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eNOS (show NOS3 Proteins) does not play an important role in this cerebrocortical blood flow redistribution after carotid artery occlusion.
there were significant decreases in intercellular adhesion molecules 1 (ICAM1 (show ICAM1 Proteins)), ezrin (EZR (show EZR Proteins)), mitogen-activated protein kinase kinase 2 (MAP2K2 (show MAP2K2 Proteins)), and nitric oxide synthase 3 (NOS3) gene expressions in metabolic syndrome patients.
data can be used to further elucidate the role of NANOS3 and DAZL (show DAZL Proteins) in germ cell development both in vitro and in vivo
Activation of eNOS (show NOS3 Proteins) by HDL (show HSD11B1 Proteins) is decreased in MetS (show ETV3 Proteins) patients before the appearance of diabetes mellitus and that sphingosine-1-phosphate depletion of HDL (show HSD11B1 Proteins) is the main factor responsible for this defect.
The thrombotic predisposition to arterial ischemia of the lower limbs in type 2 diabetic patients is assessed by evaluating polymorphisms in the three major platelet membrane adhesion receptors and endothelial nitric oxide synthase (show NOS3 Proteins).
In conclusion, exenatide significantly improves coronary endothelial function in patients with newly diagnosed type 2 diabetes. The effect may be mediated through activation of AMPK (show PRKAA1 Proteins)/PI3K (show PIK3CA Proteins)-Akt (show AKT1 Proteins)/eNOS (show NOS3 Proteins) pathway via a GLP-1R (show GLP1R Proteins)/cAMP-dependent mechanism.
The downregulation of beta-arrestins 1/2 in saphenous vein endothelial cells (SVECs) prevented the shear stress-induced rise in levels of phosphorylation of Akt (show AKT1 Proteins) and endothelial nitric oxide synthase (eNOS (show NOS3 Proteins), Serine 1177).
PGC-1alpha overexpression improves AngII-induced eNOS (show NOS3 Proteins) dysfunction via activated PI3K (show PIK3CA Proteins)/Akt (show AKT1 Proteins) pathway, impaired PP2A (show PPP2R4 Proteins) activity and reduced PP2A (show PPP2R4 Proteins)-A/eNOS (show NOS3 Proteins) association.
eNOS (show NOS3 Proteins) G894T and T-786C SNPs were both significantly correlated with hypertension. Additionally, the T allele of G894T SNP and C allele of T-786C SNP may serve as potential biological markers for hypertension susceptibility in Asians. [Meta-Analysis}
Results suggest a relation between different endothelial nitric oxide synthase (show NOS3 Proteins) geno-types and risk of acute rejection episodes in liver transplant recipients.
NANOS3 mutation is one possible cause of primary ovarian insufficiency.
Study suggests that Nanos3-3'UTR has an essential role in translational control in embryo.
Report distinct posttranscriptional regulations in primordial germ cells soon after their specification, for which RNA-binding proteins such as NANOS3 would play critical functions.
cloning and functional analyses of nanos2 (show NANOS2 Proteins) and nanos3; demonstrated an evolutionarily conserved function of nanos proteins in germ cell development [nanos2 (show NANOS2 Proteins)] [nanos3]
eNOS (show NOS3 Proteins) is the main isoform influencing the initiation of caerulein-induced AP. eNOS (show NOS3 Proteins)-derived NO exerts a protective effect through actions on nonacinar cell types, most likely endothelial cells, to produce greater pancreatic microvascular blood flow.
Potential role of Nanos3 in maintaining the undifferentiated spermatogonia population is reported.
Results suggest that Nanos3 maintains the germ cell lineage by suppressing both Bax (show BAX Proteins)-dependent and Bax (show BAX Proteins)-independent apoptotic pathways.
detection of the germ cell-specific proteins Nanos2 (show NANOS2 Proteins) and Nanos3 as markers of spermatogonia has enabled the clear dissection of complex populations of these cells as Nanos2 (show NANOS2 Proteins) was recently shown to be involved in the maintenance of stem cells
Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.
nanos homolog 3
, nanos homolog 3 (Drosophila)
, NOS type III
, constitutive NOS
, endothelial NOS
, nitric oxide synthase, endothelial
, endothelial nitric oxide synthase 3