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anti-Human F2RL2 Antibodies:
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These data highlight the importance of the carboxy-terminal motif of the E6 protein and downregulation of PAR3 in tumorigenic transformation of human cervical keratinocytes.
Results suggest that Par3 expression is likely involved in ovarian cancer progression, especially in peritoneal metastasis, probably through modulating IL-6 (show IL6 Antibodies) /STAT3 (show STAT3 Antibodies) signaling.
reduced keratinocytes Par3 function fosters a permissive P-cadherin-dependent niche for melanocytes transformation, invasion, and metastasis.
These results demonstrate the importance of Par3 and SNAIL (show SNAI1 Antibodies) in development of KSHV-induced B-cells cancers
PAR-3 protein expression is frequently lost in primary esophageal squamous cell carcinoma and loss of the PAR-3 protein is associated with aggressive clinicopathological features.
Taken together, these results suggest that the Par3 regulates invasion and metastasis in pancreatic cancers by controlling tight junction assembly via Tiam1 (show TIAM1 Antibodies).
PAR3 is essential in thrombin (show F2 Antibodies) stimulated insulin (show INS Antibodies) secretion.
Knockdown of the polarity protein Par3 reversed the effects of Galpha13 (show GNA13 Antibodies) knockdown on cell-cell adhesion and proteolytic invasion in three-dimensional collagen.
Par-3 plays an important role in the modulation of intestinal barrier function by regulating delivery of occludin as well as suppression of MLC phosphorylation.
TFRGAP, a PAR-3 mimicking peptide significantly induced the phosphorylation of eNOS (show NOS3 Antibodies)-Thr (show TRH Antibodies)-495 with minimal phosphorylation of eNOS (show NOS3 Antibodies)-Ser (show SIGLEC1 Antibodies)-1177 with no change in nitric oxide production.
found a hypomethylated region mapping to Iqgap2 (IQ motif-containing GTPase activating protein 2 (show IQGAP2 Antibodies)) and F2rl2
Simultaneous depletion of PAR-1 (show MARK2 Antibodies) and PAR-3 almost completely inhibited epithelial-mesenchymal transition in bleomycin-induced lung fibrosis.
these data point to a novel kallikrein 6 (show KLK1 Antibodies) -signaling axis in neurons that is mediated by PAR1 (show F2R Antibodies) and PAR2 (show F2RL1 Antibodies) and is positioned to contribute to neurodegeneration
Par3(-/-) mice were protected against ferric chloride-induced thrombosis of mesenteric arterioles and against thromboplastin (show F3 Antibodies)-induced pulmonary embolism (protease-activated receptor-3 (PAR3)
PAR3 receptors and analogues can mediate cell signaling by interaction with PAR1 (show F2R Antibodies)-type thrombin (show F2 Antibodies) receptors
Crystal structures reveal the molecular basis of the cofactor function of cleaved PAR3, bound to exosite I, on recognition of PAR4 (show F2RL3 Antibodies) by the active site of thrombin (show F2 Antibodies).
This gene encodes a member of the PARD protein family. PARD family members interact with other PARD family members and other proteins\; they affect asymmetrical cell division and direct polarized cell growth. Multiple alternatively spliced transcript variants have been described for this gene.
proteinase-activated receptor 3
, Coagulation factor II receptor-like 2 (protease-actovated receptor 3)
, protease-activated receptor 3
, proteinase-activated receptor-3
, thrombin receptor-like 2
, Proteinase activated receptor 3 precursor (PAR-3) (Thrombin receptor-like 2) (Coagulation factor II receptor-like 2)
, coagulation factor II receptor-like 2
, CTCL tumor antigen se2-5
, atypical PKC isotype-specific interacting protein
, atypical PKC isotype-specific-interacting protein
, par-3 family cell polarity regulator alpha
, par-3 partitioning defective 3 homolog
, partitioning defective 3 homolog
, partitioning-defective 3 protein
, partitioning-defective protein 3 homolog