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The low density lipoprotein receptor (LDLR) gene family consists of cell surface proteins involved in receptor-mediated endocytosis of specific ligands. Additionally we are shipping VLDLR Antibodies (118) and VLDLR Proteins (8) and many more products for this protein.
Showing 10 out of 31 products:
These results for the first time demonstrated that SalA protected against IS/RP-induced endothelial barrier dysfunction through suppression of VLDL receptor expression
these results suggest that VLDLR functions in vivo as an HCV receptor independent of canonical CD81 (show CD81 ELISA Kits)-mediated HCV entry.
the results obtained indicate that minimal fibrin-binding structures are located within the second and third CR domains of the VLDL receptor and the presence of the fourth CR domain is required for high-affinity binding
The results of this study demonstrated the presence of reelin (show RELN ELISA Kits), its receptors VLDLR and ApoER2 (show LRP8 ELISA Kits) as well as Dab1 (show DAB1 ELISA Kits) in the ENS and might indicate a novel role of the reelin (show RELN ELISA Kits) system in regulating neuronal plasticity and pre-synaptic functions in the ENS.
these results suggested that the miR (show MLXIP ELISA Kits)-135a-VLDLR-p38 (show CRK ELISA Kits) axis may contribute to gallbladder cancer cell proliferation
study identified a novel homozygous VLDLR c.2248C>T mutation (p.Q750X) and distinctive MRI (show C7ORF49 ELISA Kits) findings in 2 siblings with ataxia (show USP14 ELISA Kits); also marked vitamin E deficiency was detected in the proband
these results identify a novel role for the VLDLR as a negative regulator of DC-mediated adaptive immune responses in HDM (show HDAC3 ELISA Kits)-induced allergic airway inflammation.
ectopic expression of HIC1 (show HIC1 ELISA Kits) in U2OS and MDA-MB-231 cell lines decreases expression of the ApoER2 (show LRP8 ELISA Kits) and VLDLR genes, encoding two canonical tyrosine kinase (show TXK ELISA Kits) receptors for Reelin (show RELN ELISA Kits).
an unusual constellation of VLDLR mutations in Cerebellar ataxia (show USP14 ELISA Kits), mental retardation and dysequilibrium syndrome 1 is reported
Results show variation in VLDLR is implicated in disordered gambling
The absence of PCSK9 (show PCSK9 ELISA Kits) results in a sex- and tissue-specific subcellular distribution of the LDLR (show LDLR ELISA Kits) and VLDLR, which is determined by estradiol levels.
In the retinas of Vldlr(-/-) mice with low fatty acid uptake but high circulating lipid levels, we found that Ffar1 (show FFAR1 ELISA Kits) suppresses expression of the glucose transporter Glut1 (show SLC2A1 ELISA Kits)
neuronal stress differentially regulates lipoprotein receptor expression in neurons, with VLDLR upregulation as a common element as a modulator of neuronal Wnt (show WNT2 ELISA Kits) signaling
Subretinal vascularization (SRV) in the Vldlr-/- model is associated with mistargeted neurites and that SRV is preceded by altered retinal vascular development.
VLDLR requires RasGRF1 (show RASGRF1 ELISA Kits)/CaMKII (show CAMK2G ELISA Kits) to alter dendritic spine formation.
This study demonstrated that VLDLR is expressed in distinct spatiotemporal patterns in developing mouse cerebral cortex.
LRP5 (show LRP5 ELISA Kits) signaling is a prerequisite for neovascularization in VLDLR knockout mice. LRP5 (show LRP5 ELISA Kits) may be an effective target for inhibiting intraretinal neovascularization.
Nuclear factor (erythroid-derived 2)-like 2 (show NFE2L2 ELISA Kits) activation-induced hepatic very-low-density lipoprotein receptor overexpression in response to oxidative stress contributes to alcoholic liver disease in mice.
Clusterin (show CLU ELISA Kits) is a ligand for apolipoprotein E receptor 2 (ApoER2 (show LRP8 ELISA Kits)) and very low density lipoprotein receptor (VLDLR) and signals via the Reelin (show RELN ELISA Kits)-signaling pathway.
The low density lipoprotein receptor (LDLR) gene family consists of cell surface proteins involved in receptor-mediated endocytosis of specific ligands. This gene encodes a lipoprotein receptor that is a member of the LDLR family and plays important roles in VLDL-triglyceride metabolism and the reelin signaling pathway. Mutations in this gene cause VLDLR-associated cerebellar hypoplasia. Alternative splicing generates multiple transcript variants encoding distinct isoforms for this gene.
, very low-density lipoprotein receptor
, VTG receptor
, very low density lipoprotein (VLDL)/vitellogenin receptor
, vitellogenin receptor