KIR3DL1 antibody (FITC)
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- Target See all KIR3DL1 Antibodies
- KIR3DL1 (Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1))
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Reactivity
- Human
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Host
- Mouse
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Clonality
- Monoclonal
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Conjugate
- This KIR3DL1 antibody is conjugated to FITC
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Application
- Western Blotting (WB), Flow Cytometry (FACS), Immunoprecipitation (IP)
- Purification
- The antibody was purified by affinity chromatography, and conjugated with FITC under optimal conditions. The solution is free of unconjugated FITC.
- Clone
- DX9
- Isotype
- IgG1 kappa
- Top Product
- Discover our top product KIR3DL1 Primary Antibody
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anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (AA 206-340) antibody
KIR3DL1 Reactivity: Human WB, ELISA, IHC, FACS, ICC, Neut Host: Mouse Monoclonal 2C3B6 unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (AA 81-336) antibodyKIR3DL1 Reactivity: Human WB, ELISA, IHC, IF Host: Rabbit Polyclonal unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (AA 22-340) antibodyKIR3DL1 Reactivity: Human, Rat WB, ELISA, IHC, FACS, ICC, Neut Host: Mouse Monoclonal 6D9A4 unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (AA 22-340) antibodyKIR3DL1 Reactivity: Human, Rat, Monkey WB, ELISA, IHC, FACS, ICC, Neut Host: Mouse Monoclonal 6D9F6 unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (N-Term) antibodyKIR3DL1 Reactivity: Human WB, ELISA, IHC Host: Rabbit Polyclonal unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (Internal Region) antibodyKIR3DL1 Reactivity: Human WB, ELISA, IHC, ICC, IF Host: Rabbit Polyclonal unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) antibodyKIR3DL1 Reactivity: Human, Mouse WB Host: Rabbit Polyclonal unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (AA 251-350) antibodyKIR3DL1 Reactivity: Human WB, IHC (p), IHC (fro) Host: Rabbit Polyclonal unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (AA 1-382) antibodyKIR3DL1 Reactivity: Human WB Host: Rabbit Polyclonal unconjugated
anti-Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1) (Center) antibodyKIR3DL1 Reactivity: Human WB Host: Rabbit Polyclonal unconjugated
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- Application Notes
- Optimal working dilution should be determined by the investigator.
- Restrictions
- For Research Use only
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- Buffer
- Phosphate-buffered solution, pH 7.2, containing 0.09 % sodium azide and 0.2 % (w/v) BSA .
- Preservative
- Sodium azide
- Precaution of Use
- This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
- Handling Advice
- Protect from prolonged exposure to light. Do not freeze.
- Storage
- 4 °C
- Storage Comment
- The antibody solution should be stored undiluted between 2°C and 8°C.
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- Target
- KIR3DL1 (Killer Cell Immunoglobulin-Like Receptor, three Domains, Long Cytoplasmic Tail, 1 (KIR3DL1))
- Alternative Name
- CD158e1 (KIR3DL1 Products)
- Background
- CD158e1, also known as NKB1, is a 70 kD member of the immunoglobulin superfamily that is expressed on a subset of natural killer cells and T cells at varying levels among individuals. NKB1 is a type I membrane protein containing two immunoglobulin C2-type domains. The interaction of NKB1 with specific HLA-B antigens on a target cell (the HLA-Bw4 allele, for example) inhibits cytotoxicity and prevents target cell lysis and death. The interactions between KIR and MHC class I are thought to be important in NK and T cell regulation following antigen stimulation. The absence of ligands for KIRs may lower the threshold for activation through activating receptors and increase inflammation and susceptibility to autoimmune disease.
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