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Rat (Rattus) TGFB1 ELISA Kit for Sandwich ELISA - ABIN416258
Cao, Deng, Wei, Yang, Su, Wei, Xu, Yu et al.: Incorporating pTGF-?1/calcium phosphate nanoparticles with fibronectin into 3-dimensional collagen/chitosan scaffolds: efficient, sustained gene delivery to stem cells for chondrogenic ... in European cells & materials 2012
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Human TGFB1 ELISA Kit for Sandwich ELISA - ABIN414922
Zhang, Long, Sun, Wang, Li, Wu, Guo, Li, Niu, Li, Liu, Mei et al.: Evidence for the complementary and synergistic effects of the three-alkaloid combination regimen containing berberine, hypaconitine and skimmianine on the ulcerative colitis rats induced by ... in European journal of pharmacology 2010
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Mouse (Murine) TGFB1 ELISA Kit for Sandwich ELISA - ABIN415536
Obrador, Benlloch, Pellicer, Asensi, Estrela: Intertissue flow of glutathione (GSH) as a tumor growth-promoting mechanism: interleukin 6 induces GSH release from hepatocytes in metastatic B16 melanoma-bearing mice. in The Journal of biological chemistry 2011
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Human TGFB1 ELISA Kit for Sandwich ELISA - ABIN625094
Zou, Zou, Zhao, Li, Ran: Nicotine-induced epithelial-mesenchymal transition via Wnt/β-catenin signaling in human airway epithelial cells. in American journal of physiology. Lung cellular and molecular physiology 2013
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Human TGFB1 ELISA Kit for Sandwich ELISA - ABIN365402
A Hamid, Mohamed Ali, Yusof, George: Platelet-rich plasma (PRP): an adjuvant to hasten hamstring muscle recovery. A randomized controlled trial protocol (ISCRTN66528592). in BMC musculoskeletal disorders 2012
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Human TGFB1 ELISA Kit for Sandwich ELISA - ABIN1889382
Mantel, Schmidt-Weber: Transforming growth factor-beta: recent advances on its role in immune tolerance. in Methods in molecular biology (Clifton, N.J.) 2010
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Mouse (Murine) TGFB1 ELISA Kit for Sandwich ELISA - ABIN365456
Hirata, Arima, Fukushima, Sugiyama, Tokuhisa, Fukuda: Leukotriene C4 aggravates bleomycin-induced pulmonary fibrosis in mice. in Respirology (Carlton, Vic.) 2013
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Mouse (Murine) TGFB1 ELISA Kit for Sandwich ELISA - ABIN411355
Chen, Lin, Li: Ganoderma lucidum polysaccharides reduce methotrexate-induced small intestinal damage in mice via induction of epithelial cell proliferation and migration. in Acta pharmacologica Sinica 2011
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Chicken TGFB1 ELISA Kit for Sandwich ELISA - ABIN364782
Zhang, Huang, Li, Sui, Wang, Liu, Xu, Yan, Song, Li: Identification and Molecular Characterization of Microneme 5 of Eimeria acervulina. in PLoS ONE 2014
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Rat (Rattus) TGFB1 ELISA Kit for Sandwich ELISA - ABIN411354
Tu, Qin, Dong, Lu, Guan: Effects of Panax notoginoside on the expression of TGF-β1 and Smad-7 in renal tissues of diabetic rats. in Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban 2011
Show all 9 Pubmed References
TGF-beta1 regulated pAKT (show AKT1 ELISA Kits) and IFNgamma expressions were associated with epithelial cell survival in rhesus macaque colon explants and suggest a potential role of mucosal TGF-beta1 in regulating intestinal homeostasis and EC integrity.
These findings suggest that FBLN5 (show FBLN5 ELISA Kits) may interfere with choroidal neovascularization by downregulating VEGF (show VEGFA ELISA Kits), CXCR4 (show CXCR4 ELISA Kits), and TGFB1 expression and inhibiting choroidl endothelial cell proliferation.
SIV infection of rhesus macaques results in the emergence of IL-17 (show IL17A ELISA Kits)-expressing cells during the acute phase. This subpopulation appears at day 14 postinfection concomitantly with an increase in TGF-beta and IL-18 (show IL18 ELISA Kits) expression.
SIV-infected macaques exhibiting progression to AIDS displayed greater expression of TGF-beta and indoleamine 2,3 dioxygenase in CD8 (show CD8A ELISA Kits)+ T cells from mesentric lymph nodes.
transforming growth factor beta (TGFbeta) is required for hematopoietic progenitor cell specification. The requirement for TGFbeta is two fold and sequential: autocrine via Tgfbeta1a and Tgfbeta1b produced in the endothelial cells themselves, followed by a paracrine input of Tgfbeta3 from the notochord, suggesting that the former programs the hemogenic endothelium and the latter drives endothelial-to-hematopoietic tran...
The fine tuning of TGF-beta signaling derives from positive and negative control by Ldb2a (show LDB2 ELISA Kits).
TGFbeta1a regulates zebrafish posterior lateral line formation via Smad5 (show SMAD5 ELISA Kits) mediated pathway.
PCSK7 (show PCSK7 ELISA Kits) is essential for zebrafish development and regulates the expression and proteolytic cleavage of TGFbeta1a.
TGFbeta signaling orchestrates the beneficial interplay between scar-based repair and cardiomyocyte-based regeneration to achieve complete heart regeneration.
TGFbeta1 plays a role in zebrafish keratocyte migration.
data presented show that Zili (show PIWIL2 ELISA Kits) suppresses TGF-beta signaling by physically associating with Smad4 (show SMAD4 ELISA Kits) and preventing the formation of Smad2 (show SMAD2 ELISA Kits)/3/4 and Smad1 (show SMAD1 ELISA Kits)/5/9/4 complexes
TGF-beta1 acts at multiple sites, including LH receptor (show LHCGR ELISA Kits), 20beta-HSD (show HAL ELISA Kits) and membrane progestin receptor-beta (show PAQR8 ELISA Kits), to inhibit zebrafish oocyte maturation
These data suggest Pez (show PTPN14 ELISA Kits) plays a crucial role in organogenesis by inducing TGFbeta and epithelial-mesenchymal transition.
Data show that Rock2 (show ROCK2 ELISA Kits) acts as a negative regulator of the TGFbeta signaling pathway.
These data indicate that TGF-beta signaling is crucial for the function of the transition zone, which in turn may affect the regulation of cilia length.
R-Smads are the key components of TGFbeta beta signals in germ layer induction. SCP3 (show SYCP3 ELISA Kits) serves as a vegetally enriched, intrinsic factor (show GIF ELISA Kits) to ensure a prepared status of Smads for their activation.
the present in vitro system, which permits not only the cell contraction-mediated cell sorting but also the TGF-b-directed mesodermal induction such as cartilage formation, may fairly reflect the embryogenesis in vivo.
Loss of XTgfbi impaired blastopore formation and dorsal tissue morphogenesis.
TGF-beta signaling has a role in nuclear localization of transcription factor Smad4 (show SMAD4 ELISA Kits)
sortilin (show SORT1 ELISA Kits) negatively regulates TGF-beta signaling by diverting trafficking of precursor proteins to the lysosome during transit through the biosynthetic pathway
CRT (show SLC6A8 ELISA Kits) regulates TGF-beta1-induced-EMT (show ITK ELISA Kits) through modulating Smad (show SMAD1 ELISA Kits) signaling
The results suggest that selective RNA decay via TGF-beta and BMP4 (show BMP4 ELISA Kits) signaling is critical for specifying the developmental fate of specific human embryonic cell lineages.
TGF-beta1 levels were found significantly higher in the hemodialysis patients than those of the control groups.
Smad7 (show SMAD7 ELISA Kits) expression in necrotizing enterocolitis macrophages interrupts TGF-beta signaling and promotes NF-kappaB (show NFKB1 ELISA Kits)-mediated inflammatory signaling in these cells through increased expression of IKK-beta (show IKBKB ELISA Kits)
P311 (show C5orf13 ELISA Kits) is a novel TGFbeta1/Smad (show SMAD1 ELISA Kits) signaling-mediated regulator of transdifferentiation in epidermal stem cells during cutaneous wound healing.
Paroxysmal atrial fibrillation is associated with a higher TGF-beta1 level. Lower TGF-beta1 level in AF patients could be a cause of recurrent AF after catheter ablation.
This study demonstrated that the Indian subjects TGFB1 rs1800469 were associated with higher and lower morphine use.
UCN (show UCN ELISA Kits) counteracted TGF-beta-mediated cPLA2 (show PLA2G4A ELISA Kits) expression and activation, thereby contributing to TGF-beta-mediated mitoinhibition of VSMCs
High TGFB1 expression is associated with prostate cancer.
Cardiac overexpression of TGF-beta1 led to an increase in fibrosis and myocyte size in the atria, and to progressive P-wave prolongation, increasing atrial fibrillation susceptibility.
Activated TGF-beta signaling rescued miR (show MYLIP ELISA Kits)-143-reduced FSHR (show FSHR ELISA Kits) and intracellular signaling molecules, and miR (show MYLIP ELISA Kits)-143-induced porcine granulosa cell apoptosis.
TGF-beta1-induced epithelial-myofibroblast plasticity is FAK (show PTK2 ELISA Kits)- dependent, whereas TGF-beta1-induced apoptosis is favored when FAK (show PTK2 ELISA Kits) signaling is inhibited.
this study shows that TGF-beta1 protects intestinal integrity and influences Smad (show SMAD1 ELISA Kits) and MAPK (show MAPK1 ELISA Kits) signal pathways in intestinal epithelium cells after TNF-alpha (show TNF ELISA Kits) challenge
this study shows that anemonin may ameliorate LPS (show IRF6 ELISA Kits)-induced intestinal injury and improve restoration by regulating the TGF-b1 and EGFR (show EGFR ELISA Kits) signaling pathways
The results indicated that TGF-beta-1 was associated with the restoration of intestinal morphology and barrier function following weaning stress.
Data (including data from in vitro and in vivo experiments) suggest that day 14 elongated conceptus secretes proteins that up-regulate TGFbeta1 mRNA and TGFbeta1 expression in endometrium; TGFbeta1 may be important during pregnancy maintenance.
The effects of semen, spermatozoa in extender, or extender alone on the expression of TGF-beta1, IL-10 (show IL10 ELISA Kits), and IL-6 (show IL6 ELISA Kits) in ovarian follicles are reported.
Dietary (1,3/1,6)-beta-D-glucan reduced the mRNA expression of transforming growth factor (TGF) beta2 (show TGFB2 ELISA Kits) and tended to reduce the mRNA expression of TGF-beta1 in lung tissue of neonatal piglet.
TGF-beta1, via TGF-beta1 receptor I and p38 MAPK (show MAPK14 ELISA Kits) signaling, reduces CFTR (show CFTR ELISA Kits) expression to impair CFTR (show CFTR ELISA Kits)-mediated anion secretion, which would likely compound the effects associated with mild CFTR (show CFTR ELISA Kits) mutations and ultimately would compromise male fertility.
High yield isolation of BMP-2 (show BMP2 ELISA Kits) from bone and in vivo activity of a combination of BMP-2 (show BMP2 ELISA Kits)/TGF-beta1.
TGF-beta1 modulates the expression of syndecan-4 (show SDC4 ELISA Kits) in cultured vascular endothelial cells in a biphasic manner.
Taken together, Staphylococcus aureus induces TGF-beta1 and bFGF (show FGF2 ELISA Kits) expression through the activation of AP-1 (show JUN ELISA Kits) and NF-kappaB (show NFKB1 ELISA Kits) in bovine mammary gland fibroblasts.
The results identify TGFB1 and ESRRA (show ESRRA ELISA Kits) as likely transcriptional regulators of rumen epithelial development and energy metabolism, respectively, and provide targets for modulation of rumen development and function in the growing calf.
the combined treatment with TGF-beta1 and BMP-7 (show BMP7 ELISA Kits) or treatment first with TGF-beta1 followed by BMP-7 (show BMP7 ELISA Kits) was more effective than other treatment groups in both chondrogenic differentiation and SZP (show PRG4 ELISA Kits) secretion.
Tenascin-X (show TNXB ELISA Kits) promotes activation of latent TGF-beta1 and subsequent epithelial to mesenchymal transition in mammary epithelial cells.
a detailed computational model for TGF-beta signalling that incorporates elements of previous models together with crosstalking between Smad1 (show SMAD1 ELISA Kits)/5/8 and Smad2 (show SMAD2 ELISA Kits)/3 channels through a negative feedback loop dependent on Smad7 (show SMAD7 ELISA Kits).
Endogenous TGF-beta1 became more bioactive following activation of the transgene protein product in chondrocytes.
A novel peptide, P2K, regulating TGF-beta1 signaling had an anabolic effect on bovine intervertebral disc cells and rabbit degenerated discs.
Data show that TGF-beta pathways operate during ovarian fetal development, and fibrillin 3 (show FBN3 ELISA Kits) is highly expressed at a critical stage early in developing human and bovine fetal ovaries.
Role of TGF-beta1 and TNF-alpha (show TNF ELISA Kits) in IL-1beta (show IL1B ELISA Kits) mediated activation of proMMP-9 in pulmonary artery smooth muscle cells: involvement of an aprotinin sensitive protease.
PAR1 (show F2R ELISA Kits) in its inactive unligated state functions as a scaffold for TGFbetaRII to downregulate TGF-beta signaling, and thereby promote embryonic stem cell transition to functional endothelial cells.
HSP22 (show HSPB8 ELISA Kits) functions as a negative regulator in the TGF-beta-stimulated migration of osteoblasts.
Lnc-LFAR1 binds directly to Smad2 (show SMAD2 ELISA Kits)/3 and promotes transcription of TGFbeta, Smad2 (show SMAD2 ELISA Kits), Smad3 (show SMAD3 ELISA Kits), Notch2 (show NOTCH2 ELISA Kits) and Notch3 (show NOTCH3 ELISA Kits) which, in turn, results in TGFbeta and Notch (show NOTCH1 ELISA Kits) pathway activation.
how that limiting CCR2 (show CCR2 ELISA Kits)(+) monocytic myeloid-derived suppressor cells accumulation reduces the pulmonary contents of TGF-beta1, TIMP-1 (show TIMP1 ELISA Kits) and collagen after silica treatment
RORgamma mediates epithelial-mesenchymal transition of hepatocytes during hepatic fibrosis facilitated by TGFbeta1.
these data unravel the previously unrecognized role of TGF-beta1 in promoting Treg viability, coinciding with the pronounced immunomodulatory role of these cells during later phase of oropharyngeal candidiasis infection
findings define a novel physiological function of Shp2 (show PTPN11 ELISA Kits) in TGF-beta1/MMP12 (show MMP12 ELISA Kits)-dependent emphysema, adding insights into potential etiologies for this chronic lung disorder.
TGF-beta and PPARalpha (show PPARA ELISA Kits) signaling pathways are involved in radiation-induced heart fibrosis, metabolic dysregulation, and impaired heart contractility, a pathophysiological condition that is often observed in patients that received high radiation doses in thorax.
Data show that Clostridium difficile Toxin A induces TGF-beta1 signaling pathway activation and suggest that this pathway might play a protective role against the effect of C. difficile-toxin.
Data indicate that cardiac contractility modulation (CCM) therapy exerted protective effects against myocardial fibrosis potentially by inhibiting TGF-beta1/Smad3 (show SMAD3 ELISA Kits) signaling pathway in chronic heart failure .
study suggested that TGF-beta1/Smad3/smad7 is a major pathway which plays an important role in the regulation of the IUA and specific inhibitor of Smad3 (SIS3) may provide a new therapeutic strategy for IUA.
cell therapy promoted TGF-beta1 expression in nucleus pulposus, leading to anti-inflammatory effects via the inhibition of NF-kappaB (show NFKB1 ELISA Kits), and the amelioration of disc degradation.
that prenatal tracheal occlusion increases TGF-beta/Rho kinase (show ROCK1 ELISA Kits) pathway, myofibroblast differentiation, and matrix deposition in neonatal rabbit and human congenital diaphragmatic hernia lungs
observation. Based on the above results, we conclude that TGF-beta1-immobilized PLGA-gelatin scaffold seeded with ASCs considerably enhances the quality of the tissue-engineered cartilage, therefore, advancing the field of cartilage tissue engineering
Smad7 (show SMAD7 ELISA Kits) plays a crucial role in antagonizing epithelial-mesenchymal transition induced by TGFbeta signaling and is a Notch (show NOTCH1 ELISA Kits) signaling target in limbal epithelial stem cells.
the role of IL-10 in inhibited allograft rejection may be associated with CD4+CD25+ regulatory T cells and IL-10, and may be independent of TGF-b
After NOTCH1 (show NOTCH1 ELISA Kits) knockdown and TGFB1 stimulation, rabbit mesenchymal stem cells expressed higher levels of proteoglycan (show Vcan ELISA Kits) and collagen II.
TGF-beta1 gene transcription significantly correlates with the surgical vaginal and dermal wound closure rate.
TGF beta is involved in the pathogenesis of tympanosclerosis.
There was significantly higher expression of TGF-beta1 and MMP-9 (show MMP9 ELISA Kits) in nasal mucosa of experimental allergic rhinitis guinea pigs than in controls.
Within the limitations of the study design, production of COMP (show COMP ELISA Kits) during healing of skin wounds does not appear to be influenced by wound type or anatomic site, nor does it appear to be correlated with TGF-beta1 concentrations.
Peritoneal TGF-beta(1) concentration was higher in horses with severe gastrointestinal diseases, in horses with an altered peritoneal fluid, and in nonsurvivors.
This gene encodes a member of the transforming growth factor beta (TGFB) family of cytokines, which are multifunctional peptides that regulate proliferation, differentiation, adhesion, migration, and other functions in many cell types. Many cells have TGFB receptors, and the protein positively and negatively regulates many other growth factors. The secreted protein is cleaved into a latency-associated peptide (LAP) and a mature TGFB1 peptide, and is found in either a latent form composed of a TGFB1 homodimer, a LAP homodimer, and a latent TGFB1-binding protein, or in an active form composed of a TGFB1 homodimer. The mature peptide may also form heterodimers with other TGFB family members. This gene is frequently upregulated in tumor cells, and mutations in this gene result in Camurati-Engelmann disease.
transforming growth factor beta1
, transforming growth factor, beta 1
, transforming growth factor, beta-induced, 68kDa
, transforming growth factor-beta-induced protein ig-h3-like
, transforming growth factor beta-1-like
, transforming growth factor beta 4
, transforming growth factor beta-1
, TGF-beta 1 protein
, latency-associated peptide
, transforming growth factor, beta 1 (Camurati-Engelmann disease)
, transforming growth factor-beta-1
, TGF-beta 1
, transforming growth factor-beta 1
, transforming growth factor, beta-1
, transforming growth factor beta 1
, regulatory protein
, transforming growth factor-beta
, tgf beta
, tgf-beta 5
, transforming gorwth factor-Beta5
, transforming growth factor-B5
, transforming growth factor-beta 5
, Transforming growth factor beta-1