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Cow (Bovine) PLG ELISA Kit for Sandwich ELISA - ABIN612673
Forsgren, Råden, Israelsson, Larsson, Hedén: Molecular cloning and characterization of a full-length cDNA clone for human plasminogen. in FEBS letters 1987
Show all 3 references for ABIN612673
Mouse (Murine) PLG ELISA Kit for Sandwich ELISA - ABIN820647
Ly, Taylor, Tsatsaronis, Monteleone, Skora, Donald, Maddocks, Nizet, West, Ranson, Walker, McArthur, Sanderson-Smith: Plasmin(ogen) acquisition by group A Streptococcus protects against C3b-mediated neutrophil killing. in Journal of innate immunity 2014
Human PLG ELISA Kit for Sandwich ELISA - ABIN416924
Wang, Yuan, Zhong, Wen, Deng, Liang, Zheng: The anticoagulant effect of PGI2S and tPA in transgenic umbilical vein endothelial cells is linked to up-regulation of PKA and PKC. in International journal of molecular sciences 2014
S. aureus NCTC 8325-4 adheres to immobilized plasminogen in vitro and that the adhesion may be mediated by a C-terminal fragment of the PBP3 protein.[PBP3]
we demonstrated that PLG functions as a molecular bridge between tricellulin and streptococcal surface enolase (SEN). The wild type strain efficiently translocated across the epithelial monolayer, accompanied by cleavage of transmembrane junctional proteins.
Suggest that tubulointerstitial plasmin is associated with inflammation leading to renal fibrosis, and can cause the decline in renal function seen in patients with IgA nephropathy.
Plasminogen binding and activation by different glycolytic enzymes of M. pneumoniae play a role in successful colonization of the human respiratory tract.
reduced proteolytic activity of plasmin on structures of growing thrombi, rather than on complement activation fragments, explains the association of plasminogen deficiency with aHUS.
Zinc modulates fibrinolysis by attenuating tPA (show PLAT ELISA Kits)-mediated plasminogen activation and plasmin-induced fibrin degradation.
Plasmin cleavage of iC3b provides a complement regulatory pathway that is as efficient as FI/CR1 (show CR1 ELISA Kits) but does not require a cellular cofactor.
PLG is the third replicated shared genetic risk factor of atherosclerosis and periodontitis.
Data show that preincubation with plasminogen, wild-type group A Streptococcus (GAS) NS88.2 degraded complement C3b.
whereas the presence of plasminogen did not affect the factor I cofactor activity of C4BP (show C4BPA ELISA Kits), the activation of plasminogen by urokinase-type plasminogen activator (show PLAU ELISA Kits) to active plasmin was significantly augmented in the presence of C4BP (show C4BPA ELISA Kits).
The plasminogen/plasminogen activator/plasmin system is activated during gamete interaction and regulates the sperm entry into the oocyte.
The current study suggests the existence of an active TNF-alpha (show TNF ELISA Kits)-plasminogen-plasmin autocrine/paracrine loop on the massively infiltrated polymorphonuclear neutrophils inside udders of drying-off cows.
examination of interaction with streptococcal plasminogen activator PauA
Plasminogen may regulate DPP-4 (show DPP4 ELISA Kits) activity and glucose metabolism.
Plasminogen-deficient mice frequently develop colon lesions and rectal prolapses.
manganese transport protein (show SLC11A2 ELISA Kits) C (show PROC ELISA Kits) (MntC) is an extracellular matrix- and plasminogen-binding protein (show ENO1 ELISA Kits)
Plg from mouse plasma contains oxPtdPC adducts that are not affected by the action of Lp-PLA(2 (show Lp-PLA2 ELISA Kits)), suggesting that linkage to Plg protects oxPtdPCs from metabolism during their transport in the plasma.
plg-/- and tPA (show PLAT ELISA Kits)-/- mice exhibit brain parenchymal fibrin deposits that appear to result from reduced neurovascular integrity
plasminogen-dependent proteolysis has a beneficial effect during neurological recovery after stroke.
Pla-induced mononuclear cell recruitment in vivo was dependent on protease-activated receptor-1 activation of the MEK/ERK/NF-kappaB pathway, which led to the release of CCL2 and activation of CCR2.
Plg may play an important role in innate immunity by changing expression of genes that contribute to phagocytosis.
Plasminogen is a key molecular determinant of inflammatory joint disease capable of simultaneously driving or ameliorating arthritis pathogenesis in distinct anatomic locations in the same subject.
Plasmin suppresses brain metastasis in two ways: by converting membrane-bound astrocytic FasL (show FASL ELISA Kits) into a paracrine death signal for cancer cells, and by inactivating the axon pathfinding molecule L1CAM (show L1CAM ELISA Kits), which metastatic cells express for spreading along brain capillaries and for metastatic outgrowth.
The protein encoded by this gene is a secreted blood zymogen that is activated by proteolysis and converted to plasmin and angiostatin. Plasmin dissolves fibrin in blood clots and is an important protease in many other cellular processes while angiostatin inhibits angiogenesis. Defects in this gene are likely a cause of thrombophilia and ligneous conjunctivitis. Two transcript variants encoding different isoforms have been found for this gene.
, lipoprotein, Lp(a)
, plasmin heavy chain A
, plasmin light chain B