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TNFRSF18 encodes a member of the TNF-receptor superfamily. Additionally we are shipping TNFRSF18 Proteins (30) and TNFRSF18 Kits (19) and many more products for this protein.
Showing 10 out of 329 products:
Human Monoclonal TNFRSF18 Primary Antibody for FACS - ABIN4896950
Godfrey, Spoden, Ge, Baker, Liu, Levine, June, Blazar, Porter: Cord blood CD4(+)CD25(+)-derived T regulatory cell lines express FoxP3 protein and manifest potent suppressor function. in Blood 2005
Show all 7 references for ABIN4896950
Human Monoclonal TNFRSF18 Primary Antibody for FACS - ABIN4896944
Mandapathil, Hilldorfer, Szczepanski, Czystowska, Szajnik, Ren, Lang, Jackson, Gorelik, Whiteside: Generation and accumulation of immunosuppressive adenosine by human CD4+CD25highFOXP3+ regulatory T cells. in The Journal of biological chemistry 2010
Show all 3 references for ABIN4896944
Human Monoclonal TNFRSF18 Primary Antibody for FACS - ABIN4896942
Tuyaerts, Van Meirvenne, Bonehill, Heirman, Corthals, Waldmann, Breckpot, Thielemans, Aerts: Expression of human GITRL on myeloid dendritic cells enhances their immunostimulatory function but does not abrogate the suppressive effect of CD4+CD25+ regulatory T cells. in Journal of leukocyte biology 2007
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Mouse (Murine) Monoclonal TNFRSF18 Primary Antibody for FACS - ABIN4895259
Cobb, Hertweck, Smith, OConnor, Graf, Cook, Smale, Sakaguchi, Livesey, Fisher, Merkenschlager: A role for Dicer in immune regulation. in The Journal of experimental medicine 2006
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Mouse (Murine) Monoclonal TNFRSF18 Primary Antibody for FACS - ABIN320239
Cobbold, Castejon, Adams, Zelenika, Graca, Humm, Waldmann: Induction of foxP3+ regulatory T cells in the periphery of T cell receptor transgenic mice tolerized to transplants. in Journal of immunology (Baltimore, Md. : 1950) 2004
Mouse (Murine) Polyclonal TNFRSF18 Primary Antibody for FACS, ELISA - ABIN1169032
Yu, Kim, Song, Min, Jeong, Youn: Identification of a ligand for glucocorticoid-induced tumor necrosis factor receptor constitutively expressed in dendritic cells. in Biochemical and biophysical research communications 2003
Mouse (Murine) Monoclonal TNFRSF18 Primary Antibody for FACS - ABIN4895260
Fecci, Sweeney, Grossi, Nair, Learn, Mitchell, Cui, Cummings, Bigner, Gilboa, Sampson: Systemic anti-CD25 monoclonal antibody administration safely enhances immunity in murine glioma without eliminating regulatory T cells. in Clinical cancer research : an official journal of the American Association for Cancer Research 2006
Human Polyclonal TNFRSF18 Primary Antibody for WB - ABIN2792139
Liu, Li, Mahesh, Pantanelli, Hwang, Siu, Nussenblatt: Glucocorticoid-induced tumor necrosis factor receptor negatively regulates activation of human primary natural killer (NK) cells by blocking proliferative signals and increasing NK cell apoptosis. in The Journal of biological chemistry 2008
Human Polyclonal TNFRSF18 Primary Antibody for IHC, WB - ABIN2792138
Clark, Gurney, Abaya, Baker, Baldwin, Brush, Chen, Chow, Chui, Crowley, Currell, Deuel, Dowd, Eaton, Foster, Grimaldi, Gu, Hass, Heldens, Huang, Kim, Klimowski, Jin, Johnson, Lee, Lewis, Liao, Mark et al.: The secreted protein discovery initiative (SPDI), a large-scale effort to identify novel human secreted and transmembrane proteins: a bioinformatics assessment. ... in Genome research 2003
these results indicate that blockade of GITR signaling can ameliorate arthritis progression mainly by modulating the follicular helper T cell response
GITR appears as a potential target for intervention during infection by the parasite Toxoplasma gondii, even though further studies are still necessary to better characterize the immune response triggered by GITR activation during T. gondii infection
our data suggest a critical role for GITR in Treg cell homeostasis and indicate that Ptpn22 (show PTPN22 Antibodies) independently affects the differentiation status of Treg cells and their homeostatic behavior
These findings provide further support for the continued development of agonist anti-GITR antibodies as an immunotherapeutic strategy for osteosarcoma. We suggest that our proposed immunotherapy could be developed further to improve osteosarcoma treatment
Th9 cells and iTregs are developmentally linked and GITR can subvert tolerogenic conditions to boost Th9 immunity.
GITR is a crucial player in differentiation of thymic regulatory T cells and expansion of regulatory T cells, including both thymic regulatory T cells and peripheral regulatory T cells.
enhanced GITR-triggering mediates its protective, anti-viral effect on the CD8 (show CD8A Antibodies) T cell compartment by boosting CD4 (show CD4 Antibodies) T cell help.
A CD4 (show CD4 Antibodies) T cell-intrinsic role for GITR in sustaining early CD8 (show CD8A Antibodies) and late humoral responses to collectively promote control of chronic LCMV clone 13 infection.
GITRL (show TNFSF18 Antibodies) expressed on macrophages drives cytokine release and T cell activation, resulting in neuropathic pain via GITR-dependent actions. The GITRL (show TNFSF18 Antibodies)-GITR pathway might represent a novel target for the treatment of neuropathic pain.
Data show that GITR agonist antibody alters Treg lineage stability inducing an inflammatory effector T cell phenotype. The resultant loss of lineage stability causes Treg to lose their intra-tumor immune suppressive function.
Aberrant expression of GITR may contribute to systemic lupus erithematosus pathogenesis. Glucocorticoid may achieve its therapeutic effect partly by inducing GITR expression on Tresps rather than Tregs, which initiates the apoptosis of Tresp cells in SLE patients.
GITR expression can enhance the sensitivity to Bortezomib by inhibiting Bortezomib-induced NF-kappaB (show NFKB1 Antibodies) activation.
Data may suggest a key role of regulatory GITR+CD25 (show IL2RA Antibodies) low/-CD4 (show CD4 Antibodies)+ T cells subset in the modulation of the abnormal immune response in lupus erythematosus (SLE) patients.
results suggest that the GITR rs3753348 polymorphism may be involved in the development and susceptibility of CWP.
these results show a higher susceptibility to apoptosis in patients' versus controls' T(reg (show EXTL3 Antibodies)) cells, suggesting that GITR is a T(reg (show EXTL3 Antibodies))-cell marker that would be primarily involved in T(reg (show EXTL3 Antibodies))-cell survival rather than in their suppressor function.
Our findings indicate the possible involvement of GITR-GITRL (show TNFSF18 Antibodies) pathway in the pathogenesis of pSS (show CDSN Antibodies).
GITR acts as a potential tumor suppressor in MM.
Data indicate that the mRNAs of CTLA-4 (show CTLA4 Antibodies) and GITR genes were expressed at lower levels in CVID (show TNFRSF13B Antibodies) patients compared to control group.
GITR is pathologically expressed on Treg cells in systemic lupus erythematosus.
This gene encodes a member of the TNF-receptor superfamily. The encoded receptor has been shown to have increased expression upon T-cell activation, and it is thought to play a key role in dominant immunological self-tolerance maintained by CD25(+)CD4(+) regulatory T cells. Knockout studies in mice also suggest the role of this receptor is in the regulation of CD3-driven T-cell activation and programmed cell death. Three alternatively spliced transcript variants of this gene encoding distinct isoforms have been reported.
tumor necrosis factor receptor superfamily member 18
, glucocorticoid-induced TNFR-related
, tumor necrosis factor receptor superfamily, member 18
, glucocorticoid-induced TNFR-related protein
, TNF receptor superfamily activation-inducible protein
, activation-inducible TNFR family receptor