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Human IL6ST Primary Antibody for ELISA - ABIN625085
Lu, Zhu, Jiang, Sun, Jia, Qian, Li, Ma: Matrine increases NKG2D ligand ULBP2 in K562 cells via inhibiting JAK/STAT3 pathway: a potential mechanism underlying the immunotherapy of matrine in leukemia. in American journal of translational research 2015
The expression of IL-6 (show IL6 ELISA Kits), IL-6R and gp130 transcripts were detected very early, increased during the first week of life and were predominantly expressed in the head, epidermis and neuromasts of the anterior and posterior lateral line system.
C2C12 myotubes possesses a mechanism for regulating IL-6R and gp130 (show LRPPRC ELISA Kits) expression following lipoic acid treatment or heat shock.
Results suggest that the protective role of gp130 (show LRPPRC ELISA Kits)-dependent STAT3 (show STAT3 ELISA Kits) activation in experimental colitis involves the expansion and activation of mucosal myeloid-derived suppressor cells.
IL-6 (show IL6 ELISA Kits)/gp130 (show LRPPRC ELISA Kits) signalling in the osteoclast is not essential for normal bone resorption in vivo, but maintains both trabecular and periosteal bone formation in male mice by promoting osteoblast activity through the stimulation of osteoclast-derived coupling factors and osteotransmitters
gp130 (show LRPPRC ELISA Kits)-mediated adipose tissue lipolysis promotes hepatic steatosis and insulin (show INS ELISA Kits) resistance.
differences exist in the expression of receptors utilized by the IL-6 (show IL6 ELISA Kits)/gp130 (show LRPPRC ELISA Kits) family of cytokines in astrocytes and microglia, and (2) the findings provide a molecular basis for the differential response to oncostatin M (show OSM ELISA Kits) by astrocytes versus microglia
Deregulation of gp130 (show LRPPRC ELISA Kits)/STAT3 (show STAT3 ELISA Kits) signalling augments the acute phase reponse and macrophage infiltration during atherosclerosis without impacting on the development of aortic plaques.
Significant differential expression (both increases and decreases) of multiple IL-6 (show IL6 ELISA Kits)/gp130 (show LRPPRC ELISA Kits) cytokines, such as LIF (show LIF ELISA Kits), oncostatin-M (show OSM ELISA Kits), and ciliary neurotrophic factor (show CNTF ELISA Kits), occurred after Muller cell ablation
GP130 (show LRPPRC ELISA Kits) signaling collaborated with MYC (show MYC ELISA Kits) to induce MM and was responsible and sufficient for directing the plasma cell phenotype.
gp130 (show LRPPRC ELISA Kits), a co-receptor for IL-6 (show IL6 ELISA Kits) cytokines, triggers activation of YAP (show YAP1 ELISA Kits) and Notch (show NOTCH1 ELISA Kits), transcriptional regulators that control tissue growth and regeneration, independently of the gp130 (show LRPPRC ELISA Kits) effector STAT3 (show STAT3 ELISA Kits)
osteocytic gp130 (show LRPPRC ELISA Kits) signaling is required for normal trabecular bone mass and proper cortical bone composition.
IL-6sR and gp130 (show ANPEP ELISA Kits), but not IL-6R alpha (show IL6R ELISA Kits), play important roles in regulation of granulosa cell survival
IL-15 (show IL15 ELISA Kits) alone, similarly to IL-18 (show IL18 ELISA Kits), has no significant ability for regulation of both soluble IL-6 (show IL6 ELISA Kits) receptors, sIL (show PMEL ELISA Kits)-6R and sgp130, released by human neutrophils.
IL-6 signal transducing molecule
interleukin-6 receptor subunit beta
, interleukin 6 signal transducer
, IL-6 receptor subunit beta
, IL-6R subunit beta
, interleukin-6 signal transducer
, membrane glycoprotein 130
, oncostatin-M receptor subunit alpha
, glycoprotein 130