Browse our ENOS (NOS3) ELISA Kits

On www.antibodies-online.com are 97 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) ELISA Kits from 14 different suppliers available. Additionally we are shipping ENOS Antibodies (478) and ENOS Proteins (23) and many more products for this protein. A total of 612 ENOS products are currently listed.
Synonyms:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII
list all ELISA KIts Gene Name GeneID UniProt
NOS3 4846 P29474
NOS3 18127 P70313
NOS3 24600 Q62600

ENOS (NOS3) ELISA Kits by Reactivity

More ELISA Kits for ENOS Interaction Partners

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. In the subgroup analysis based on ethnicity, both T786C and 4a4b eNOS polymorphisms could influence the risk of male infertility in Asian and Caucasian.[meta-analysis].

  2. Phosphocreatine exerts an antiapoptotic effect in HUVECs exposed to oxidative stress by methylglyoxal through the mitochondrial pathway and the modulation of PI3K (show PIK3CA ELISA Kits)/Akt (show AKT1 ELISA Kits)/eNOS and NF-kappaB (show NFKB1 ELISA Kits) signaling.

  3. NOS2 rs2779248, NOS2 rs1137933, and NOS3 rs3918188 genetic polymorphisms are potentially related to the susceptibility to type 2 diabetes mellitus (T2DM), and the rs1800783 polymorphism might be considered as genetic risk factors for diabetic nephropathy.

  4. This study determined the genetic polymorphisms of the eNOS (Glu298Asp) and Cav-1 (show CAV1 ELISA Kits) (G14713A and T29107A) genes in association with susceptibility risk in patients who had suffered from a large artery atherosclerotic stroke. It demonstrated that the eNOS Glu298Asp in combination with 14713A/29107A at the CAV1 (show CAV1 ELISA Kits) locus was associated with a significant risk of an large artery atherosclerotic stroke.

  5. The eNOS T786C polymorphism was associated with ISR in Chinese Han patients treated with DES (show DES ELISA Kits). Genotyping may be helpful to identify patients with higher risks of ISR after drug-eluting stent implantation.

  6. Meta-analysis. eNOS gene G894T polymorphism was related to pregnancy-induced hypertension risk, especially for Asians.

  7. Data suggest that, in T-lymphocytes, nitric oxide generated by eNOS S-nitrosylates Cys374 on ACTB (show ACTB ELISA Kits) and thus regulates activation/recruitment of PRKCQ (show PRKCQ ELISA Kits) at immune synapse; S-nitrosylation of beta-actin (show ACTB ELISA Kits) impairs actin binding to PFN1 (show PFN1 ELISA Kits) and regulates protein transport in lamellipodia. (eNOS = nitric oxide synthase (show NOS ELISA Kits) 3; ACTB (show ACTB ELISA Kits) = beta-actin (show ACTB ELISA Kits); PRKCQ (show PRKCQ ELISA Kits) = protein kinase C-theta (show PRKCQ ELISA Kits); PFN1 (show PFN1 ELISA Kits) = profilin-1 (show PFN1 ELISA Kits))

  8. The GG eNOS allele showed greater susceptibility to atherosclerosis.

  9. NOS3 TT894 genotype protected against doxorubicin cardiotoxicity in survivors of childhood acute lymphoblastic leukemia.

  10. These findings indicated that miR (show MLXIP ELISA Kits)-126 protects HCMECs from H/R-induced injury and inflammatory response by activating the PI3K (show PIK3CA ELISA Kits)/Akt (show AKT1 ELISA Kits)/ eNOS signaling pathway.

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR ELISA Kits) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 ELISA Kits), iNOS (show NOS2 ELISA Kits), and eNOS; up-regulation of NOS (show NOS ELISA Kits) by leptin (show LEP ELISA Kits) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 ELISA Kits) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (show NOS1 ELISA Kits) and eNOS expression and NOS (show NOS ELISA Kits) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (show CTNNB1 ELISA Kits) and link eNOS-derived NO to the modulation by VEGF (show VEGFA ELISA Kits) of Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits)-induced endothelial cell proliferation.

  2. Endothelial cell autophagy maintains shear stress-induced nitric oxide generation via glycolysis-dependent ATP/P2Y1 receptor (show P2RY1 ELISA Kits) signaling to endothelial nitric oxide synthase.

  3. TNFalpha (show TNF ELISA Kits)reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 binding.

  4. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (show AKT1 ELISA Kits)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  5. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  6. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  7. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  8. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  9. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 ELISA Kits))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  10. Fluorescence decays of fluorescently labeled CaM (show KRIT1 ELISA Kits) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Kidneys from circulation-restricted fetuses showed endothelial nitric oxide synthase phosphorylation inhibition.

  2. Antidiabetic drug pioglitazone protects the heart via activation of endothelial nitric oxide synthase (eNOS/Nos3) pathway in a rabbit model of myocardial infarction.

  3. VEGFR2 (show KDR ELISA Kits) activation was not affected by Slit2 (show SLIT2 ELISA Kits), but eNOS phosphorylation was diminished

  4. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 ELISA Kits) along the radial arteries in the developing placenta.

  5. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  6. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  7. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. these data suggest that CD NOS3 may be involved in the diuretic response to a water load in a sex-specific manner; the mechanism of this effect remains to be determined.

  2. CAV1 (show CAV1 ELISA Kits) KO mice have elevated IOP and reduced conventional outflow facility when compared with WT mice. CAV2 (show CAV2 ELISA Kits) expression was absent in CAV1 (show CAV1 ELISA Kits) KO mice, but we observed increased expressions of eNOS

  3. TNF-alpha (show TNF ELISA Kits) does not regulate eNOS activity in murine endothelial cells through serine 116 phosphorylation and Pin1 (show PIN1 ELISA Kits) binding.

  4. The eNOS expressed in smooth muscle cells in FAs (show FAS ELISA Kits) attenuates alpha-adrenergic vasoconstriction.

  5. Thioredoxin (show TXN ELISA Kits)-mediated deglutathionylation of eNOS in the coronary artery in vivo protected against reperfusion injury, even in the presence of normal levels of glutathione.

  6. Eph (show EPHA1 ELISA Kits)-B4 stimulates eNOS phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS activity in vivo.

  7. Loss of endothelial NO plays an important role in the generation of p25 (show CDK5R1 ELISA Kits) and resulting tau phosphorylation in neuronal tissue. Endothelial nitric oxide synthase (eNOS)-deficient (eNOS-/-) mice display increased levels of p25 (show CDK5R1 ELISA Kits), an aberrant activator of cyclin-dependent kinase 5 (show CDK5 ELISA Kits), which is one of the primary kinases responsible for tau hyperphosphorylation, and a statistically higher p25/p35 (show CDK5R1 ELISA Kits) ratio.

  8. Pulse pressure finely regulates eNOS, controlling cerebral artery reactivity.

  9. TNF-alpha (show TNF ELISA Kits) induces vascular insulin (show INS ELISA Kits) resistance by mechanisms that involve positive modulation of PTEN and inhibition of Akt (show AKT1 ELISA Kits)/eNOS/NO signaling.

  10. Beta2AR (show ADRB2 ELISA Kits) overexpression enhances endothelial progenitor cells (EPC (show TCF21 ELISA Kits)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (show ADRB2 ELISA Kits)/Akt (show AKT1 ELISA Kits)/eNOS pathway.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen Profile

Antigen Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) Elisa Kit
  • nitric oxide synthase 3 (endothelial cell) (nos3) Elisa Kit
  • endothelial nitric oxide synthase (23B) Elisa Kit
  • nitric oxide synthase 3, endothelial cell (Nos3) Elisa Kit
  • 2310065A03Rik Elisa Kit
  • cNOS Elisa Kit
  • EC-NOS Elisa Kit
  • ecNOS Elisa Kit
  • ENOS Elisa Kit
  • NOS Elisa Kit
  • Nos-3 Elisa Kit
  • NOS3 Elisa Kit
  • NOSIII Elisa Kit

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPECIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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