI2 (show SNAI2 Proteins) expression; IRF6 (show IRF6 Proteins) appears to regulate epithelial mesenchymal transition during palatal fusion via SNAI2 (show SNAI2 Proteins).
Analysis of the Tgf-beta-3 knockout mouse model has enabled identification of miRNAs with altered expression that may contribute to the cleft palate phenotype.
Pathological TGF-beta (show TGFB1 Proteins) release from osteolytic bone metastases contributes to muscle weakness in cancer by decreasing Ca(2 (show CA2 Proteins)+)-induced muscle force production.
Ephrin reverse signaling mediates palatal fusion and epithelial-to-mesenchymal transition independently of Tgfss3.
TGF-beta3 significantly downregulates JAM-B (show JAM2 Proteins) expression via post-transcriptional and post-translational modulation and results in the disruption of BTB and apical ES.
Controlled chondrogenesis from adipose-derived stem cells by recombinant transforming growth factor-beta3 fusion protein in peptide scaffolds.
TGF-beta3 mediates palate fusion in part by down-regulating Jagged2 (show JAG2 Proteins) expression in palatal medial edge epithelium.
This study provides a comprehensive list of genes differentially expressed in the healing corneal epithelial cells of diabetic corneas and suggests the therapeutic potential of TGF-beta3 for treating corneal and skin wounds in diabetic patients.
Transforming growth factor-beta3 (TGF-beta3) knock-in ameliorates inflammation due to TGF-beta1 (show TGFB1 Proteins) deficiency while promoting glucose tolerance.
The addition of TGF-beta3 to the 3D cultures further up-regulates the expression of these genes and also induces the expression of mature tenocyte markers Tenomodulin (show TNMD Proteins) and Thrombospondin-4 (show THBS4 Proteins).
TGFbeta (show TGFB1 Proteins) may play a role in the overall process of luteinization, but it appears not to influence steroidogenesis in luteinizing pig follicles.
In swine, TGF-beta3 mRNA is expressed throughout the oestrus cycle.
These data suggest Pez plays a crucial role in organogenesis by inducing TGFbeta (show TGFB1 Proteins) and epithelial-mesenchymal transition.
Tissues exposed to TGF-beta3 had significantly increased glycosaminoglycan and total collagen protein production along with upregulated cartilage-specific gene expression, resulting in tissues with a higher Young's Modulus
Data show that TGF-beta pathways operate during ovarian fetal development, and fibrillin 3 is highly expressed at a critical stage early in developing human and bovine fetal ovaries.
This gene encodes a member of the TGF-beta family of proteins. The encoded protein is secreted and is involved in embryogenesis and cell differentiation. Defects in this gene are a cause of familial arrhythmogenic right ventricular dysplasia 1.
transforming growth factor, beta 3
, transforming growth factor beta-3
, transforming growth factor beta-3 preproprotein
, transforming growth factor beta-3-like
, transforming growth factor-beta3
, transforming growth factor b3
, transforming growth factor-beta 3
, protein kinase
, tgf beta 3