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rs699 in the AGT gene and rs5186 in the ATR1 gene were not associated with the risk and clinical outcomes of IgAN.
The threonine variant of AGT (show AGXT Proteins) M268T is a significant risk factor for abnormalities in specific white matter tracts and cognitive domains in healthy older adults.
Increased plasma renin, AT and VEGF might play a role in the onset or development of superficial infantile hemangiomas.
Blood pressure was significantly lower in angiotensinogen (show AGT Proteins) knock-out mice compared to controls fed a normal or high sodium diet.
Detection of hANG(1-12) in hearts of homozygous transgenic rats expressing the hAGT gene suggests existence of non-renin (show REN Proteins)-dependent enzyme mechanism capable of cleavage of human substrate.
AGT (show AGXT Proteins)-H1 haplotype was identified as protective Factor for Essential Hypertension in Kazakhs in Xinjiang.
dominance of the aminopeptidase A (show ENPEP Proteins)/Ang (2 (show ANGPT2 Proteins)-10) and aminopeptidase A (show ENPEP Proteins)/Ang III (show AGT Proteins) (2-8) pathways in the metabolism of Ang I (show AGT Proteins) (1-10) and Ang II (show AGT Proteins) (1-8) over the ACE2 (show ACE2 Proteins)/Ang (1 (show ANGPT1 Proteins)-7) axis
The ACE2 (show ACE2 Proteins)-Ang (1 (show ANGPT1 Proteins)-7)-Mas (show MAS1 Proteins) axis serves as an important signal pathway in endothelial cell protection in diabetic human arteries.
Urinary AGT (show AGXT Proteins) could be a prognostic marker of renoprotective effects of alogliptin in patients with T2D
urinary AGT (show AGXT Proteins)/protein ratio represents a novel specific marker of podocyte injury
Results indicate that Ang III (show AGT Proteins) produces nociceptive behavior similar to Ang II (show AGT Proteins), and suggest that the phosphorylation of p38 MAPK (show MAPK14 Proteins) mediated through AT1 (show SLC33A1 Proteins) receptors on spinal astrocytes and neurons contributes to Ang II (show AGT Proteins)- and III-induced nociceptive behavior
SIRT1 (show SIRT1 Proteins) and the axis AII (show ARG2 Proteins)/AT1 (show SLC33A1 Proteins)/NADPH-oxidase (show NOX1 Proteins) regulate each other
Angiotensin (1-7) and Bradykinin may have roles in diabetic nephropathy induced by increased ACE (show ACE Proteins) gene dosage
MAS (show MAS1 Proteins) receptors mediate vasoprotective and atheroprotective effects of candesartan upon the recovery of vascular angiotensin-converting enzyme 2 (show ACE2 Proteins)-angiotensin-(1-7)-MAS (show MAS1 Proteins) axis functionality
AGT (show AGXT Proteins) contributes to body weight gain and liver steatosis through functions of the des (show DES Proteins)(angiotensin I)AGT domain, which are independent of angiotensin II production.
the augmented AGT (show AGXT Proteins) released from apoptotic endothelial cells acts as a vital progenitor of Ang II (show AGT Proteins) to accelerate vascular remodelling
Humid heat stress increased oxidative stress and caused apoptosis of cardiomyocytes through the Ang II (show AGT Proteins) signaling pathway.
ACE2 (show ACE2 Proteins) and Ang-(1 (show ANGPT1 Proteins)-7) significantly inhibit early atherosclerotic lesion formation via protection of endothelial function and inhibition of inflammatory response.
reductions in plasma angiotensin concentrations in obese hepatocyte AGT (show AGXT Proteins)-deficient mice may have limited megalin (show LRP2 Proteins)-dependent uptake of AGT (show AGXT Proteins) into adipocytes for the production of Ang II (show AGT Proteins) in the development of obesity-hypertension.
Overexpression of clusterin (show CLU Proteins) in proximal tubular epithelial cells decreased the levels of Ang II (show AGT Proteins)-stimulated fibrotic markers and AT1R (show AGTRAP Proteins).
The protein encoded by this gene, pre-angiotensinogen or angiotensinogen precursor, is expressed in the liver and is cleaved by the enzyme renin in response to lowered blood pressure. The resulting product, angiotensin I, is then cleaved by angiotensin converting enzyme (ACE) to generate the physiologically active enzyme angiotensin II. The protein is involved in maintaining blood pressure and in the pathogenesis of essential hypertension and preeclampsia. Mutations in this gene are associated with susceptibility to essential hypertension, and can cause renal tubular dysgenesis, a severe disorder of renal tubular development. Defects in this gene have also been associated with non-familial structural atrial fibrillation, and inflammatory bowel disease.
alpha-1 antiproteinase, antitrypsin
, angiotensin I
, angiotensin II
, serine (or cysteine) proteinase inhibitor
, serpin A8
, angiotensinogen (PAT)
, angiotensin ll