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KLF9 status was associated with differentiation and vascular invasion expression and was found to be a valuable prognostic factor for patients with pancreatic ductal adenocarcinoma
In conclusion, LPA (show APOA ELISA Kits) plays dual roles as a ligand mediator through the activation of cell surface G-coupled protein receptors and as an intracellular second messenger through the activation of PPARgamma (show PPARG ELISA Kits). We discuss the contribution of the LPA1 (show LPAR1 ELISA Kits)-PPARgamma (show PPARG ELISA Kits)-KLF9 axis to neurite outgrowth and proliferation in human iPSC-derived neurons.
miR (show MLXIP ELISA Kits)-141-3p/KLF9 may play an important role in regulating the growth of prostate cancer
The expression of KLF9 is down-regulated in esophageal squamous cell carcinoma and inversely correlated with the clinical features.
Suggest role for miR (show MLXIP ELISA Kits)-570/KLF9 molecular network in controlling lung carcinoma progression.
TRs cooperate with KLF9 to regulate hepatocyte proliferation and differentiation and early stages of organogenesis and that TRs exert widespread and important influences on ESC biology.
Myometrial KLF9 may contribute to the onset of human parturition through its regulation of PGR (show PGR ELISA Kits) expression and inflammatory signaling networks.
the expression of KLF9 is up-regulated in human ovarian cancer; KLF9 knockdown significantly inhibited cell proliferation and resulted in cell cycle arrest in the G0/G1 phase
In this review, we focus on the functions, roles, and regulatory networks of these five KLFs in HCC (show FAM126A ELISA Kits), summarize key pathways, and propose areas for further investigation
Palmitic acid increases Ppargamma (show PPARG ELISA Kits) and Klf6 (show KLF6 ELISA Kits) & Klf9 gene expression and promotes triglyceride accumulation in HepG2 cells.
Study identified genomic targets of Klf9 in hippocampal neurons which raise some indication about it functions in chromatin, and neuronal morphology regulation and survival across the lifespan.
Kruppel-like factor 9 (KLF9) is a haploinsufficient tumor suppressor in the ApcMin/+ mouse colon by suppressing expression of ISG15 (show ISG15 ELISA Kits), an apoptosis-inhibiting cytokine.
in endometrial stromal cells, KLF9 provides coincident regulation of Notch (show NOTCH1 ELISA Kits), Hh, and steroid receptor (show ESR2 ELISA Kits) signaling pathways, which when deregulated, underlie establishment and/or progression of endometriosis
Nrf2 (show NFE2L2 ELISA Kits) amplifies oxidative stress via induction of Klf9.
KLF9 bound the KLF binding element at position -874 of the mouse C/EBPbeta (show CEBPB ELISA Kits) promoter.
Klf9 is essential for turning off programmed cell death of Purkinje cells in organotypic cultures.
We showed that Kruppel-like factor 9 was a key mediator of this effect of T(3); the sudden physiological increase in T(3) during development is involved in the onset of the loss of axon regenerative capacity in purkinje cells.
KLF9 is an integral component of the T3-driven signaling cascade that promotes myelin regeneration.
Common variants at CDKAL1 (show CDKAL1 ELISA Kits) and KLF9 are associated with body mass index in east Asian populations.
Data show that forced expression of Kruppel-like factor 9 (Klf9) in the brain of thyroid-intact tadpoles increased baseline thyroid hormone receptor-beta (trb (show THRB ELISA Kits)) mRNA and enhanced trb autoinduction.
Induced expression of BTEB1 in XTC-2 cells caused accelerated and enhanced autoinduction of the TrbetaA gene.
KLF9 was verified to be directly regulated by TH in a TH receptor (TR)-dependent manner in hypothyroid mutants
knockdown of klf9 in zebrafish embryos impaired hematopoietic development including erythroid maturation and T lymphopoiesis
The protein encoded by this gene is a transcription factor that binds to GC box elements located in the promoter. Binding of the encoded protein to a single GC box inhibits mRNA expression while binding to tandemly repeated GC box elements activates transcription.
BTE-binding protein 1
, GC-box-binding protein 1
, Krueppel-like factor 9
, basic transcription element binding protein 1
, basic transcription element-binding protein 1
, transcription factor BTEB1
, Kruppel-like factor 9
, gene 1
, gene 3
, basic transcription element binding protein 1 (gene 1)