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TGFBI encodes an RGD-containing protein that binds to type I, II and IV collagens. Additionally we are shipping Transforming Growth Factor, beta-Induced, 68kDa Kits (30) and Transforming Growth Factor, beta-Induced, 68kDa Proteins (11) and many more products for this protein.
Showing 10 out of 101 products:
Human Polyclonal TGFBI Primary Antibody for EIA, WB - ABIN955176
Edelstein, Huang, Harocopos, Waltman: Genotype of lattice corneal dystrophy (R124C mutation in TGFBI) in a patient presenting with features of avellino corneal dystrophy. in Cornea 2010
Show all 3 references for ABIN955176
Human Polyclonal TGFBI Primary Antibody for ELISA, WB - ABIN563175
Han, Sim, Vora, Huang: A unique TGFBI protein in granular corneal dystrophy types 1 and 2. in Current eye research 2012
downregulation of CSD2 (show TGFB1 Antibodies) by the heat-inducible miR398 is required for thermotolerance in Arabidopsis.
Data indicate that in the MKK5 (show MAP2K5 Antibodies)-RNAi (RNA interference) lines, the induction of CSD1 and CSD2 (show TGFB1 Antibodies) as well as the activation of MKK5 (show MAP2K5 Antibodies) activity were completely arrested.
Data indicate that transgenic plants expressing miR398-resistant forms of CSD1, CSD2 (show TGFB1 Antibodies) and CCS (show CCS Antibodies) under the control of their native promoters are more sensitive to heat stress.
miR398 links expression of the three major chloroplast copper proteins, plastocyanin, CCS1 and Csd2 (show TGFB1 Antibodies), to copper availability.
CSD2 (show TGFB1 Antibodies) expression is fine-tuned by miR398-directed mRNA cleavage.
Results show that CSD1, but not CSD2 (show TGFB1 Antibodies), mRNA levels were negatively correlated with miR398 levels during ozone, salinity and biotic stress.
TGF-beta (show TGFB1 Antibodies)-induced matricellular protein-null lungs displayed diminished elastic recoil and gas exchange efficiency
the data suggest a model by which cytokine-induced Myt3 suppression leads to Tgfbi de-repression and subsequently to impaired islet-cell migration, revealing a novel role for Myt3 in regulating islet-cell migration.
Studies reveal that growth hormone (GH (show GH1 Antibodies))increases TGFBI (show TGFB1 Antibodies) expression in a mouse and rat models of diabetic nephropathy (DN) which correlate with clinical results, and suggest that this mechanism can explain the podocytes depletion in DN.
Tgfbi (show TGFB1 Antibodies) deficiency is associated with the degradation of cartilage matrix
The results support a novel inflammatory pathway for diabetic retinopathy. This pathway is initiated by TGFbeta (show TGFB1 Antibodies) released from macrophages, which promotes synthesis and release of BIGH3 (show TGFB1 Antibodies) protein by REC (show ZDHHC2 Antibodies) and REC (show ZDHHC2 Antibodies) apoptosis.
TGFBI (show TGFB1 Antibodies) is a potential risk gene for human diabetes.
High blood TGFBIp levels were observed in cecal ligation and puncture (CLP)-induced septic mice. The absence of the TGFBIp gene in mice attenuated CLP-induced sepsis.
Collectively, our findings indicate that betaig-h3 (show TGFB1 Antibodies) promotes the fibrogenic influence of TGFbeta (show TGFB1 Antibodies) signaling, neutralizing the prochondrogenic influence of the hypoxic-inducible factor 1 activated by the microenvironment of limb mesenchymal aggregates.
the observed effect of TGFBI (show TGFB1 Antibodies) on bone mass and bone size was largely caused by the effect of TGFBI (show TGFB1 Antibodies) on periosteal bone formation
betaig-h3 (show TGFB1 Antibodies) bears alphavbeta3 integrin-interacting motifs that mediate endothelial cell adhesion and migration and angiogenesis
This study shows that upregulation of bigh3 and bambia mRNA correlated with the process of fin regeneration and regulation of TGF-b signaling, suggesting a new role for these proteins.
Results describe the distribution of TGFBI (show TGFB1 Antibodies) in the developing skeletal muscle of zebrafish embryos, and use antisense morpholino oligonucleotides to investigate the function of the endogenous protein.
LASIK surgery of granular corneal dystrophy type 2 patients leads to accumulation and differential proteolytic processing TGFBI protein.
High TGFBI expression in Esophageal squamous cell carcinoma is associated with hematogenous recurrence.
We observed that knockdown of Cul4A (show CUL4A Antibodies) was associated with increased sensitivity to gemcitabine through upregulation of TGFBI in lung cancer cells.
TGFBIp expression is positively regulated by TGF-beta1 (show TGFB1 Antibodies) in corneal fibroblasts, whereas TGF-beta1 (show TGFB1 Antibodies)-induced miR (show MLXIP Antibodies)-21 and miR (show MLXIP Antibodies)-181a negatively regulate TGFBIp expression.
Data suggest that periostin (show POSTN Antibodies) and TGFBI (transforming growth factor-beta induced protein) can be considered paralogs in some ways: structural homology, phylogeny, and functions in extracellular matrix formation/cell adhesion/cellular immunity. [REVIEW]
No mutations were detected in the TGFBI gene in Korean patients with macular corneal dystrophy.
Studies reveal that growth hormone (GH (show GH1 Antibodies))increases TGFBI expression in a mouse and rat models of diabetic nephropathy (DN) which correlate with clinical results, and suggest that this mechanism can explain the podocytes depletion in DN.
the functional role of H3K4me in TGFbeta1 (show TGFB1 Antibodies)-mediated TGFBIp and ECM (show MMRN1 Antibodies) gene expression in corneal fibroblasts
19 novel TGFB1 (show TGFB1 Antibodies) regulatory region and exon 1 alleles are presented: p018-p036. p018, p019, and p020 are formed by novel combinations of known polymorphic positions. Another 16 novel alleles are shown to carry additional known and unknown SNPs.
The p. Arg555Trp mutation of the TGFBI gene was associated with Thiel-Behnke corneal dystrophy, which revealed a novel phenotype-genotype correlation within the mutational spectrum of phenotypically diverse corneal dystrophies.
This gene encodes an RGD-containing protein that binds to type I, II and IV collagens. The RGD motif is found in many extracellular matrix proteins modulating cell adhesion and serves as a ligand recognition sequence for several integrins. This protein plays a role in cell-collagen interactions and may be involved in endochondrial bone formation in cartilage. The protein is induced by transforming growth factor-beta and acts to inhibit cell adhesion. Mutations in this gene are associated with multiple types of corneal dystrophy.
, transforming growth factor, beta induced, 68 kDa
, transforming growth factor-beta-induced protein ig-h3
, RGD-containing collagen-associated protein
, Transforming growth factor-beta-induced protein ig-h3
, transforming growth factor induced protein