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Acts as a negative mediator of apoptosis. Additionally we are shipping TNFAIP8 Antibodies (43) and TNFAIP8 Proteins (12) and many more products for this protein.
Expression of TNFAIP8 is up-regulated in human gastric cancer and regulates cell proliferation, invasion and migration.
Our data suggest that TNFAIP8 overexpression may contribute to lymph node metastasis and poor prognosis in intestinal-type gastric adenocarcinoma.
TNFAIP8 may be used as a prognostic marker for the recurrence of endometrial cancer (EC), and its promotion of the proliferation and metastasis in EC may be due to its mediation of Ki-67 (show MKI67 ELISA Kits) and MMP9 (show MMP9 ELISA Kits).
our findings indicate that TNFAIP8 overexpression is an independent predictor of platinum resistance in epithelial ovarian cancer
SCC-S2 may play roles in affecting both immune cells and tumor cells in the thyroid and may indicate a novel pathway for understanding the pathogenesis of the disease.
These results provide evidence that TNFAIP8 plays critical roles in non-small cell lung cancer
the TNFAIP8-rs11064 Single nucleotide polymorphism may function by affecting the affinity of miR (show MLXIP ELISA Kits)-22 binding to the 3'-untranslated region of TNFAIP8 and regulating TNFAIP8 expression, thus contributing to cervical cancer risk
TNFAIP8 functions in oncogenesis are likely to involve activation of the integrin, MMP and VEGFR-2 (show KDR ELISA Kits) signaling pathways. TNFAIP8 expression revealed correlation of both cytoplasmic and nuclear TNFAIP8 overexpression with high grade prostatic adenocarcinomas
SCC-S2 is overexpressed in colon cancers.
results showed stronger staining of TNFAIP8 protein in pancreatic cancer tissues compared with normal pancreas tissue; TNFAIP8 expression positively correlated with EGFR (show EGFR ELISA Kits) levels; results indicate TNFAIP8 may play important roles in the progression of pancreatic cancer
TNFAIP8 regulates RAC1 signaling during TNF (show TNF ELISA Kits)-alphaR activation and L. monocytogenes invasion of cells and that this regulation is crucial for controlling cell death and lethal L. mono-cytogenes infection of mice.
Data indicate that mutant Tnfaip8/Oxi-alpha has been crystallized and X-ray data have been collected to 1.8 A using synchrotron radiation.
Tnfaip8 expression is associated with susceptibility to Staphylococcus aureus sepsis.
Data suggest that Galphai-TNFAIP8-mediated rescue of pre-oncogenic cells enhances progression to oncogenic transformation, providing a selective target to inhibit cellular transformation.
Tnfaip8 is crucial in regulating glucocorticoid-mediated apoptosis of thymocytes
Acts as a negative mediator of apoptosis. Suppresses the TNF-mediated apoptosis by inhibiting caspase-8 activity but not the processing of procaspase-8, subsequently resulting in inhibition of BID cleavage and caspase-3 activation (By similarity).
tumor necrosis factor, alpha-induced protein 8
, TNF alpha-induced protein 8
, tumor necrosis factor alpha-induced protein 8
, NF-kappa-B-inducible DED-containing protein
, TNF-induced protein GG2-1
, head and neck tumor and metastasis-related protein
, TNF-induced protein