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Acts as a negative mediator of apoptosis. Additionally we are shipping TNFAIP8 Antibodies (45) and TNFAIP8 Proteins (11) and many more products for this protein.
induction of TNFAIP8 is critical for the evasion of apoptosis by tumor cells expressing the K120R variant of p53 (show TP53 ELISA Kits).
Of these, three CpG sites on TNFAIP8 and PON1 (show PON1 ELISA Kits) genes (corresponding to: cg23917399; cg07086380; and cg07404485, respectively) were significantly differentially methylated between black and non-black individuals. The three CpG sites showed lower methylation status among infants of black women.
TNFAIP8 regulates Hippo (MST1/2) signaling through its interaction with LATS1.
TNFAIP8 overexpression is correlated with axillary lymph node metastasis and poor prognosis in invasive ductal breast carcinoma.
that MicroRNA-9-TNFAIP8 might represent a promising diagnostic biomarker for gastric cancer patients and could be a potential therapeutic target in the prevention and treatment of gastric cancer
these findings suggest that TNFAIP8 overexpression is a potential biomarker to identify pN0 esophageal squamous cell carcinoma patients at higher risk of lymphatic recurrence who may benefit from adjuvant therapy.
Expression of TNFAIP8 is up-regulated in human gastric cancer and regulates cell proliferation, invasion and migration.
Our data suggest that TNFAIP8 overexpression may contribute to lymph node metastasis and poor prognosis in intestinal-type gastric adenocarcinoma.
TNFAIP8 may be used as a prognostic marker for the recurrence of endometrial cancer (EC), and its promotion of the proliferation and metastasis in EC may be due to its mediation of Ki-67 (show MKI67 ELISA Kits) and MMP9 (show MMP9 ELISA Kits).
our findings indicate that TNFAIP8 overexpression is an independent predictor of platinum resistance in epithelial ovarian cancer
CD4 (show CD4 ELISA Kits)+ T lymphocyte proliferative activity was significantly down-regulated when TNFAIP8 gene was silenced by siRNA in mice at 24 h post burn. Down-regulation of TNFAIP8 can significantly decrease expression levels of IL-2 (show IL2 ELISA Kits) and soluble IL-2R at 24 h after thermal injury.
TNFAIP8 regulates RAC1 signaling during TNF (show TNF ELISA Kits)-alphaR activation and L. monocytogenes invasion of cells and that this regulation is crucial for controlling cell death and lethal L. mono-cytogenes infection of mice.
Data indicate that mutant Tnfaip8/Oxi-alpha has been crystallized and X-ray data have been collected to 1.8 A using synchrotron radiation.
Tnfaip8 expression is associated with susceptibility to Staphylococcus aureus sepsis.
Data suggest that Galphai-TNFAIP8-mediated rescue of pre-oncogenic cells enhances progression to oncogenic transformation, providing a selective target to inhibit cellular transformation.
Tnfaip8 is crucial in regulating glucocorticoid-mediated apoptosis of thymocytes
Acts as a negative mediator of apoptosis. Suppresses the TNF-mediated apoptosis by inhibiting caspase-8 activity but not the processing of procaspase-8, subsequently resulting in inhibition of BID cleavage and caspase-3 activation (By similarity).
tumor necrosis factor, alpha-induced protein 8
, TNF alpha-induced protein 8
, tumor necrosis factor alpha-induced protein 8
, NF-kappa-B-inducible DED-containing protein
, TNF-induced protein GG2-1
, head and neck tumor and metastasis-related protein
, TNF-induced protein