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The low density lipoprotein receptor (LDLR) gene family consists of cell surface proteins involved in receptor-mediated endocytosis of specific ligands. Additionally we are shipping VLDLR Kits (42) and VLDLR Proteins (8) and many more products for this protein.
Showing 10 out of 70 products:
Human Polyclonal VLDLR Primary Antibody for ELISA - ABIN1998173
Trommsdorff, Gotthardt, Hiesberger, Shelton, Stockinger, Nimpf, Hammer, Richardson, Herz: Reeler/Disabled-like disruption of neuronal migration in knockout mice lacking the VLDL receptor and ApoE receptor 2. in Cell 1999
Show all 5 references for ABIN1998173
Human Polyclonal VLDLR Primary Antibody for FACS, IHC (p) - ABIN651940
Sakai, Tiebel, Ljungberg, Sullivan, Lee, Terashima, Li, Kobayashi, Lu, Chan, Oka: A neuronal VLDLR variant lacking the third complement-type repeat exhibits high capacity binding of apoE containing lipoproteins. in Brain research 2009
Show all 2 references for ABIN651940
Human Monoclonal VLDLR Primary Antibody for EIA - ABIN263866
Ruiz, Kouiavskaia, Migliorini, Robinson, Saenko, Gorlatova, Li, Lawrence, Hyman, Weisgraber, Strickland: The apoE isoform binding properties of the VLDL receptor reveal marked differences from LRP and the LDL receptor. in Journal of lipid research 2005
Human Polyclonal VLDLR Primary Antibody for IF (p), IHC (p) - ABIN705551
Fredriksson, Mishra, Lam, Mushaben, Cuento, Meyer, Yao, Keeran, Nugent, Qu, Yu, Yang, Raghavachari, Dagur, McCoy, Levine: The very low density lipoprotein receptor attenuates house dust mite-induced airway inflammation by suppressing dendritic cell-mediated adaptive immune responses. in Journal of immunology (Baltimore, Md. : 1950) 2014
These results for the first time demonstrated that SalA protected against IS/RP-induced endothelial barrier dysfunction through suppression of VLDL receptor expression
these results suggest that VLDLR functions in vivo as an HCV receptor independent of canonical CD81 (show CD81 Antibodies)-mediated HCV entry.
the results obtained indicate that minimal fibrin-binding structures are located within the second and third CR domains of the VLDL receptor and the presence of the fourth CR domain is required for high-affinity binding
The results of this study demonstrated the presence of reelin (show RELN Antibodies), its receptors VLDLR and ApoER2 (show LRP8 Antibodies) as well as Dab1 (show DAB1 Antibodies) in the ENS and might indicate a novel role of the reelin (show RELN Antibodies) system in regulating neuronal plasticity and pre-synaptic functions in the ENS.
these results suggested that the miR (show MLXIP Antibodies)-135a-VLDLR-p38 (show CRK Antibodies) axis may contribute to gallbladder cancer cell proliferation
study identified a novel homozygous VLDLR c.2248C>T mutation (p.Q750X) and distinctive MRI (show C7ORF49 Antibodies) findings in 2 siblings with ataxia (show USP14 Antibodies); also marked vitamin E deficiency was detected in the proband
these results identify a novel role for the VLDLR as a negative regulator of DC-mediated adaptive immune responses in HDM (show HDAC3 Antibodies)-induced allergic airway inflammation.
ectopic expression of HIC1 (show HIC1 Antibodies) in U2OS and MDA-MB-231 cell lines decreases expression of the ApoER2 (show LRP8 Antibodies) and VLDLR genes, encoding two canonical tyrosine kinase (show TXK Antibodies) receptors for Reelin (show RELN Antibodies).
an unusual constellation of VLDLR mutations in Cerebellar ataxia (show USP14 Antibodies), mental retardation and dysequilibrium syndrome 1 is reported
Results show variation in VLDLR is implicated in disordered gambling
The absence of PCSK9 (show PCSK9 Antibodies) results in a sex- and tissue-specific subcellular distribution of the LDLR (show LDLR Antibodies) and VLDLR, which is determined by estradiol levels.
In the retinas of Vldlr(-/-) mice with low fatty acid uptake but high circulating lipid levels, we found that Ffar1 (show FFAR1 Antibodies) suppresses expression of the glucose transporter Glut1 (show SLC2A1 Antibodies)
neuronal stress differentially regulates lipoprotein receptor expression in neurons, with VLDLR upregulation as a common element as a modulator of neuronal Wnt (show WNT2 Antibodies) signaling
Subretinal vascularization (SRV) in the Vldlr-/- model is associated with mistargeted neurites and that SRV is preceded by altered retinal vascular development.
VLDLR requires RasGRF1 (show RASGRF1 Antibodies)/CaMKII (show CAMK2G Antibodies) to alter dendritic spine formation.
This study demonstrated that VLDLR is expressed in distinct spatiotemporal patterns in developing mouse cerebral cortex.
LRP5 (show LRP5 Antibodies) signaling is a prerequisite for neovascularization in VLDLR knockout mice. LRP5 (show LRP5 Antibodies) may be an effective target for inhibiting intraretinal neovascularization.
Nuclear factor (erythroid-derived 2)-like 2 (show NFE2L2 Antibodies) activation-induced hepatic very-low-density lipoprotein receptor overexpression in response to oxidative stress contributes to alcoholic liver disease in mice.
Clusterin (show CLU Antibodies) is a ligand for apolipoprotein E receptor 2 (ApoER2 (show LRP8 Antibodies)) and very low density lipoprotein receptor (VLDLR) and signals via the Reelin (show RELN Antibodies)-signaling pathway.
The low density lipoprotein receptor (LDLR) gene family consists of cell surface proteins involved in receptor-mediated endocytosis of specific ligands. This gene encodes a lipoprotein receptor that is a member of the LDLR family and plays important roles in VLDL-triglyceride metabolism and the reelin signaling pathway. Mutations in this gene cause VLDLR-associated cerebellar hypoplasia. Alternative splicing generates multiple transcript variants encoding distinct isoforms for this gene.
, very low-density lipoprotein receptor
, VTG receptor
, very low density lipoprotein (VLDL)/vitellogenin receptor
, vitellogenin receptor