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Human BID Primary Antibody for ELISA - ABIN365900
Lim, Loh, Ting, Bradshaw, Zeenathul: Antiproliferation and induction of caspase-8-dependent mitochondria-mediated apoptosis by ?-tocotrienol in human lung and brain cancer cell lines. in Biomedicine & pharmacotherapy 2014
Stage-specific expression of TNFalpha (show TNF ELISA Kits) regulates bad/bid-mediated apoptosis and RIP1 (show RALBP1 ELISA Kits)/ROS (show ROS1 ELISA Kits)-mediated secondary necrosis in Birnavirus-infected fish cells.
Hyperosmotic Shock Engages Two Positive Feedback Loops through Caspase-3 (show CASP3 ELISA Kits)-dependent Proteolysis of JNK1 (show MAPK8 ELISA Kits)-2 and Bid.
tail regression at metamorphosis implicates an apoptotic pathway inducible by T(3) hormone in an organ autonomous manner and involving the cell death executioners BID and Caspases-2 and -8
Here, we identified and characterized the Xenopus homologs of caspase-10 (show CASP10 ELISA Kits) (xCaspase-10beta), a novel initiator caspase (show CASP3 ELISA Kits), and Bid (xBid), a BH3-only (show BBC3 ELISA Kits) molecule of the Bcl-2 (show BCL2 ELISA Kits) family involved in both the extrinsic and intrinsic pathways.
diubiquitylation is a specific feature of xBid.
Results from genome-wide DNA methylation (show HELLS ELISA Kits), functional network analysis and pyrosequencing, show selective CpG sites (NOS1AP (show NOS1AP ELISA Kits), BID, and GABRB1 (show GABRB1 ELISA Kits)) differentially methylated in smokers and chronic obstructive pulmonary disease patients compared to nonsmokers.
The adenovirus-mediated truncated Bid overexpression induced by the Cre/LoxP system can effectively eliminate CD133+ ovarian cancer stem cells, representing a novel therapeutic strategy for the treatment of ovarian cancer.
MiR (show MLXIP ELISA Kits)-20a-directed regulation of BID in colorectal cancer.
the results establish that cleavage by caspase 8 (show CASP8 ELISA Kits) and the subsequent association with the outer mitochondrial membrane are two critical events that activate Bid during death receptor-mediated apoptosis.
The results indicate that BID-independent pathways are responsible for FAS (show FAS ELISA Kits)-dependent human islet cell death in Type 1 diabetes.
The BID-MTCH2 (show MTCH2 ELISA Kits) axis regulates the differentiation/apoptosis of stem cells and mitochondrial metabolism. (Review)
Data suggest, in models of spatial propagation of mitochondrial permeabilization during apoptosis, there appears to be requirement for cooperative signaling involving truncated-BID and ROS (show ROS1 ELISA Kits) (reactive oxygen species) for efficient/ robust propagation.
this study emphasizes that the coordinated action of hGzmB-activated p53 (show TP53 ELISA Kits) and GzmB (show Gzmb ELISA Kits)-cleaved Bid is important for GzmB (show Gzmb ELISA Kits)-induced cell death and for cytotoxic lymphocyte/Natural Killer Cell-mediated killing of target cells.
Motifs of VDAC2 (show VDAC2 ELISA Kits) required for mitochondrial Bak (show BAK1 ELISA Kits) import and tBid-induced apoptosis.
Tax (show CNTN2 ELISA Kits) confers apoptosis resistance to HTLV-1-infected T cells by suppressing the expression of Bim (show BCL2L11 ELISA Kits) and Bid.
In the present study, we identified the proapoptotic Bcl-2 (show BCL2 ELISA Kits) family protein Bid as a positive regulator of mutant SOD1 (show SOD1 ELISA Kits)-induced TLR-nuclear factor-kappaB (NF-kappaB (show NFKB1 ELISA Kits)) signaling in microglia.
ATM (show ATM ELISA Kits)-BID-MTCH2 (show MTCH2 ELISA Kits) pathway that we have identified plays a critical role in the DDR (show DDR1 ELISA Kits) via regulation of mitochondrial metabolism.
DRP1 (show CRMP1 ELISA Kits)-dependent apoptotic mitochondrial fission occurs independently of BAX (show BAX ELISA Kits), BAK (show BAK1 ELISA Kits) and APAF1 (show APAF1 ELISA Kits) to amplify cell death by BID and oxidative stress.
A phosphorylation-deficient mutant of BID, MTCH2 (show MTCH2 ELISA Kits)'s ligand, induces an increase in OXPHOS, but with higher ROS (show ROS1 ELISA Kits) and reduced ATP levels than Mtch2 (show MTCH2 ELISA Kits) loss, and is associated with hypersensitivity to irradiation.
BID protein suppresses p38 (show CRK ELISA Kits) activity and induce malignant cell transformation of hepatocytes.
compare membrane permeabilization by Bax (show BAX ELISA Kits) activated by either cBid [cleaved Bid (p7 and p15 (show CDKN2B ELISA Kits))] or Bim (show BCL2L11 ELISA Kits) and examine the role of membrane lipids in the recruitment and activation of these three Bcl-2 (show BCL2 ELISA Kits) pro-apoptotic proteins
Hepatocyte Bid suppression is critical for the resistance to the lethal effects of Fas (show FAS ELISA Kits) activation. Fas (show FAS ELISA Kits) signaling induces differential activation of non-canonical interleukin-1beta maturation, amplified in the absence of apoptotic Bid in hepatocytes.
Data demonstrate a new role for Bid as a mediator of astrocyte activation during neuroinflammation, and suggest that Bid activation may contribute to non-cell autonomous motoneuron degeneration in amyotrophic lateral sclerosis
BID is required for tumor suppression by NSAIDs in APCMin/+ mice. It mediates these chemopreventive effects via selective killing of APC (show APC ELISA Kits)-deficient intestinal stem cells.
Bid and Bax (show BAX ELISA Kits) are signal transduction factors in granulosa cells and play proapoptotic roles.
This gene encodes a death agonist that heterodimerizes with either agonist BAX or antagonist BCL2. The encoded protein is a member of the BCL-2 family of cell death regulators. It is a mediator of mitochondrial damage induced by caspase-8 (CASP8)\; CASP8 cleaves this encoded protein, and the COOH-terminal part translocates to mitochondria where it triggers cytochrome c release. Multiple alternatively spliced transcript variants have been found, but the full-length nature of some variants has not been defined.
BH3 interacting domain death agonist
, BH3-interacting domain death agonist
, BID isoform ES(1b)
, BID isoform L(2)
, BID isoform Si6
, Human BID coding sequence
, apoptic death agonist
, desmocollin type 4
, p22 BID
, apoptotic death agonist BID