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There was an association between the AGT (show AGXT Proteins) rs699 polymorphism and diabetes mellitus type 2 in the Brazilian patients.
Results indicate that urinary angiotensinogen (show AGT Proteins) was associated with blood pressure elevation in this population of obese young adults.
our analysis revealed that the associations of the AGT (show AGXT Proteins) variants with T2DM were independently associated.
The urinary angiotensinogen (show AGT Proteins) level correlates with the overall maturity of renal function during the early postnatal period in critically ill neonates.
the association of polymorphisms in angiotensinogen (AGT (show AGT Proteins)) and angiotensin II type 1 receptor (AGTR1 (show AGTR1 Proteins)), with blood lead levels and lead-related blood pressure in lead-exposed male workers in Korea, was investigated.
the AGT (show AGXT Proteins) polymorphisms have played a vital role in determining an individual's susceptibility to essential hypertension.
There were significant differences in genotype and allele distributions of ACE (show ACE Proteins) gene rs4343 (pgentoype = 0.002 and pallele < 0.001) and AGTR1 (show AGTR1 Proteins) gene rs5186 polymorphisms.
rs699 in the AGT gene and rs5186 in the ATR1 gene were not associated with the risk and clinical outcomes of IgAN.
The threonine variant of AGT (show AGXT Proteins) M268T is a significant risk factor for abnormalities in specific white matter tracts and cognitive domains in healthy older adults.
Increased plasma renin, AT and VEGF might play a role in the onset or development of superficial infantile hemangiomas.
These results suggest that increased formation of AT1R (show AGTRAP Proteins)-P2Y6R (show P2RY6 Proteins) heterodimers with age may increase the likelihood of hypertension induced by Ang II (show AGT Proteins).
These data suggest dysregulation of renin (show REN Proteins)-angiotensin system in acute pancreatitis as evidenced by altered Ang II/Ang-(1 (show ANGPT1 Proteins)-7) levels induced by the imbalance of ACE (show ACE Proteins)/ACE2 (show ACE2 Proteins) activity.
Results indicate that Ang III (show AGT Proteins) produces nociceptive behavior similar to Ang II (show AGT Proteins), and suggest that the phosphorylation of p38 MAPK (show MAPK14 Proteins) mediated through AT1 (show SLC33A1 Proteins) receptors on spinal astrocytes and neurons contributes to Ang II (show AGT Proteins)- and III-induced nociceptive behavior
SIRT1 (show SIRT1 Proteins) and the axis AII (show ARG2 Proteins)/AT1 (show SLC33A1 Proteins)/NADPH-oxidase (show NOX1 Proteins) regulate each other
Angiotensin (1-7) and Bradykinin may have roles in diabetic nephropathy induced by increased ACE (show ACE Proteins) gene dosage
MAS (show MAS1 Proteins) receptors mediate vasoprotective and atheroprotective effects of candesartan upon the recovery of vascular angiotensin-converting enzyme 2 (show ACE2 Proteins)-angiotensin-(1-7)-MAS (show MAS1 Proteins) axis functionality
AGT (show AGXT Proteins) contributes to body weight gain and liver steatosis through functions of the des (show DES Proteins)(angiotensin I)AGT domain, which are independent of angiotensin II production.
the augmented AGT (show AGXT Proteins) released from apoptotic endothelial cells acts as a vital progenitor of Ang II (show AGT Proteins) to accelerate vascular remodelling
Humid heat stress increased oxidative stress and caused apoptosis of cardiomyocytes through the Ang II (show AGT Proteins) signaling pathway.
ACE2 (show ACE2 Proteins) and Ang-(1 (show ANGPT1 Proteins)-7) significantly inhibit early atherosclerotic lesion formation via protection of endothelial function and inhibition of inflammatory response.
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll