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Browse our ENOS (NOS3) ELISA Kits

Full name:
Nitric Oxide Synthase 3 (Endothelial Cell) ELISA Kits (NOS3)
On www.antibodies-online.com are 99 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) ELISA Kits from 10 different suppliers available. Additionally we are shipping ENOS Antibodies (447) and ENOS Proteins (19) and many more products for this protein. A total of 579 ENOS products are currently listed.
Synonyms:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII

Top referenced ENOS ELISA Kits

  1. Human ENOS ELISA Kit for Sandwich ELISA - ABIN414413 : Ge, Mo, Januzzi, Jin, Yang, Han, Wood, Levine: B-type natriuretic peptide, vascular endothelial growth factor, endothelin-1, and nitric oxide synthase in chronic mountain sickness. in American journal of physiology. Heart and circulatory physiology 2011 (PubMed)
    Show all 7 references for ABIN414413

  2. Rat (Rattus) ENOS ELISA Kit for Sandwich ELISA - ABIN416101 : Suo, Liu, Ferreri, Zhang, Liu, Mu, Han: Impact of matrine on inflammation related factors in rat intestinal microvascular endothelial cells. in Journal of ethnopharmacology 2009 (PubMed)
    Show all 3 references for ABIN416101

  3. Mouse (Murine) ENOS ELISA Kit for Sandwich ELISA - ABIN366370 : Cheng, Jiang, Hu, Lv: Potential mechanism for endothelial progenitor cell therapy in acute myocardial infarction: Activation of VEGF- PI3K/Akte-NOS pathway. in Annals of clinical and laboratory science 2013 (PubMed)

More ELISA Kits for ENOS Interaction Partners

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS 3 gene variant rs1799983 is strongly associated with coronary artery disease

  2. The decreased levels of NOx and eNOS found in this study indicate the co-existence of endothelial dysfunction and hypertension once more.

  3. The eNOS T allele is not more prevalent among hypertensive children than among healthy ones, but it is associated with early vascular damage in children with PH, independent of metabolic abnormalities

  4. Two functionally distinct pools of eNOS in endothelium are facilitated by myoendothelial junction lipid composition.

  5. The meta-analysis showed that the polymorphism G894T of NOS3 was associated with risk of AD.

  6. Genotype frequencies are similar for both the eNOS G894T and T-786C polymorphisms in normal and high-tension glaucoma patients

  7. endothelial nitric oxide synthase (ENOS) G894T polymorphism might be a beneficial factor for high altitude adaptation through enhancing the level of nitric oxide (meta-analysis)

  8. This study failed to demonstrate an association between eNOS gene G894T polymorphism and serum NO levels in patients with pregnancy-induced hypertensive disorders.

  9. Upregulation of microRNA335 and microRNA584 contributes to the pathogenesis of severe preeclampsia through downregulation of endothelial nitric oxide synthase

  10. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR ELISA Kits) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 ELISA Kits), iNOS (show NOS2 ELISA Kits), and eNOS; up-regulation of NOS (show NOS ELISA Kits) by leptin (show LEP ELISA Kits) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 ELISA Kits) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (show NOS1 ELISA Kits) and eNOS expression and NOS (show NOS ELISA Kits) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  2. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  3. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  4. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  5. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 ELISA Kits))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  6. Fluorescence decays of fluorescently labeled CaM (show KRIT1 ELISA Kits) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

  7. Endothelial nitric oxide synthase is regulated by ERK (show MAPK1 ELISA Kits) phosphorylation at Ser602.

  8. key regulatory role of CaM (show KRIT1 ELISA Kits) involves the stabilization of structural intermediates and precise positioning of the pivot for the FMN (show FMN1 ELISA Kits) domain tethered shuttling motion to accommodate efficient and rapid electron transfer in the homodimer of eNOS.

  9. radiation-induced eNOS activation in bovine aortic endothelial cells is regulated by ATM (show ATM ELISA Kits) and HSP90 (show HSP90 ELISA Kits)

  10. Heparan sulfate is involved in both centralized and decentralized glycocalyx-mediated mechanotransduction, with GPC1 (show GPC1 ELISA Kits) acting as a centralized agent and SDC1 (show SDC1 ELISA Kits) functioning in decentralized mechanotransmission. GPC1 (show GPC1 ELISA Kits) mediates NOS3 activation.

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. VEGFR2 (show KDR ELISA Kits) activation was not affected by Slit2 (show SLIT2 ELISA Kits), but eNOS phosphorylation was diminished

  2. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 ELISA Kits) along the radial arteries in the developing placenta.

  3. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  4. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  5. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Normal and high eNOS levels are detrimental in both mild and severe cardiac pressure-overload.

  2. eNOS may be necessary to maintain podocyte integrity, especially mitochondrial function

  3. Disruption of the eNOS-NO signaling pathway exacerbates peritoneal fibrosis by delaying wound healing.

  4. BMP4 (show BMP4 ELISA Kits) has a role in inducing NOX1 (show NOX1 ELISA Kits)-dependent eNOS uncoupling in T2DM, which may promote development of novel therapeutics restoring endothelial function in T2DM

  5. The eNOS expression was induced with adipocyte differentiation and inhibition of eNOS/NO enhanced lipolysis in vitro and in vivo.

  6. Diet-induced obesity leads to l-arginine (show GATM ELISA Kits) deficiency and eNOS uncoupling in perivascular adipose tissue.

  7. Data show that the transient receptor potential vanilloid type 1 (TRPV1 (show TRPV1 ELISA Kits)) ligand- induced increase in AMP-activated protein kinase (AMPK (show PRKAA2 ELISA Kits)) and nitric oxide synthase (show NOS ELISA Kits) type III (eNOS) phosphorylation was markedly lower in TRPV1 (show TRPV1 ELISA Kits)-/- (knockout) than WT aortas.

  8. Data show that the number of S-nitrosocysteine sites was significantly reduced in the brains lacking endothelial nitric oxide synthase (eNOS(-/-)) or neuronal nitric oxide synthase (nNOS (show NOS1 ELISA Kits)(-/-)).

  9. The data identify a novel mechanism for host defense via NOS3 and suggest a potential therapeutic strategy to reduce secondary bacterial pneumonia after influenza.

  10. Provide evidence for purinergic glio-endothelial coupling during neuronal activity, highlighting the role of ATP-mediated activation of eNOS via P2Y1 (show P2RY1 ELISA Kits) receptors in functional hyperemia.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen Profile

Antigen Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) Elisa Kit
  • nitric oxide synthase 3 (endothelial cell) (nos3) Elisa Kit
  • endothelial nitric oxide synthase (23B) Elisa Kit
  • nitric oxide synthase 3, endothelial cell (Nos3) Elisa Kit
  • 2310065A03Rik Elisa Kit
  • cNOS Elisa Kit
  • EC-NOS Elisa Kit
  • ecNOS Elisa Kit
  • ENOS Elisa Kit
  • NOS Elisa Kit
  • Nos-3 Elisa Kit
  • NOS3 Elisa Kit
  • NOSIII Elisa Kit

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPECIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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