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TREM1 encodes a receptor belonging to the Ig superfamily that is expressed on myeloid cells. Additionally we are shipping TREM1 Kits (60) and TREM1 Proteins (32) and many more products for this protein.
Showing 10 out of 264 products:
Human Monoclonal TREM1 Primary Antibody for FACS - ABIN4896332
Oku, Oda, Nakada, Sadahiro, Nakamura, Hirayama, Abe, Tateishi, Ito, Iseki, Hirasawa: Differential pattern of cell-surface and soluble TREM-1 between sepsis and SIRS. in Cytokine 2012
Show all 4 references for ABIN4896332
Human Monoclonal TREM1 Primary Antibody for FACS - ABIN2663405
Gingras, Lapillonne, Margolin: TREM-1, MDL-1, and DAP12 expression is associated with a mature stage of myeloid development. in Molecular immunology 2002
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Human Monoclonal TREM1 Primary Antibody for ELISA - ABIN1997001
Bouchon, Facchetti, Weigand, Colonna: TREM-1 amplifies inflammation and is a crucial mediator of septic shock. in Nature 2001
Show all 3 references for ABIN1997001
Human Monoclonal TREM1 Primary Antibody for FACS - ABIN2661061
Bouchon, Dietrich, Colonna: Cutting edge: inflammatory responses can be triggered by TREM-1, a novel receptor expressed on neutrophils and monocytes. in Journal of immunology (Baltimore, Md. : 1950) 2000
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Mouse (Murine) Monoclonal TREM1 Primary Antibody for FACS - ABIN4896328
Gibot, Massin, Marcou, Taylor, Stidwill, Wilson, Singer, Bellingan: TREM-1 promotes survival during septic shock in mice. in European journal of immunology 2007
Show all 3 references for ABIN4896328
Human Monoclonal TREM1 Primary Antibody for Func, EIA - ABIN1109324
Radsak, Taube, Haselmayer, Tenzer, Salih, Wiewrodt, Buhl, Schild: Soluble triggering receptor expressed on myeloid cells 1 is released in patients with stable chronic obstructive pulmonary disease. in Clinical & developmental immunology 2008
Show all 2 references for ABIN1109324
this study shows higher levels of TREM-1 in plasma of systemic lupus erythematosus in Egyptian patients
Increased expression of TREM-1 in S compared to AS patients involving TNFa (show TNF Antibodies), MMP-1 (show MMP1 Antibodies) and MMP-9 (show MMP9 Antibodies) suggest a potential role of TREM-1 in plaque destabilization
Plasma levels of sTREM-1 were found to identify patients with septic shock more effectively than procalcitonin and C-reactive protein (show CRP Antibodies). Moreover, sTREM-1 was identified to be an early predictor for survival in patients with septic shock.
This study provides new insights into the possible mechanism of TREM-1 and HIF-1alpha (show HIF1A Antibodies) in psoriasis.
TREM-1 may play an important role in the inflammatory cascade after subarachnoid hemorrhage and act as a monitoring biomarker
This study shows increased expression of TREM-1 in monocytes from patients with advanced cervical cancer.
TREM-1 expression is decreased during the generation of human osteoclast precursors
TREM-1 activation may be involved in the development of Kawasaki disease.
LPS (show IRF6 Antibodies)-induced TREM-1 transcription.
The study simultaneously evaluated both PCT (show UROD Antibodies) and soluble TREM-1 along with CRP (show CRP Antibodies) in febrile patients with autoimmune diseases.
TREM-1 is upregulated in renal inflammation and plays a vital role in driving disease. Thus, TREM-1 blockade emerges as a potential therapeutic avenue for immune-mediated renal diseases such as lupus nephritis
TGF-beta1 (show TGFB1 Antibodies) increased the expression of TREM-1 in mouse macrophages partly via the transcription factor AP-1 (show JUN Antibodies).
in macrophages, oxidized low-density lipoprotein enhanced expression of TREM-1, which amplifies the innate immune response of Toll (show TLR4 Antibodies)-like receptor pathway; activation of TREM-1 contributes to atherogenesis process by enhancing proinflammatory cytokine production and foam cell formation.
TREM-1 and Dectin-1 (show CLEC7A Antibodies) function concurrently in the corneal innate immune response by regulating inflammatory cytokine expression in fungal keratitis.
a novel positive feedback cycle mediated by TREM-1 and GM-CSF (show CSF2 Antibodies) was revealed to enhance macrophage polarization under unilateral ureteral obstruction.
TREM-1-mediated innate immune response played an essential role in the activation of neutrophils and Streptococcus suis clearance.
Data indicate that triggering receptor expressed on myeloid cells-1(TREM-1) inhibition improves heart function after myocardial infarction (MI).
TREM-1 is significantly associated with LPS (show TLR4 Antibodies)-induced left ventricular systolic dysfunction in BALB/c mice
TREM-1 deficiency can attenuate disease severity without affecting pathogen clearance.
Data indicate that triggering receptor expressed on myeloid cells 1 (TREM-1) induces anti-apoptotic protein Bcl-2 (show BCL2 Antibodies) and prolongs macrophage survival.
Molecular cloning of bovine TREM-1 extends the repertoire of bovine pattern recognition receptors and provides information important for investigating their role in bovine innate immunity.
This gene encodes a receptor belonging to the Ig superfamily that is expressed on myeloid cells. This protein amplifies neutrophil and monocyte-mediated inflammatory responses triggered by bacterial and fungal infections by stimulating release of pro-inflammatory chemokines and cytokines, as well as increased surface expression of cell activation markers. Alternatively spliced transcript variants encoding different isoforms have been noted for this gene.
triggering receptor expressed on myeloid cells 1
, triggering receptor expressed on monocytes 1
, triggering-receptor TREM1
, triggering receptor expressed in monocytes 1
, triggering receptor expressed on myeloid cells-1