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TNFRSF6B belongs to the tumor necrosis factor receptor superfamily. Additionally we are shipping TNFRSF6B Antibodies (82) and TNFRSF6B Kits (8) and many more products for this protein.
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High serum DcR3 levels are associated with disease severity in nonatopic asthma patients.
Results show that DcR3 engages TNF (show TNF Proteins) ligands: FasL (show FASL Proteins), LIGHT, and TL1A (show TNFSF15 Proteins) in a structurally similar fashion and its promiscuity is due to recognition of backbone atoms and invariant side chains.
These results raise the possibility for involvement of TL1A (show TNFSF15 Proteins)/DR3/DR3 (show TNFRSF25 Proteins)-mediated mechanisms in epithelial-mesenchymal interactions and the development of inflammation-induced intestinal fibrosis in Crohn's disease.
when idiopathic pulmonary fibrosis cells interact with collagen matrix, aberrantly activated Akt (show AKT1 Proteins) increases DcR3 expression via GSK-3beta-NFATc1 (show NFATC1 Proteins) and protects IPF cells from the FasL (show FASL Proteins)-dependent apoptotic pathway.
Coexpression of TNFRSF6B and PCDHGA3 (show PCDHgA3 Proteins) was observed immunohistochemically in FL18 cells, suggesting potential cooperation in tumor cell maintenance.
Loss of DcR3 impaired the growth and invasive property of HCC (show FAM126A Proteins) cell line of HepG2.
Overexpression of DcR3 was significantly related to the risk of female reproductive cancers (OR=10.69, 95% CI: 6.33-18.05), TNM (show ODZ1 Proteins) stage (OR=5.51, 95% CI: 2.83-10.71), differentiation (OR=4.16, 95% CI: 2.28-7.60), lymph node metastasis (OR=5.89, 95% CI: 3.16-10.9), age (OR=0.85, 95% CI: 0.51-1.44), and overall survival time (OR=1.84, 95% CI: 0.58-5.83).
The serum level of DcR3 may be a useful marker for disease activity in ANCA-associated renal vasculitis.
DcR3 inhibits influenza a virus-induced JNK (show MAPK8 Proteins) and ERK (show EPHB2 Proteins) activation in human macrophages
Decoy receptor 3 (DcR3) expression in kidneys has been shown to predict progression of chronic kidney disease.
This gene belongs to the tumor necrosis factor receptor superfamily. The encoded protein is postulated to play a regulatory role in suppressing FasL- and LIGHT-mediated cell death. It acts as a decoy receptor that competes with death receptors for ligand binding. Over-expression of this gene has been noted in gastrointestinal tract tumors. Read-through transcription into this gene from the neighboring upstream gene, which encodes regulator of telomere elongation helicase 1 (RTEL1), generates a non-coding transcript.
decoy receptor 3
, decoy receptor for Fas ligand
, tumor necrosis factor receptor superfamily member 6B
, tumor necrosis factor receptor superfamily, member 6b
, tumor necrosis factor receptor superfamily, member 6b, decoy