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Browse our ENOS (NOS3) ELISA Kits

Full name:
Nitric Oxide Synthase 3 (Endothelial Cell) ELISA Kits (NOS3)
On www.antibodies-online.com are 81 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) ELISA Kits from 11 different suppliers available. Additionally we are shipping ENOS Antibodies (467) and ENOS Proteins (19) and many more products for this protein. A total of 581 ENOS products are currently listed.
Synonyms:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII

Top referenced ENOS ELISA Kits

  1. Human ENOS ELISA Kit for Sandwich ELISA - ABIN414413 : Ge, Mo, Januzzi, Jin, Yang, Han, Wood, Levine: B-type natriuretic peptide, vascular endothelial growth factor, endothelin-1, and nitric oxide synthase in chronic mountain sickness. in American journal of physiology. Heart and circulatory physiology 2011 (PubMed)
    Show all 7 references for ABIN414413

  2. Rat (Rattus) ENOS ELISA Kit for Sandwich ELISA - ABIN416101 : Suo, Liu, Ferreri, Zhang, Liu, Mu, Han: Impact of matrine on inflammation related factors in rat intestinal microvascular endothelial cells. in Journal of ethnopharmacology 2009 (PubMed)
    Show all 3 references for ABIN416101

  3. Mouse (Murine) ENOS ELISA Kit for Sandwich ELISA - ABIN366370 : Cheng, Jiang, Hu, Lv: Potential mechanism for endothelial progenitor cell therapy in acute myocardial infarction: Activation of VEGF- PI3K/Akte-NOS pathway. in Annals of clinical and laboratory science 2013 (PubMed)

More ELISA Kits for ENOS Interaction Partners

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Our results demonstrated that GRP94 (show HSP90B1 ELISA Kits) is a key molecule in Hepatocellular carcinoma (HCC (show FAM126A ELISA Kits)) progression that modulates the AKT (show AKT1 ELISA Kits) pathway and eNOS levels

  2. eNOS-T786C and PAI-1 (show SERPINE1 ELISA Kits)(4G/5G) are important polymorphisms in developing Buerger's disease

  3. eNOS G894T polymorphism might protect men against erectile dysfunction risk.

  4. The G894T eNOS polymorphism was associated with increased intima-media thickness in the right carotid in patients with systemic sclerosis.

  5. The findings of the present study suggest that polymorphism in G894T position of eNOS gene might be a risk factor for ischemic stroke mainly for large vessel disease stroke subtype in North Indian population

  6. We analyzed TNFalpha (show TNF ELISA Kits), NOS 3 and MDR1 in 77 patients with MM and 77 healthy controls. The genotyping was performed with PCR and/or PCR-RFLP.

  7. We investigated NOS3 T-786C, G894T, and 4b/a polymorphisms in 102 patients with varicocele and 100 healthy controls. The 4b/a polymorphism may have a protective effect for varicocelem and G894T polymorphism may contribute to varicocele occurrence by lowering the level of NO. The higher NOS3 expression levels in the patient group may be a kind of dilator compensatory mechanism to protect vascular anatomy in varicocele.

  8. Results suggest that variants in intron 4 of NOS3, with pre-transcriptional effects as susceptibility factors, influencing the risk thalidomide embryopathy development.

  9. Results suggested that G894T and T-786C polymorphisms of the nitric oxide synthase (show NOS ELISA Kits) 3 gene, but not VNTR 4b/a, were associated with an increased risk of preeclampsia.

  10. Uric acid induced HUVEC apoptosis and endothelial dysfunction by triggering oxidative and endoplasmic reticulum stress through protein kinase C (show PKC ELISA Kits)/eNOS-mediated eNOS activity and NO production.

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR ELISA Kits) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 ELISA Kits), iNOS (show NOS2 ELISA Kits), and eNOS; up-regulation of NOS (show NOS ELISA Kits) by leptin (show LEP ELISA Kits) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 ELISA Kits) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (show NOS1 ELISA Kits) and eNOS expression and NOS (show NOS ELISA Kits) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (show AKT1 ELISA Kits)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  2. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  3. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  4. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  5. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  6. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 ELISA Kits))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  7. Fluorescence decays of fluorescently labeled CaM (show KRIT1 ELISA Kits) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

  8. Endothelial nitric oxide synthase is regulated by ERK (show MAPK1 ELISA Kits) phosphorylation at Ser602.

  9. key regulatory role of CaM (show KRIT1 ELISA Kits) involves the stabilization of structural intermediates and precise positioning of the pivot for the FMN (show FMN1 ELISA Kits) domain tethered shuttling motion to accommodate efficient and rapid electron transfer in the homodimer of eNOS.

  10. radiation-induced eNOS activation in bovine aortic endothelial cells is regulated by ATM (show ATM ELISA Kits) and HSP90 (show HSP90 ELISA Kits)

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. VEGFR2 (show KDR ELISA Kits) activation was not affected by Slit2 (show SLIT2 ELISA Kits), but eNOS phosphorylation was diminished

  2. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 ELISA Kits) along the radial arteries in the developing placenta.

  3. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  4. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  5. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Beta2AR (show ADRB2 ELISA Kits) overexpression enhances endothelial progenitor cells (EPC (show TCF21 ELISA Kits)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (show ADRB2 ELISA Kits)/Akt (show AKT1 ELISA Kits)/eNOS pathway.

  2. Through the abrogation of NAD(P)H (show NQO1 ELISA Kits) oxidase-driven eNOS uncoupling.

  3. The in vitro concordance of early Nos3(-/-) disease signatures supports the utility of iPSCs as a cellular model of developmental heart defects

  4. Findings do not support the hypothesis that reduced NO production from eNOS contributes to obesity-related adipose tissue inflammation.

  5. Normal and high eNOS levels are detrimental in both mild and severe cardiac pressure-overload.

  6. eNOS may be necessary to maintain podocyte integrity, especially mitochondrial function

  7. Disruption of the eNOS-NO signaling pathway exacerbates peritoneal fibrosis by delaying wound healing.

  8. BMP4 (show BMP4 ELISA Kits) has a role in inducing NOX1 (show NOX1 ELISA Kits)-dependent eNOS uncoupling in T2DM, which may promote development of novel therapeutics restoring endothelial function in T2DM

  9. The eNOS expression was induced with adipocyte differentiation and inhibition of eNOS/NO enhanced lipolysis in vitro and in vivo.

  10. Diet-induced obesity leads to l-arginine (show GATM ELISA Kits) deficiency and eNOS uncoupling in perivascular adipose tissue.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen Profile

Antigen Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) Elisa Kit
  • nitric oxide synthase 3 (endothelial cell) (nos3) Elisa Kit
  • endothelial nitric oxide synthase (23B) Elisa Kit
  • nitric oxide synthase 3, endothelial cell (Nos3) Elisa Kit
  • 2310065A03Rik Elisa Kit
  • cNOS Elisa Kit
  • EC-NOS Elisa Kit
  • ecNOS Elisa Kit
  • ENOS Elisa Kit
  • NOS Elisa Kit
  • Nos-3 Elisa Kit
  • NOS3 Elisa Kit
  • NOSIII Elisa Kit

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPECIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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