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TREM1 encodes a receptor belonging to the Ig superfamily that is expressed on myeloid cells. Additionally we are shipping TREM1 Kits (64) and TREM1 Proteins (32) and many more products for this protein.
Showing 10 out of 271 products:
Human Monoclonal TREM1 Primary Antibody for FACS - ABIN4896332
Oku, Oda, Nakada, Sadahiro, Nakamura, Hirayama, Abe, Tateishi, Ito, Iseki, Hirasawa: Differential pattern of cell-surface and soluble TREM-1 between sepsis and SIRS. in Cytokine 2012
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Human Monoclonal TREM1 Primary Antibody for FACS - ABIN2661061
Gingras, Lapillonne, Margolin: TREM-1, MDL-1, and DAP12 expression is associated with a mature stage of myeloid development. in Molecular immunology 2002
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Mouse (Murine) Monoclonal TREM1 Primary Antibody for FACS - ABIN4896328
Gibot, Massin, Marcou, Taylor, Stidwill, Wilson, Singer, Bellingan: TREM-1 promotes survival during septic shock in mice. in European journal of immunology 2007
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Human Monoclonal TREM1 Primary Antibody for ELISA - ABIN1997001
Bouchon, Facchetti, Weigand, Colonna: TREM-1 amplifies inflammation and is a crucial mediator of septic shock. in Nature 2001
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Human Monoclonal TREM1 Primary Antibody for Func, EIA - ABIN1109324
Radsak, Taube, Haselmayer, Tenzer, Salih, Wiewrodt, Buhl, Schild: Soluble triggering receptor expressed on myeloid cells 1 is released in patients with stable chronic obstructive pulmonary disease. in Clinical & developmental immunology 2008
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study found that soluble TREM-1 is increased and correlates with the clinical and laboratory findings in Crimean Congo Haemorrhagic Fever, a viral infection characterized by activation of inflammation.
The findings suggest that there are no differences in soluble TREM-1 levels in gingival crevicular fluid between healthy and periodontally diseased elderly adults
TREM-1 was found to be significantly higher in tissues biopsies and blood of subjects with obesity. Greater expression and activity of TREM-1 suggest a possible role in the underlying pathophysiology of obesity and associated comorbidities.
Carriage of the TREM1 rs2234237T allele appears to be a risk factor for the development of severe malaria.
TREM-1, HMGB1 (show HMGB1 Antibodies) and RAGE (show AGER Antibodies) in the Shoulder Tendon: Dual Mechanisms for Inflammation Based on the Coincidence of Glenohumeral Arthritis
is concluded that both HMGB1 (show HMGB1 Antibodies) and sTREM-1 are released during the inflammatory response of periodontal tissues and they can promote inflammatory process, which leads to tissue destruction.
The soluble TREM-1 level, core temperature and SAPS (show SKAP2 Antibodies) II value were the only independent predictors of death after adjustment for potential confounders in sepsis patients admitted to intensive care.
TREM-1 plays a critical role in osteoarthritis development and IL1B (show IL1B Antibodies) induced chondrocyte injury through regulation of NF-kappaB (show NFKB1 Antibodies) signaling
this study shows higher levels of TREM-1 in plasma of systemic lupus erythematosus in Egyptian patients
Increased expression of TREM-1 in S compared to AS patients involving TNFa (show TNF Antibodies), MMP-1 (show MMP1 Antibodies) and MMP-9 (show MMP9 Antibodies) suggest a potential role of TREM-1 in plaque destabilization
This study is the first to show the regulatory role of TREM-1 in RLRs and TLRs expression.
TREM-1 is upregulated in renal inflammation and plays a vital role in driving disease. Thus, TREM-1 blockade emerges as a potential therapeutic avenue for immune-mediated renal diseases such as lupus nephritis
TGF-beta1 (show TGFB1 Antibodies) increased the expression of TREM-1 in mouse macrophages partly via the transcription factor AP-1 (show JUN Antibodies).
in macrophages, oxidized low-density lipoprotein enhanced expression of TREM-1, which amplifies the innate immune response of Toll (show TLR4 Antibodies)-like receptor pathway; activation of TREM-1 contributes to atherogenesis process by enhancing proinflammatory cytokine production and foam cell formation.
TREM-1 and Dectin-1 (show CLEC7A Antibodies) function concurrently in the corneal innate immune response by regulating inflammatory cytokine expression in fungal keratitis.
a novel positive feedback cycle mediated by TREM-1 and GM-CSF (show CSF2 Antibodies) was revealed to enhance macrophage polarization under unilateral ureteral obstruction.
TREM-1-mediated innate immune response played an essential role in the activation of neutrophils and Streptococcus suis clearance.
Data indicate that triggering receptor expressed on myeloid cells-1(TREM-1) inhibition improves heart function after myocardial infarction (MI).
TREM-1 is significantly associated with LPS (show TLR4 Antibodies)-induced left ventricular systolic dysfunction in BALB/c mice
TREM-1 deficiency can attenuate disease severity without affecting pathogen clearance.
Molecular cloning of bovine TREM-1 extends the repertoire of bovine pattern recognition receptors and provides information important for investigating their role in bovine innate immunity.
This gene encodes a receptor belonging to the Ig superfamily that is expressed on myeloid cells. This protein amplifies neutrophil and monocyte-mediated inflammatory responses triggered by bacterial and fungal infections by stimulating release of pro-inflammatory chemokines and cytokines, as well as increased surface expression of cell activation markers. Alternatively spliced transcript variants encoding different isoforms have been noted for this gene.
triggering receptor expressed on myeloid cells 1
, triggering receptor expressed on monocytes 1
, triggering-receptor TREM1
, triggering receptor expressed in monocytes 1
, triggering receptor expressed on myeloid cells-1