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RBMX may control SREBP-1c (show SREBF1 ELISA Kits) expression in mouse liver in response to high-fructose diet.
These results indicate a major role for SAFB1 (show SAFB ELISA Kits) in the activation of Srebp-1c (show SREBF1 ELISA Kits) through its interaction with RBMX.
HNRNPG binds m6A (show GPM6A ELISA Kits)-methylated RNAs through its C-terminal low-complexity region, which self-assembles into large particles in vitro. The Arg-Gly-Gly repeats within the low-complexity region are required for binding to the RNA motif exposed by m6A (show GPM6A ELISA Kits) methylation.
A sequence deletion within RBMX is identified as associated with with Shashi X-linked intellectual disability syndrome.
Host RBMX is required for the maintenance of Borna disease virus nuclear viral factories.
Results showed that HNRNPG and HTRA2 (show HTRA2 ELISA Kits)-BETA1 were specific antagonistic regulators of ERa exon7 splicing and increased HNRNPG levels were associated with improved clinical outcome of endometrial cancer through up-regulation of ERaD7 expression.
RBMX is a cohesion regulator that maintains the proper cohesion of sister chromatids.
Data show that RBMX accumulated at DNA lesions through multiple domains in a poly(ADP-ribose) polymerase 1 (show PARP1 ELISA Kits)-dependent manner and promoted HR by facilitating proper BRCA2 (show BRCA2 ELISA Kits) expression.
studied the genetic and expression states of hnRNP G in normal, premalignant and malignant human oral tissues to further understand the relationship between the hnRNP G alterations and the development of human oral cancer
evidence provided that deletions in or around RBMX may be involved in non-obstructive azoospermia(NOA (show DLAT ELISA Kits))
From these results, it seems that the X-chromosome, through its RBM (show RBMY1A1 ELISA Kits) genes, plays a formerly unknown role in the regulation of programmed cell death (apoptosis) in breast cancer.
p53 (show TP53 ELISA Kits) modulates DNA DSB repair by, in part, inducing hnRNP G
rbmx is required for normal embryonic development, in particular of the brain, consistent with a role in X-linked mental retardation
This gene belongs to the RBMY gene family which includes candidate Y chromosome spermatogenesis genes. This gene, an active X chromosome homolog of the Y chromosome RBMY gene, is widely expressed whereas the RBMY gene evolved a male-specific function in spermatogenesis. Pseudogenes of this gene, found on chromosomes 1, 4, 9, 11, and 6, were likely derived by retrotransposition from the original gene. Alternatively spliced transcript variants encoding different isoforms have been identified. A snoRNA gene (SNORD61) is found in one of its introns.
Heterogeneous nuclear ribonucleoprotein G
, RNA-binding motif protein, X chromosome
, hnRNP G
, RNA binding motif protein, X-linked
, heterogeneous nuclear ribonucleoprotein G
, RNA binding motif protein, X chromosome retrogene
, RNA binding motif protein, X-linked-like-1
, heterogeneous nuclear ribonucleoprotein G (hnRNP G)
, heterogeneous nuclear ribonucleoprotein G-like 1
, RNA binding motif protein, X chromosome
, glycoprotein p43
, RNA-binding motif protein, X chromosome retrogene
, RNA binding motif protein, X-linked-like 1
, testes-specific heterogenous nuclear ribonucleoprotein G-T