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these results demonstrate that DNAJB9 is a downstream target of p53 (show TP53 ELISA Kits) that belongs to the group of negative feedback regulators of p53 (show TP53 ELISA Kits).
ERdj4 and ERdj5 (show DNAJC10 ELISA Kits) promote turnover of misfolded SP-C (show SFTPC ELISA Kits) and this activity is dependent on their ability to stimulate BiP (show GDF10 ELISA Kits) ATPase (show DNAH8 ELISA Kits) activity.
ERdj4 gene trap mice showed non-specific hypergammaglobulinemia and gene trap B (show SSR2 ELISA Kits) cells exhibited increased proliferation, survival and isotype switching in response to LPS (show TLR4 ELISA Kits) stimulation.
Deficiency of the BiP (show HSPA5 ELISA Kits) cochaperone ERdj4 causes constitutive endoplasmic reticulum stress and metabolic defects.
directly link ERdj4 to the ERAD machinery and suggest a model in which ERjd4 could help recruit clients from throughout the ER to ERAD sites
DnaJ (show DNAJA2 ELISA Kits) homolog, which stimulates ATPase (show DNAH8 ELISA Kits) activity of BiP (show HSPA5 ELISA Kits) and is induced by endoplasmic reticulum stress
This gene is a member of the J protein family. J proteins function in many cellular processes by regulating the ATPase activity of 70 kDa heat shock proteins. This gene is a member of the type 2 subgroup of DnaJ proteins. The encoded protein is localized to the endoplasmic reticulum. This protein is induced by endoplasmic reticulum stress and plays a role in protecting stressed cells from apoptosis.
dnaJ homolog subfamily B member 9
, endoplasmic reticulum DnaJ homolog 4
, microvascular endothelial differentiation gene 1 protein
, DNA j protein b9
, microvascular endothelial differentiation gene 1
, dnaJ homolog, subfamily b, member 9