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Inhibition of kif3a in the human dental follicle cells (hDFCs) and human dental pulp cells (hDPCs) is disrupted primary cilia formation and/or function, and it was shown that kif3a is important in the differentiation of hDFCs and hDPCs through the Wnt (show WNT2 ELISA Kits) pathway.
High KIF3A expression is associated with glioblastoma.
These data suggest that GLI (show GLI1 ELISA Kits) interactions with KIF3A-KIF3B (show KIF3B ELISA Kits)-KAP3 (show KIFAP3 ELISA Kits) complexes are essential for proper GLI (show GLI1 ELISA Kits) transcriptional activity.
KIF3A and OVOL1 (show OVOL1 ELISA Kits) are involved in the development of Atopic dermatitis in the Chinese pediatric population.
both motor domains of KIF3A/B coordinate for processive motility and move at different speeds
Ablation of this phosphorylation abolished herpes simplex virus 1 US3-mediated downregulation of CD1d (show CD1D ELISA Kits) expression, suggesting that phosphorylation of KIF3A is the primary mechanism of viral suppression of CD1d (show CD1D ELISA Kits) expression.
Findings support the notion that upregulation of KIF3a is causal of aberrant activation of Wnt (show WNT2 ELISA Kits) signaling in advanced prostate cancer through the KIF3a-DVL2 (show DVL2 ELISA Kits)-beta-catenin (show CTNNB1 ELISA Kits) axis.
POPX2 (show PPM1F ELISA Kits) affects trafficking by determining the phosphorylation status of KIF3A at serine 690.
Association of KIF3A, but not OVOL1 and ACTL9, with atopic eczema in Italian patients
in addition to its ciliogenic roles, Kif3a recruits p150(Glued (show DCTN1 ELISA Kits)) to the subdistal appendages of mother centrioles, critical for centrosomes to function as microtubule-organizing centres.
Longitudinal microcomputed tomography (muCT) imaging and histopathological analyses revealed an increased rate of cyst formation, increased proportion of cysts with proliferating cells, higher frequency of atypical cysts as well as the development of neoplasms in Vhl (show VHL ELISA Kits)/Kif3a/Trp53 (show TP53 ELISA Kits) mutant kidneys compared to Kif3a/Trp53 (show TP53 ELISA Kits) or Vhl (show VHL ELISA Kits)/Kif3a mutant kidneys.
Data suggest biocatalysis by Kif3a homodimer, Kif3c (show KIF3C ELISA Kits) homodimer, or Kif3a/Kif3c (show KIF3C ELISA Kits) heterodimer vary; microtubule interaction is very fast for Kif3a homodimer; Kif3c (show KIF3C ELISA Kits) homodimer interaction is much slower; Kif3a/Kif3c (show KIF3C ELISA Kits) heterodimer interaction is intermediate.
KIF1-binding protein (show KIAA1279 ELISA Kits) interacts with KIF3A in haploid male germ cells, promoting spermatid elongation.
Vhl (show VHL ELISA Kits) and Kif3a deletion accelerates renal cyst formation
The results of this study suggested thatactivity-dependent cargo loading, in which phosphorylation of the KIF3A C terminus upregulates the loading and transport of N-cadherin (show CDH2 ELISA Kits) in homeostatic synaptic plasticity.
The results demonstrate that visual pigments transport to the retinal outer segment despite removal of KIF3 and IFT88 (show IFT88 ELISA Kits), and KIF3-mediated anterograde IFT is responsible for photoreceptor transition zone and axoneme formation.
Processivity of the kinesin-2 (show KIF2A ELISA Kits) KIF3A results from rear head gating and not front head gating.
A ciliopathy with hydrocephalus, isolated craniosynostosis, hypertelorism, and clefting caused by deletion of Kif3a
KIF3AB exhibits novel ATPase (show DNAH8 ELISA Kits) activity.
single-molecule motility from different kinesin-1 and kinesin-2 (show KIF2A ELISA Kits) neck-linker chimeras stepping along microtubules, was studied.
mouse homolog is a molecular motor protein involved in the development of primary cilia
kinesin family protein 3A
, kinesin-like protein KIF3A
, microtubule plus end-directed kinesin motor 3A
, DNA sequence AF180004
, DNA sequence AF180009
, N-4 kinesin
, kinesin-II subunit