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Human Angiotensin II Type 2 Receptor ELISA Kit for Sandwich ELISA - ABIN417009
Clancy, Koblar, Golledge: Involvement of Angiotensin II Type 1 and 2 Receptors in Gelatinase Regulation in Human Carotid Atheroma in vitro. in Journal of atherosclerosis and thrombosis 2016
Suggest a gender-specific association between the AT2R -1332 A/G polymorphism and the occurrence of carotid plaque and the history of cerebrovascular insult in advanced carotid atherosclerosis.
peripheral and central arterial pressures and pulse wave augmentation indexes (AIx(P), AIx(C1), AIx(C2)), pulse wave velocity (PWV), daily urinary sodium excretion were measured and did genetic studies of AGTR1 (show AGTR1 ELISA Kits) A1166C and AGTR2 G1675A polymorphisms.
Significant overrepresentation of AT2R-1332 AA genotype in female multiple sclerosis patients was found, compared to female controls.
These data suggest that AT2R inhibits ligand-induced AT1R signaling through the PKC-dependent pathway.
Ang II (show AGT ELISA Kits)-induced upregulation of ATF3 (show ATF3 ELISA Kits) and SUMO1 (show SUMO1 ELISA Kits) in vitro and in vivo was blocked by Ang II (show AGT ELISA Kits) type I receptor antagonist olmesartan. Moreover, Ang II (show AGT ELISA Kits) induced ATF3 (show ATF3 ELISA Kits) SUMOylation at lysine 42, which is SUMO1 (show SUMO1 ELISA Kits) dependent
The A1166C polymorphism of AT1R (show AGTR1 ELISA Kits), A1675G and C3123A polymorphisms of AT2R were analyzed. The A1675G polymorphism of AT2R might be associated with preeclampsia
Ten tagging single nucleotide polymorphisms on AGTR1 (show AGTR1 ELISA Kits)/AGTR2 were genotyped for all subjects with primary aldosteronism and controls.
AT2R downregulates the expression of TGF-betaRII in human proximal tubule cells
Angiotensin II type 2 receptor is present in human semen and may be involved in the control of sperm motility and male fertility.
Ang-(1 (show ANGPT1 ELISA Kits)-7) downregulated AT1R (show AGTR1 ELISA Kits) mRNA, upregulated mRNA levels of Ang II (show AGT ELISA Kits) type 2 receptor (AT2R) and Mas (show MAS1 ELISA Kits) receptor (MasR) and p38-MAPK (show MAPK14 ELISA Kits) phosphorylation and suppressed H22 cell-endothelial cell communication
AT2R inhibits adipogenic differentiation in mesenchymal stem cells. Moreover, this inhibitory effect is associated with Wnt10b (show WNT10B ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling.
These results suggest that nerve growth factor-mediated neurite outgrowth is suppressed by AT2 receptor signaling via the nitric oxide-cyclic GMP (show NT5C2 ELISA Kits)-PKG (show PRKG1 ELISA Kits) pathway.
AT2R expressing neurons make GABAergic synapses onto AVP (show AVP ELISA Kits) neurons that inhibit AVP (show AVP ELISA Kits) neuronal activity and suppress baseline systemic AVP (show AVP ELISA Kits) levels.
Further deletion/mutation analysis revealed that multiple transcription factor binding sites in the +286/+690 region within intron 2 coordinately regulate AT2R transcription. Importantly, +286/+690 enhancer activity was suppressed in CHF mouse skeletal muscle, suggesting that AT2R expression is suppressed in CHF via inhibition of AT2R intronic enhancer activity, leading to lowered muscle regeneration.
This study suggests that deletion of AT2R decreases the expression of the beneficial ACE2/Ang-(1-7)/MasR.
Hypercholesterolemia blunts the oxidative stress and inflammatory cell recruitment elicited by hypertension in venules through a mechanism that involves AT2 receptor activation.
These results suggest that in the absence of AT2R, wound healing rate is accelerated, but yielded worse skin quality.
Bone marrow-AT2 deficiency aggravates atherosclerosis, at least in part, by eliminating the anti-atherogenic properties of macrophages elicited by LXRbeta (show NR1H2 ELISA Kits) activation.
the possible roles of AT2 receptor-mediated dopamine regulation in the pathogenesis of Binge eating disorder, was investigated.
AT2R signaling positively regulates myoblast differentiation and potentiates skeletal muscle regenerative potential.
AT1 (show AGTR1 ELISA Kits) and AT2 receptors heterodimerize and are involved in the angiotensin II effect on SERCA (show ATP2A3 ELISA Kits) in proximal kidney tubules.
AT2 receptors are positively coupled to the proliferative response of vascular smooth muscle cells to angiotensin II.
Glomerular eNOS (show NOS3 ELISA Kits) gene expression was studied during postnatal maturation and AT1 receptor (show AGTRAP ELISA Kits) inhibition.
Data suggest that AGTR2 (and angiotensin-converting enzyme (show ACE ELISA Kits)) mRNA levels are transiently up-regulated in ovarian theca cells during preovulatory period.
data suggest that G alpha(i3), Shc (show SHC1 ELISA Kits), Grb2 (show GRB2 ELISA Kits), Ras, and Raf-1 (show RAF1 ELISA Kits) link Src (show SRC ELISA Kits) to activation of MAPK (show MAPK1 ELISA Kits) and to the AT(2)-dependent increase in eNOS (show NOS3 ELISA Kits) expression in PAECs
Abundance of AGTR2 mRNA in granulosa cells was higher in healthy compared with atretic follicles, whereas in theca cells, it did not change
The protein encoded by this gene belongs to the G-protein coupled receptor 1 family, and functions as a receptor for angiotensin II. It is an intergral membrane protein that is highly expressed in fetus, but scantily in adult tissues, except brain, adrenal medulla, and atretic ovary. This receptor has been shown to mediate programmed cell death and this apoptotic function may play an important role in developmental biology and pathophysiology. Mutations in this gene are been associated with X-linked mental retardation.
angiotensin II type-2 receptor
, type-2 angiotensin II receptor
, AT2 receptor
, angiotensin II type 2 receptor
, angiotensin receptor 2
, angiotensin II receptor type 2
, angiotensin type II receptor
, Type-2 angiotensin II receptor
, angiotensin II subtype 2 receptor, AT2