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Human Polyclonal CLDN5 Primary Antibody for IHC (p), IHC - ABIN266211
Davis, Sonnet, Lazard, Henslee, Gugala, Salisbury, Strecker, Davis, Forsberg, Davis, Olmsted-Davis: Location-dependent heterotopic ossification in the rat model: The role of activated matrix metalloproteinase 9. in Journal of orthopaedic research : official publication of the Orthopaedic Research Society 2016
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Human Polyclonal CLDN5 Primary Antibody for ELISA, WB - ABIN563201
Escudero-Esparza, Jiang, Martin: Claudin-5 participates in the regulation of endothelial cell motility. in Molecular and cellular biochemistry 2012
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Cow (Bovine) Polyclonal CLDN5 Primary Antibody for WB - ABIN2778047
Fontijn, Volger, Fledderus, Reijerkerk, de Vries, Horrevoets: SOX-18 controls endothelial-specific claudin-5 gene expression and barrier function. in American journal of physiology. Heart and circulatory physiology 2008
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Human Polyclonal CLDN5 Primary Antibody for IHC (p), IHC - ABIN266210
Hongo, Watanabe, Arita, Kanome, Kageyama, Shioda, Miyazaki: Leptin modulates ACAT1 expression and cholesterol efflux from human macrophages. in American journal of physiology. Endocrinology and metabolism 2009
Human Polyclonal CLDN5 Primary Antibody for IF (p), IHC (p) - ABIN731243
Ruan, Hu, Ao, Ma, Gao, Liu, Kong, Bao, Yu: The neurovascular protective effects of huperzine A on D-galactose-induced inflammatory damage in the rat hippocampus. in Gerontology 2014
In conclusion, our present study indicated that miR (show MYLIP Antibodies)-34c regulated the permeability of BTB via MAZ (show MAZ Antibodies)-mediated expression changes of ZO-1 (show TJP1 Antibodies), occludin (show OCLN Antibodies), and claudin-5.
The results of light- and electron-microscopic observations suggested that cldn5a and 5b are required for Xenopus heart tube formation through epithelialization of the precardiac mesoderm.
Downregulation of cldn5a in zebrafish showed a failure in organ laterality that resulted from malformed Kupffer's vesicle
identify Claudin5a as a core component of an early cerebral-ventricular barrier system that is required for ventricular lumen expansion in the zebrafish embryonic brain before the establishment of the embryonic blood-brain barrier.
Claudin-5 single-nucleotide polymorphism (SNP) rs885985 has two major alleles, G and A, which encode for glutamine (show GFPT1 Antibodies) (Q) or a stop signal, respectively, resulting in distinct overlapping open readings.
Data show that the charge of Lys65 in claudin 1 (Cldn1 (show CLDN1 Antibodies)) and Glu158 in claudin 3 (Cldn3 (show CLDN3 Antibodies)), and of Gln57 in claudin 5 (Cldn5) are necessary for tight junction (TJ) strand formation.
suggest that the reduction of CLDN5, 7, and 18 expression loses the suppressive ability of interaction between PDK1 (show PDK1 Antibodies) and Akt (show AKT1 Antibodies) and causes sustained phosphorylation of Akt (show AKT1 Antibodies), resulting in the disordered proliferation in lung squamous carcinoma cells
The levels of ESM1 (show ESM1 Antibodies), CLDN5, IL-1beta (show IL1B Antibodies), IL-6 (show IL6 Antibodies), and TNF-alpha (show TNF Antibodies) were significantly higher in the migraine attack group than in the control group
analysis of the membrane driven cis (show CISH Antibodies) interactions of claudin-5 proteins in the formation of the blood brain barrier tight junctions
the expression of claudin-5 and claudin-9 (show CLDN9 Antibodies) was down-regulated while the expression of claudin-8 (show CLDN8 Antibodies) was up-regulated in cervical carcinoma tissues compared with adjacent non-neoplastic tissues.
Plasma CLDN5 levels decreased in patients with stable asthma compared with those in control subjects, suggesting that asthma therapy can decrease plasma CLDN5 levels
These data support that loss of claudin-5 in cardiomyocytes and endothelial cells is prevalent in human heart failure
the relationship between vWF (show VWF Antibodies) and claudin-5, which are indicators of endothelial cell dysfunction and tight junction activity, may be a predictor of disease activity in rheumatoid arthritis.
The findings suggest that down regulated CLDN1 (show CLDN1 Antibodies) and CLDN5 genes have potential relevance in relation to the progression of glioblastoma multiforme.
This study demonstrated that the claudin-5(+) leukocytes in the central nervous system during neuroinflammation: a novel role for endothelial-derived (show MED28 Antibodies) extracellular vesicles.
claudin-3 (show CLDN3 Antibodies) and claudin-5 expression is increased by DHEAS (show SULT2A1 Antibodies) and tight junction formation is stimulated via a Gnalpha11-coupled receptor in Sertoli cells
Claudin 5 in a murine model of allergic asthma: Its implication and response to steroid treatment
while MMP-9 (show MMP9 Antibodies) is not essential for hypoxic-induced cerebral angiogenesis, it plays an important role in post-hypoxic vascular pruning by degrading laminin and claudin-5
Studied if circulating TGFbeta1 (show TGFB1 Antibodies) drives changes in tight junction protein (show OCLN Antibodies) expression and MMP9 (show MMP9 Antibodies) activity following acute liver failure; Mouse brain endothelial cells were treated with recombinant rTGFbeta1 and MMP9 (show MMP9 Antibodies) and claudin-5 expression was assessed.
In transgenic mice carrying mutated SOD1 (show SOD1 Antibodies) genes, a disrupted blood-spinal cord barrier as well as decreased levels of tight junction proteins ZO-1 (show TJP1 Antibodies), occludin (show OCLN Antibodies), and claudin-5 were detected.
The aim of the present study was to further characterize the estrogen-responsive elements of claudin-5 promoter.
Two independent pathways triggered by PI3K mediate early and late loss of paracellular claudin-5 expression.
Comparative modelling of intramolecular interfaces in the transmembrane region of claudins led to a complete Cldn5 model.
silencing of claudin-5 did significantly attenuate simvastatin-mediated endothelial cell barrier protection in response to thrombin (show F2 Antibodies)
Data show that IL-4 (show IL4 Antibodies) induces upregulation of the junction protein claudin-5 in endothelial cells (ECs) through activation of Jak (show JAK3 Antibodies)/STAT6 (show STAT6 Antibodies) and phosphorylation and translocation of FoxO1 (show FOXO1 Antibodies) from the nucleus to the cytoplasm.
This gene encodes a member of the claudin family. Claudins are integral membrane proteins and components of tight junction strands. Tight junction strands serve as a physical barrier to prevent solutes and water from passing freely through the paracellular space between epithelial or endothelial cell sheets. Mutations in this gene have been found in patients with velocardiofacial syndrome. Alternatively spliced transcript variants encoding the same protein have been found for this gene.
, claudin 5
, claudin 5 (transmembrane protein deleted in velocardiofacial syndrome)
, transmembrane protein deleted in VCFS
, transmembrane protein deleted in velocardiofacial syndrome
, brain endothelial cell clone 1 protein
, lung-specific membrane protein